Respiratory System Disorders and Therapy From a New - Louis Bolk ...

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ut is normally different in the liver than in the lungs. Under physiological circumstances, the presence of liquid components outside of the blood- and lymphstream is concentrated in organs below the diaphragm. The gastrointestinal tract contains fluid chyme, the liver has blood-filled sinuses, the gall bladder contains bile, the pancreas produces dissolved enzymes, the kidney produces urine, and the bladder holds urine. Free air in the human body can be found in the airways that are situated above the diaphragm: the nasal sinuses, the airways including the middle ear, and the lungs. When there is exudate in the airways, the lungs take on characteristics that belong to the organs below the diaphragm. There is another interesting aspect. In the Physiology Companion (Tellingen 2008. Ch. 2), we described the respiratory tract as a membranous organ system that is enclosed by physically hard bones and cartilage. Mechanically defined pressure relations and gas exchange determine lung physiology. This is in contrast to systems such as the digestive tract, the urogenital system, and the circulation where mechanical forces are less prominent. In hepatisation of the lung, the physical forces are no longer effective and are displaced by an accumulation of fluid. The balance of the dissolving and consolidating forces is then shifted in the direction of the dissolving tendency in the sense of fig.2.4. Decreased Appetite The patient will generally have no appetite, especially not for a heavy dinner. Even the smell of meal preparation can lead to nausea. Warm drinks and perhaps some easily digestible fruit will be the only nourishment that the sick person will be able to take. The organism needs all its metabolic energy to master the pneumonia. There is a decreased reaction to the stimuli of hunger and thirst and, in this respect, the organism closes itself off from sensory contact with the outside world.
Table 3.5. Characteristic metabolic changes in pneumonia Reaction to stimuli Consolidating tendency Dissolving tendency 3.2.2. Breathing Problems Metabolic changes Decreased appetite The typical lung shape disappears with exudate Infection and fever Tachypnea Tachypnea, accelerated breathing, is the main breathing characteristic of patients with pneumonia. Inhalation is short and not very deep. With the accelerated breathing, the patient will generally also have a considerable tachycardia related to the fever. Tachypnea and tachycardia are related to the intensified metabolic processes that have a formdissolving tendency. Productive Cough Initially, there is a quick and loose cough but very little is coughed up, even when there is a considerable amount of audible mucus in the airways. In health, even a small amount of mucus will lead immediately to a stimulus to cough, which will remove the mucus from the airways. In pneumonia, mucus remains in the airways that does not activate the cough stimulus after the patient stops coughing. The cough in pneumonia becomes effective only in the recovery phase of the disease. The reaction to the cough stimulus is decreased. Bolk’s Companions RespiRatoRy system DisoRDeRs anD theRapy - 41
Page 1 and 2: I N S T I T U T E Respiratory Syste
Page 3: Respiratory System Disorders and Th
Page 6 and 7: Contents Acknowledgments 6 Preface
Page 8 and 9: Acknowledgments This Companion was
Page 10 and 11: 1. Introduction This Companion for
Page 12 and 13: 2. Clinical Signs and Symptoms in P
Page 14 and 15: c. Her muscle tone is increased thr
Page 16 and 17: Conclusion: In the acute asthma att
Page 18 and 19: 2.3. The Third Patient: Acute Airsp
Page 20 and 21: 2.3.2. Pathophysiology of Bacterial
Page 22 and 23: 2.4.1. The Physiological Healing Pr
Page 24 and 25: dissolution. These diversions can l
Page 26 and 27: Table 2.1. Pathophysiological chara
Page 28 and 29: 3. Characterization of Asthma and P
Page 30 and 31: 3.1.2. Breathing Problems The time
Page 32 and 33: Cough Sometimes, a persistent, dry,
Page 34 and 35: Muscle Ache The increased muscle to
Page 36 and 37: 3.1.4. Metabolic Changes Acidosis C
Page 38 and 39: Seasonal Influences The relation be
Page 40 and 41: it were, dynamically poisoned by th
Page 44 and 45: Abrogated Inhalation A patient with
Page 46 and 47: 3.2.4. Changed Awareness Sleepiness
Page 48 and 49: for years, if not a lifetime (fig.2
Page 50 and 51: Just think of the interface between
Page 52 and 53: Jetlag Who does not know the phenom
Page 54 and 55: Dissolving tendency Fig. 3.5. The d
Page 56 and 57: 4. Other Inflammatory Disease of th
Page 58 and 59: 4.3. Hay Fever Hay fever is accompa
Page 60 and 61: Lung fibrosis can be seen as an “
Page 62 and 63: developed acute asthma attacks as a
Page 64 and 65: factors: anxiety to meet certain pe
Page 66 and 67: In asthma, • There is an intensif
Page 68 and 69: from the mesoderm in the yolk sac v
Page 70 and 71: Fig. 5.2. Relation between ventilat
Page 72 and 73: 5.4. A Dynamic Concept of Disease M
Page 74 and 75: complementary medications and thera
Page 76 and 77: Classical, individual homeopathy is
Page 78 and 79: Infection/Injury Acute Fase Reactio
Page 80 and 81: document the different methods of h
Page 82 and 83: them rhythmically. Normally, the in
Page 84 and 85: 6.1.2. Therapy for the Hyperreactiv
Page 86 and 87: we may consider Pulsatilla. Sulphur
Page 88 and 89: If the patient is exhausted, bother
Page 90 and 91: In fig. 6.2., a dynamic diagram sho
Page 92 and 93:
6.2.1. Therapy for the Dissolving D
Page 94 and 95:
c. Treatment of the rising fever by
Page 96 and 97:
d. Excess exudate is secreted in pn
Page 98 and 99:
6.2.3. Therapy for the Decreased Re
Page 100 and 101:
The various treatment modalities th
Page 102 and 103:
Sarcoidosis of the lung is a diseas
Page 104 and 105:
Fig. 7.2. Rhythm in nature: sand an
Page 106 and 107:
already observed in fever. This als
Page 109 and 110:
Appendix Therapeutic Methods The me
Page 111 and 112:
2009; Chernyshov et al 2000; Kienle
Page 113 and 114:
systematically noted and which can
Page 115 and 116:
Study of Medicine. Louis Bolk Insti
Page 117 and 118:
10.1186/1472-6882-7-10. Hamre HJ, W
Page 119 and 120:
Nishijo K, Mori H, Yosikawa K, Yaza
Page 121 and 122:
BOLK’S COMPANIONS FOR THE STUDY O
Page 123 and 124:
The Healing Process I N S T I T U T
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