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Type B insulin resistance Course and Treatment Hypoglycemia in association with IR-A a high mortality rate owing to progressive autoimmunedisease, malignancy, or hypoglycemia Because of this risk and the severity of the hypoglycemia,aggressive therapy is often warranted High-dose steroid therapy (prednisone, 1 mg/kg/d to 120mg/d) seems to be the most effective therapy Hypoglycemia may respond rapidly to steroid therapy wellbefore any change in IR-A titers is noted
Type B insulin resistance Course and Treatment Some patients remain in remission, whereas others mayrequire chronic therapy Plasmapheresis and immunosuppressive therapy withcyclophosphamide have been employed to reduce IR-Atiters but with less consistent results Plasmapharesis can dramatically lower IR-A titers; however,the effect is transient, and the impact on clinical parametersmay not match the decline in antibody level
Page 1 and 2: Autoimmune Hypoglycemia2010. 05. 08
Page 3 and 4: Introduction Hypoglycemia incidence
Page 5 and 6: IntroductionAutoimmune causes Insul
Page 7 and 8: Insulin Autoimmune Syndrome First o
Page 9 and 10: Insulin Autoimmune Syndrome Clinica
Page 11 and 12: Insulin Autoimmune Syndrome Pathoge
Page 13 and 14: Insulin Autoimmune Syndrome Origin
Page 15 and 16: Insulin Autoimmune Syndrome Numerou
Page 17 and 18: Insulin Autoimmune Syndrome Altered
Page 19 and 20: Insulin Autoimmune Syndrome Course
Page 21 and 22: Type B insulin resistance In 1975,
Page 23 and 24: Type B insulin resistance Laborator
Page 25 and 26: IR-AType B insulin resistance Polyc
Page 27: Type B insulin resistance Diagnosis
Page 31: Summary
Page 34: 감 사 합 니 다A
Page 38 and 39: Insulin Autoimmune Syndrome Laborat
Page 40 and 41: Interpretation of values after 72 h
Page 42 and 43: Target autoantigens in type 1 diabe
Page 44: Introduction IA have also been demo

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