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Causality in Time Series - ClopiNet

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L<strong>in</strong>k<strong>in</strong>g Granger <strong>Causality</strong> and the Pearl Causal Model with Settable Systemsenables us to l<strong>in</strong>k direct structural causality and f<strong>in</strong>ite order G−causality. To state thisexogeneity condition, we write Y 1,t−1 := (Y 1,t−l ,...,Y 1,t−1 ), Y 2,t−1 := (Y 2,t−l ,...,Y 2,t−1 ),and, for given τ 1 ,τ 2 ≥ 0, X t := (X t−τ1 ,..., X t+τ2 ), where X t := (W ′ t ,Z′ t )′ .Assumption A.2 For l and m as <strong>in</strong> A.1 and for τ 1 ≥ m,τ 2 ≥ 0, suppose that Y 2,t−1 ⊥U 1,t | (Y 1,t−1 , X t ), t = 1,...,T − τ 2 .The classical strict exogeneity condition specifies that (Y t−1 , Z t ) ⊥ U 1,t , which impliesY 2,t−1 ⊥ U 1,t | (Y 1,t−1 , Z t ). (Here, W t can be omitted.) Assumption A.2 is a weakerrequirement, as it may hold when strict exogeneity fails. Because of the condition<strong>in</strong>g<strong>in</strong>volved, we call this conditional exogeneity. Chalak, K. and H. White (2010) discussstructural restrictions for canonical settable systems that deliver conditional exogeneity.Below, we also discuss practical tests for this assumption.Because of the f<strong>in</strong>ite numbers of lags <strong>in</strong>volved <strong>in</strong> A.2, this is a f<strong>in</strong>ite-order conditionalexogeneity assumption. For convenience and because no confusion will arisehere, we simply refer to this as “conditional exogeneity."Assumption A.2 ensures that expected direct effects of Y 2,t−1 on Y 1,t are identified.As WL note, it suffices for A.2 that U t−1 ⊥ U 1,t | (Y 0 ,Z t−1 , X t ) and Y 2,t−1 ⊥ (Y 0 ,Z t−τ 1−1 ) |(Y 1,t−1 , X t ). Impos<strong>in</strong>g U t−1 ⊥ U 1,t | (Y 0 ,Z t−1 , X t ) is the analog of requir<strong>in</strong>g that serialcorrelation is absent when lagged dependent variables are present. Impos<strong>in</strong>g Y 2,t−1 ⊥(Y 0 ,Z t−τ 1−1 ) | (Y 1,t−1 , X t ) ensures that ignor<strong>in</strong>g Y 0 and omitt<strong>in</strong>g distant lags of Z t fromX t doesn’t matter.Our first result l<strong>in</strong>k<strong>in</strong>g direct structural causality and G−causality shows that, givenA.1 and A.2 and with proper choice of Q t and S t , G−causality implies direct structuralcausality.Proposition 5.4 Let A.1 and A.2 hold. If Y 2,t−1f<strong>in</strong>ite order G−cause Y 1 with respect to X, i.e.,DS Y 1,t , t = 1,2,..., then Y 2 does notY 1,t ⊥ Y 2,t−1 | Y 1,t−1 , X t , t = 1,...,T − τ 2 .In stat<strong>in</strong>g G non-causality, we make the explicit identifications Q t = Y 2,t−1 and S t = X t .This result leads one to ask whether the converse relation also holds: does directstructural causality imply G−causality? Strictly speak<strong>in</strong>g, the answer is no. WL discussseveral examples. The ma<strong>in</strong> issue is that with suitably chosen causal and probabilisticrelationships, Y 2,t−1 can cause Y 1,t , but Y 2,t−1 and Y 1,t can be <strong>in</strong>dependent, conditionallyor unconditionally, i.e. Granger non-causal.As WL further discuss, however, these examples are exceptional, <strong>in</strong> the sense thatmild perturbations to their structure destroy the Granger non-causality. WL <strong>in</strong>troduce aref<strong>in</strong>ement of the notion of direct structural causality that accommodates these specialcases and that does yield a converse result, permitt<strong>in</strong>g a characterization of structuraland Granger causality. Let supp(Y 1,t ) denote the support of Y 1,t , i.e., the smallest setconta<strong>in</strong><strong>in</strong>g Y 1,t with probability 1, and let F 1,t (· | Y 1,t−1 , X t ) denote the conditional distributionfunction of U 1,t given Y 1,t−1 , X t . WL <strong>in</strong>troduce the follow<strong>in</strong>g def<strong>in</strong>ition:19

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