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Biochemical pathways through which elevated glucose can formExcessive levels of reactive oxygen species (ROS)
Page 1 and 2: β cell and Hyperglycemia in Type 2
Page 3 and 4: Schema of five stages of progressio
Page 5: GlucotoxicityChronic exposure to hi
Page 9 and 10: Time-dependent disappearance of Maf
Page 11 and 12: The effects of high glucose on the
Page 13: Clinical correlations between gluco
Page 16 and 17: GSH level (mmol/mg)0.380.370.360.35
Page 18 and 19: * p
Page 20 and 21: Lipotoxicity• Acquired cause of i
Page 22 and 23: Prolonged exposure to increased FFA
Page 24 and 25: Antioxidant strategies to protectth
Page 26 and 27: Metformin↓ ACC activityPhosphoryl
Page 28 and 29: • Metformin restores insulin secr
Page 30 and 31: ControlFFAMetforminFFA+MetforminIsl
Page 32 and 33: Fluorescence Intensity654321Intrace
Page 34 and 35: INS-1 Cell704 mM G6016.7 mM G5040*(
Page 36 and 37: GLP-1 Preserved Morphologyof Human
Page 38 and 39: • Antioxidant enzymes :Superoxide
Page 40 and 41: β- cellIL-1βIL-1β receptorAminog
Page 42 and 43: Heme degradation pathwayHemeHeme Ox
Page 44 and 45: 1000 4 mM G16.7 mM G800INS-1 Cell*p
Page 46 and 47: HO-1 : Summary & Conclusion• Heme
Page 48 and 49: Relative risk for T2DM by BMI in wo
Page 50 and 51: Randomized, controlled Type 2 DM pr
Page 52 and 53: Most individuals with increased cir
Page 54 and 55: Fatty acid translocase cluster dete
Page 56 and 57:
High glucose levels increase choles
Page 58 and 59:
• To determine whether prolonged
Page 60 and 61:
Insulin mRNAㅜㅜㅜ% of control
Page 62 and 63:
•These results suggest that the i
Page 64 and 65:
• There are ample evidences at th
Page 66 and 67:
Acknowledgement• Yeungnam Univers
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