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Pell 2008 (Continued)<br />

Outcomes Self-reported <strong>exposure</strong> to SHS as defined by the number of hrs per wk in the home, work, “bars, pubs or clubs”,<br />

“cars, buses or trains”, other public places, other people’s homes and “all locations”.<br />

Number of hospital admissions and relative risk reduction (95% CI) of acute coronary syndrome (ACS) by age,<br />

gender and <strong>smoking</strong> status. Analysis of ACS in men 55 yrs, in women < 65 yrs and > 65 yrs and <strong>for</strong><br />

all patients with ACS. Acute coronary syndrome (ICD-10) defined by a detectable level of cardiac troponin after<br />

emergency admission <strong>for</strong> chest pain.<br />

Self-reported <strong>smoking</strong> status<br />

Biochemical verification: Yes; <strong>smoking</strong> status and <strong>exposure</strong> to SHS as measured by geometric mean serum cotinine<br />

ng/ml.<br />

Notes Non<strong>smoke</strong>rs defined as those with 12ng/ml serum cotinine or less. Limit of detection 0.1ng/ml.<br />

Pell 2009<br />

Methods Country: Scotland<br />

Setting: Scottish Acute Hospital Population<br />

Design: Pre and post-ban tests. Prospective cohort study. Data was collected pre and post ban in Scotlandon consecutive<br />

patients who were non <strong>smoke</strong>rs admitted with ACS to nine Scottish acute hospitals. Follow-up data were<br />

obtained from routine hospital admissions and death databases. Chi 2 tests <strong>for</strong> trend and logistic regression, both<br />

univariate and multivariate.<br />

Participants Consecutive admissions who were non <strong>smoke</strong>rs admitted with ACS to nine Scottish acute hospitals from May 2005<br />

to March 2007<br />

Baseline:1261<br />

Follow up: 30 days post admission<br />

50 had died and 35 had a non fatal MI.<br />

Interventions Smokefree legislation (Smoking, Health and Social Care (Scotland) Bill) implemented on 26 th March, 2006 prohibiting<br />

<strong>smoking</strong> in indoor workplaces, including bars, restaurants and cafes.<br />

Outcomes Cotinine levels. All cause death, cardiovascular death or readmission <strong>for</strong> a principal diagnosis of AMI.<br />

Biochemical verification: Yes. Urinary cotinine measured <strong>exposure</strong> to SHS.<br />

Notes<br />

Sargent 2004<br />

Methods Country: USA<br />

Setting: Helena, Montana<br />

Design: Observational study, cross-sectional surveys, pre and post-ban tests. Analysis: Analysis of admission admitted<br />

<strong>for</strong> AMI <strong>for</strong> people living in and outside Helenausing Poisson analysis.<br />

Participants Total sample: 304 admissions living in and outside Helena<br />

During ban period: 42 admissions<br />

Interventions Local law in place in Helena, Montana from June-Nov 2002 which banned <strong>smoking</strong> in workplaces and public places.<br />

<strong>Legislative</strong> <strong>smoking</strong> <strong>bans</strong> <strong>for</strong> <strong>reducing</strong> <strong>secondhand</strong> <strong>smoke</strong> <strong>exposure</strong>, <strong>smoking</strong> prevalence and tobacco consumption (Review)<br />

Copyright © 2010 The Cochrane Collaboration. Published by John Wiley & Sons, Ltd.<br />

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