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28<br />

NCKRI Special Paper No. 1<br />

4. Hypogenic cave features<br />

Sedimentary basins and fold-and-thrust geological<br />

structures that experienced a variable degree of uplift and<br />

denudation, and contain carbonate and sulfate formations,<br />

are widespread throughout cratonic and fold-and-thrust<br />

regions. Most of them display features of ascending<br />

hypogene transverse speleogenesis, both relict (in the<br />

entrenched and drained sections) and currently operative<br />

(in confined, deep-seated sections, but also in entrenched<br />

settings). It is argued here that hypogene speleogenesis and<br />

resultant caves are much more widespread than commonly<br />

believed.<br />

Identification and recognition of hypogenic caves is<br />

hindered and often confused due to three main causes.<br />

Firstly, as shown in Section 1.2, there is still considerable<br />

uncertainty in the scope of the hypogenic speleogenesis<br />

concept and in defining the term. Instead of restricting<br />

hypogenic speleogenesis to specific dissolutional<br />

mechanisms (hydrothermal or sulfuric acid), it is suggested<br />

here that hypogenic speleogenesis should be defined from<br />

the hydrogeological perspective, i.e. the formation of caves<br />

by recharge from below in leaky confined conditions by a<br />

number of dissolutional mechanisms that can be involved<br />

in specific cases. This approach is strongly corroborated by<br />

the remarkable similarity in morphological features<br />

exhibited by hypogenic caves formed by different<br />

dissolutional processes in different lithologies (Sections<br />

4.2 and 4.4 below), by the overall regularities in the<br />

hydrostratigraphic occurrence of such caves, and by basin<br />

evolution analysis.<br />

Secondly, the identification of hypogenic caves is<br />

difficult because, by the nature of their origin, they lack<br />

genetic relationships with the overlying or adjacent<br />

surface. They become accessible for direct observations<br />

when occasionally intercepted by denudational lowering,<br />

erosional entrenchment or human activities. However, the<br />

lack of genetic relationships with the surface may serve as<br />

one of the diagnostic criteria for hypogenic caves.<br />

Thirdly, hypogenic caves become accessible being<br />

already relict, largely decoupled from their original caveforming<br />

environment. Features created by epigenic<br />

processes often overprint original hypogenic<br />

morphologies, especially in wet climates, and sediment<br />

accumulations tend to mask important diagnostic features,<br />

especially in floors of caves.<br />

Because of these difficulties, and also due to the still<br />

overwhelming dominance of epigenic concepts and models<br />

in karst science, many caves have been genetically<br />

misinterpreted. Even more known caves with “odd”<br />

patterns, which are not large or outstanding enough to<br />

attract specific scientific attention, remain without clear<br />

speleogenetic interpretation. Examples of regional<br />

speleogenetic analyses well connected with regional<br />

geological (hydrogeological and geomorphological)<br />

evolution are still scarce.<br />

Speleogenetic considerations should be based on the<br />

broad evolutionary approach to karst development, as<br />

described in Chapter 2 and suggested by the classification<br />

of karst types presented in Figure 2. The inherent trend of<br />

basinal evolution during uplift is that deeper, confined,<br />

sections are being brought to the epigenic realm due to<br />

denudation, so that hypogenic caves pass through<br />

transitional conditions of initial breaching and draining,<br />

and get fossilized in the vadose zone. Hypogenic caves<br />

may pass the transient stages without major modification<br />

by the newly established unconfined flow patterns, or they<br />

may be considerably overprinted by epigenic processes.<br />

Where observed caves are in the transitional stages and<br />

overprint is obvious, it is most tempting to relate their<br />

origin to the contemporary epigenic conditions.

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