Respiratory System Disorders and Therapy From a New - Louis Bolk ...

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from the mesoderm in the yolk sac via the liver to the bone marrow. During life, when there is a need, the ability of the liver to contribute to erythropoiesis can be reactivated. This is also possible in regard to the vascularization of different parts of the body. Normally, the peritoneum is not vascularized. In appendicitis, a functional vascular system develops in the peritoneum through vasculoneogenesis. It will disappear again once the peritonitis has healed. In shock, people faint because there is an underfilling of the blood vessels to the brain as a result of the opening of vascular beds elsewhere in the body which draws away a large volume of blood. The infiltrate that is formed in pneumonia (and in other inflammatory processes, such as sinusitis or otitis) results from an increased blood supply and from fluid components and blood cells leaving the blood. There is a functional dislocation in these cases. Another instructive example is thrombosis. The fact that blood can clot is obviously of vital importance for the organism. However, blood clotting in veins that are not injured is a pathological process. Deep venous thrombosis is an example of this. The above examples make it clear that the ability for functional dislocation is a dynamic aspect that makes pathological processes recognizable as a healthy process in the wrong place. The Relation between Aeration and Vascularization of the Lung In the healthy lung, the relation between ventilation and perfusion varies. In the lung apex, aeration dominates vascularization and as a result there is more ventilation than perfusion. In the middle segments, ventilation and perfusion keep each other in balance and, at the base of the lung, perfusion dominates over ventilation. The dynamic approach can identify three areas of function with differentiated relations between ventilation and perfusion in the lung. The lung apex is ‘a bit asthmatic,’ and the lung base is ‘a bit pneumonia-like.’ Shifts within these relations can be described as a ‘dislocation’ in space. In asthma, COPD, and related diseases, the entire lung assumes characteristics of the lung apex (fig.5.3.); in pneumonia and related diseases, it assumes characteristics of the lung base (fig.5.2.).
Apex of the lung: Ventilation >> perfusion Lung middle segments : Ventilation = perfusion Lung lower segments Ventilation
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I N S T I T U T E Respiratory Syste
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Respiratory System Disorders and Th
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Contents Acknowledgments 6 Preface
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Acknowledgments This Companion was
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1. Introduction This Companion for
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2. Clinical Signs and Symptoms in P
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c. Her muscle tone is increased thr
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Conclusion: In the acute asthma att
Page 18 and 19: 2.3. The Third Patient: Acute Airsp
Page 20 and 21: 2.3.2. Pathophysiology of Bacterial
Page 22 and 23: 2.4.1. The Physiological Healing Pr
Page 24 and 25: dissolution. These diversions can l
Page 26 and 27: Table 2.1. Pathophysiological chara
Page 28 and 29: 3. Characterization of Asthma and P
Page 30 and 31: 3.1.2. Breathing Problems The time
Page 32 and 33: Cough Sometimes, a persistent, dry,
Page 34 and 35: Muscle Ache The increased muscle to
Page 36 and 37: 3.1.4. Metabolic Changes Acidosis C
Page 38 and 39: Seasonal Influences The relation be
Page 40 and 41: it were, dynamically poisoned by th
Page 42 and 43: ut is normally different in the liv
Page 44 and 45: Abrogated Inhalation A patient with
Page 46 and 47: 3.2.4. Changed Awareness Sleepiness
Page 48 and 49: for years, if not a lifetime (fig.2
Page 50 and 51: Just think of the interface between
Page 52 and 53: Jetlag Who does not know the phenom
Page 54 and 55: Dissolving tendency Fig. 3.5. The d
Page 56 and 57: 4. Other Inflammatory Disease of th
Page 58 and 59: 4.3. Hay Fever Hay fever is accompa
Page 60 and 61: Lung fibrosis can be seen as an “
Page 62 and 63: developed acute asthma attacks as a
Page 64 and 65: factors: anxiety to meet certain pe
Page 66 and 67: In asthma, • There is an intensif
Page 70 and 71: Fig. 5.2. Relation between ventilat
Page 72 and 73: 5.4. A Dynamic Concept of Disease M
Page 74 and 75: complementary medications and thera
Page 76 and 77: Classical, individual homeopathy is
Page 78 and 79: Infection/Injury Acute Fase Reactio
Page 80 and 81: document the different methods of h
Page 82 and 83: them rhythmically. Normally, the in
Page 84 and 85: 6.1.2. Therapy for the Hyperreactiv
Page 86 and 87: we may consider Pulsatilla. Sulphur
Page 88 and 89: If the patient is exhausted, bother
Page 90 and 91: In fig. 6.2., a dynamic diagram sho
Page 92 and 93: 6.2.1. Therapy for the Dissolving D
Page 94 and 95: c. Treatment of the rising fever by
Page 96 and 97: d. Excess exudate is secreted in pn
Page 98 and 99: 6.2.3. Therapy for the Decreased Re
Page 100 and 101: The various treatment modalities th
Page 102 and 103: Sarcoidosis of the lung is a diseas
Page 104 and 105: Fig. 7.2. Rhythm in nature: sand an
Page 106 and 107: already observed in fever. This als
Page 109 and 110: Appendix Therapeutic Methods The me
Page 111 and 112: 2009; Chernyshov et al 2000; Kienle
Page 113 and 114: systematically noted and which can
Page 115 and 116: Study of Medicine. Louis Bolk Insti
Page 117 and 118: 10.1186/1472-6882-7-10. Hamre HJ, W
Page 119 and 120:
Nishijo K, Mori H, Yosikawa K, Yaza
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BOLK’S COMPANIONS FOR THE STUDY O
Page 123 and 124:
The Healing Process I N S T I T U T
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