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Type B insulin resistance "Spare receptor hypothesis" When IR-A titers are relatively low, insulin agonist effectsmay be near maximal with little effect on insulin receptordegradation rate or receptor number, as only a smallfraction of insulin receptors are occupied When IR-A titers are high, binding of IR-A to receptors isnear maximal, resulting in receptor downregulation,decreased receptor number, insulin resistance, andhyperglycemia
Type B insulin resistance Diagnosis The diagnosis of hypoglycemia owing to IR-A requires thedemonstration of IR-A in the serum The immunoprecipitation assay is considered the moresensitive test for detecting IR-A because it will detect IR-Athat bind to the receptor at sites other than the insulinbindingsite IA are usually negative, although low titers may be presentin subjects treated with insulin during a precedinghyperglycemic phase
Page 1 and 2: Autoimmune Hypoglycemia2010. 05. 08
Page 3 and 4: Introduction Hypoglycemia incidence
Page 5 and 6: IntroductionAutoimmune causes Insul
Page 7 and 8: Insulin Autoimmune Syndrome First o
Page 9 and 10: Insulin Autoimmune Syndrome Clinica
Page 11 and 12: Insulin Autoimmune Syndrome Pathoge
Page 13 and 14: Insulin Autoimmune Syndrome Origin
Page 15 and 16: Insulin Autoimmune Syndrome Numerou
Page 17 and 18: Insulin Autoimmune Syndrome Altered
Page 19 and 20: Insulin Autoimmune Syndrome Course
Page 21 and 22: Type B insulin resistance In 1975,
Page 23 and 24: Type B insulin resistance Laborator
Page 25: IR-AType B insulin resistance Polyc
Page 29 and 30: Type B insulin resistance Course an
Page 31: Summary
Page 34: 감 사 합 니 다A
Page 38 and 39: Insulin Autoimmune Syndrome Laborat
Page 40 and 41: Interpretation of values after 72 h
Page 42 and 43: Target autoantigens in type 1 diabe
Page 44: Introduction IA have also been demo

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