CLINICAL HANDBOOK OF SCHIZOPHRENIA

7. Environmental Pre- and Perinatal Influences in Etiology 73REFERENCES AND RECOMMENDED READINGSBoin, F., Zanardini, R., Pioli, R., Altamura, C. A., Maes, M., & Gennarelli, M. (2001). Association between-G308A tumor necrosis factor alpha gene polymorphism and schizophrenia. MolecularPsychiatry, 6(1), 79–82.Brown, A. S., Begg, M. D., Gravenstein, S., Schaefer, C. A., Wyatt, R. J., Bresnahan, M., et al., (2004).Serologic evidence of prenatal influenza in the etiology of schizophrenia. Archives of GeneralPsychiatry, 61(8), 774–780.Brown, A. S., Cohen, P., Harkavy-Friedman, J., Babulas, V., Malaspina, D., Gorman, J. M., et al.(2001). A. E. Bennett Research Award: Prenatal rubella, premorbid abnormalities, and adultschizophrenia. Biological Psychiatry, 49(6), 473–486.Buka, S. L., Tsuang, M. T., Torrey, E. F., Klebanoff, M. A., Bernstein, D., & Yolken, R. H. (2001). Maternalinfections and subsequent psychosis among offspring. Archives of General Psychiatry,58(11), 1032–1037.Buka, S. L., Tsuang, M. T., Torrey, E. F., Klebanoff, M. A., Wagner, R. L., & Yolken, R. H. (2001). Maternalcytokine levels during pregnancy and adult psychosis. Brain, Behavior, and Immunity,15(4), 411–420.Cannon, M., Jones, P. B., & Murray, R. M. (2002). Obstetric complications and schizophrenia: Historicaland meta-analytic review. American Journal of Psychiatry, 159(7), 1080–1092.Cannon, T. D. (1997). On the nature and mechanisms of obstetric influences in schizophrenia: A reviewand synthesis of epidemiologic studies. International Review of Psychiatry, 9, 387–397.Cannon, T. D., Rosso, I. M., Hollister, J. M., Bearden, C. E., Sanchez, L. E., & Hadley, T. (2000). Aprospective cohort study of genetic and perinatal influences in the etiology of schizophrenia.Schizophrenia Bulletin, 26(2), 351–366.Cannon, T. D., van Erp, T. G., Rosso, I. M., Huttunen, M., Lonnqvist, J., Pirkola, T., et al. (2002). Fetalhypoxia and structural brain abnormalities in schizophrenic patients, their siblings, and controls.Archives of General Psychiatry, 59(1), 35–41.Feinberg, I. (1982). Schizophrenia: Caused by a fault in programmed synaptic elimination during adolescence?Journal of Psychiatric Research, 17(4), 319–334.Gilmore, J. H., Jarskog, L. F., Vadlamudi, S., & Lauder, J. M. (2004). Prenatal infection and risk forschizophrenia: IL-1beta, IL-6, and TNFalpha inhibit cortical neuron dendrite development.Neuropsychopharmacology, 29(7), 1221–1229.Lane, E. A., & Albee, G. W. (1966). Comparative birth weights of schizophrenics and their siblings.Journal of Psychology, 64(2), 227–231.McGlashan, T. H., & Hoffman, R. E. (2000). Schizophrenia as a disorder of developmentally reducedsynaptic connectivity. Archives of General Psychiatry, 57, 637–647.Rapoport, J. L., Addington, A. M., Frangou, S., & Psych, M. R. (2005). The neurodevelopmentalmodel of schizophrenia: Update 2005. Molecular Psychiatry, 10(5), 434–449.Torrey, E. F., & Yolken, R. H. (2003). Toxoplasma gondii and schizophrenia. Emerging InfectiousDiseases, 9(11), 1375–1380.Van Erp, T. G., Saleh, P. A., Rosso, I. M., Huttunen, M., Lonnqvist, J., Pirkola, T., et al. (2002). Contributionsof genetic risk and fetal hypoxia to hippocampal volume in patients with schizophreniaor schizoaffective disorder, their unaffected siblings, and healthy unrelated volunteers. AmericanJournal of Psychiatry, 159(9), 1514–1520.