NIH Research Festival 2012 Program - Research Festival - National ...

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Concurrent Symposia Session III Natcher Conference Center Ruth L. Kirschstein Auditorium 48 Wednesday, October 10, 2012 2:00 p.m.–4:00 p.m. Stem cells in development and diseases Chair: Steven Hou, NCI Tissues and organs in animals are generated and maintained by stem cells, which possess the potential for unlimited self-renewal. Through asymmetric cell division, a stem cell in adult tissues can produce one daughter cell whose self-renewing progeny maintain the stem-cell population and a second daughter cell that will give rise to one or many differentiated and short-lived cell types that will replace damaged or dying cells. Similarly, tumors may originate from a few transformed cells with stem-cell characteristics, called cancer stem cells. Stem cells have immense potential for therapeutic use in regenerative medicine and as targets for anticancer therapies. To make use of this potential, we must first understand the molecular parameters that define a stem cell and the mechanisms that regulate stem-cell behavior. This symposium will bring together NIH experts working on basic stem cell biology and stem cell-related diseases. Matthew Hoffman, NIDCR Kit and Fgfr2b regulate progenitor cell expansion during organogenesis Steven Hou, NCI Stem cell regulation in drosophila intestine Isaac Brownell, NCI Microenvironment in the regulation of hair follicle stem cells Ramiro Iglesias-Barthomew and Silvio Gutkind, NIDCR mTor signaling and epithelial stem cell regulation Paola Scaffidi and Tom Misteli, NCI In vitro generation of human cells with cancer stem cell properties Rita Humeniuk, NCI Tumor suppressor p15Ink4b determines cell fate of hematopoietic progenitors: Implications for development of human blood disorders FARE Award Winner
Concurrent Symposia Session III Natcher Conference Center Conference Room E1/E2 Imaging traumatic brain injury: Challenges and solutions Co-chairs: Amir H. Gandjbakhche, NICHD; and Paul Smith, NIBIB Wednesday, October 10, 2012 2:00 p.m.–4:00 p.m. Approximately 1.7 million people sustain a traumatic brain injury annually. 300,000 veterans in Iraq/Afghanistan wars experienced TBI, mostly mild. Injuries range from severe structural damages detected in (MRI/CT) imaging, to those injuries which exhibit normal structural imaging but manifest different levels of cognitive deficits such as memory problems, reduced attention, inability to concentrate on a single task. The poor quality of life associated with TBI, create severe morbidities in all aspects of patient life (family, work, and society), and cost billions of dollars. Accurate diagnosis and classification of TBI are becoming a critical need for personalized therapy. However, phenotyping TBI is still a huge challenge. Imaging methods, both structural and functional, could play an important role for phenotype classification. In this symposium we will address the role of imaging techniques for TBI diagnosis and therapy. James Smirniotopoulos, USUHS Imaging TBI: Known knowns and unknown unknowns Ramon Diaz-Arrastia, USUHS Endophenotypes of traumatic brain injury: Implications for the next generation of clinical trials Lawrence Latour, NINDS The CNRM traumatic head injury neuroimaging classification (THINC) study: Steps toward the objective diagnosis and classification of TBI Amir Gandjbakhche, NICHD Imaging cognitive function with near infrared spectroscopy for TBI diagnosis Emily Wood, NINDS Detecting axonal degeneration in multiple sclerosis with diffusion weighted spectroscopy FARE Award Winner 49
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