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Volume 11 Issue 1 (February) - Australasian Society for Ultrasound ...

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ASUM <strong>Ultrasound</strong> Bulletin <strong>February</strong> 2008; <strong>11</strong> (1): 39–43<br />

ASM 2007 ABSTRACTS<br />

Abstracts 37th Annual Scientific Meeting ­<br />

Cairns, 2007: Part 2 – Part 1 published 2007 vol 10 (4)<br />

410 <strong>Ultrasound</strong> of chronic liver disease<br />

Richard B Allan, Flinders Medical Centre, Australia<br />

Objective<br />

To review the use of ultrasound in chronic liver disease.<br />

Chronic liver disease is a common disease and the eighth<br />

leading cause of death in the developed world. Scanning the<br />

liver can be technically difficult due a variety of problems<br />

and the use of small foot print transducers, tissue harmonic<br />

imaging and spatial compounding will all improve image<br />

quality.<br />

<strong>Ultrasound</strong> is relatively insensitive to mild cirrhosis and<br />

there is a poor correlation between the ultrasound appearances<br />

and histological severity. The ultrasound appearances<br />

of cirrhosis are variable but a coarse, heterogeneous echo<br />

pattern is commonly seen. Surface irregularities may be seen<br />

and alteration of the liver architecture due to regenerating<br />

nodules may be present. Appearances may be complicated<br />

by the presence of fatty infiltration. Increased attenuation<br />

and increased echogenicity may be seen if there is an acute<br />

fatty component to liver disease. While fatty liver has variable<br />

appearances these generally fit into easily recognised<br />

patterns.<br />

Common complications of chronic liver disease are<br />

portal hypertension (PHT) and hepatic malignancy. PHT is<br />

a common sequela of chronic liver disease and ultrasound is<br />

used extensively in its diagnosis and monitoring. Of patients,<br />

1–5% will develop HCC and ultrasound is commonly used<br />

as a screening modality.<br />

<strong>Ultrasound</strong> contrast media are used extensively in Europe<br />

and Asia and offer major improvements in the accuracy of<br />

ultrasound. Un<strong>for</strong>tunately, these agents are not available <strong>for</strong><br />

general use in Australia and great opportunities <strong>for</strong> improved<br />

assessment are not being utilised.<br />

4<strong>11</strong> Peyronie’s disease: the long and the short of it<br />

Michelle K Pedretti, Australia<br />

Objective<br />

This presentation will cover the causes, risk factors, signs,<br />

symptoms and treatment variations of Peyronie’s disease.<br />

Sonographic demonstration and appearances will be presented,<br />

although Peyronie’s s disease is generally a clinical<br />

diagnosis.<br />

Cases of Peyronie’s disease have been documented from<br />

as early as 1687. First described by Francois de la Peyronie,<br />

a French surgeon, in 1743. Peyronie’s disease had previously<br />

been classified as a <strong>for</strong>m of sexual incompetence but is now<br />

recognised as a component of erectile dysfunction.<br />

Peyronie’s disease is a benign condition that affects<br />

approximately 1% of the male population. It is most commonly<br />

seen in males between the ages of 45 and 60 years of age. It<br />

can, however, occur in the young and elderly male population.<br />

Thought to have genetic links, its exact nature and etiology<br />

remain uncertain. The condition may spontaneously resolve<br />

in 1–2 years from onset. Surgical intervention is required <strong>for</strong><br />

those in whom the condition does not resolve.<br />

A hard lump, comprised of plaque, in the tunica albuginea<br />

of the penis characterises Peyronie’s disease. The plaque<br />

is thought to originate as local inflammation and develop<br />

into a hardened scar. Plaque development may be on any surface<br />

of the penile shaft, though more commonly is located<br />

on the superior surface of the penis. With penile erection the<br />

plaque causes the penis to bend and can be associated with<br />

pain and distortion.<br />

412 Interventional ultrasound: the basics<br />

Matthew W Andrews, ASUM President, Australia<br />

Objectives<br />

Knowledge of the ultrasound principles and skills underpinning<br />

all ultrasound-guided interventional ultrasound<br />

procedures.<br />

The initial common pathway of all ultrasound-guided<br />

procedures involves the placement of a needle tip into a target<br />

within the body. Once the needle has been placed, a wide<br />

variety of procedures can then be per<strong>for</strong>med.<br />

This talk reviews the current status of the multiple steps<br />

and factors which all contribute to successful needle placement.<br />

Examples of particular procedures will be demonstrated.<br />

The construction of phantoms <strong>for</strong> developing skills<br />

will also be described.<br />

413 Liver Doppler<br />

Richard B Allan, Flinders Medical Centre, Australia<br />

Objective<br />

To review the portal venous system and outline the most<br />

effective methods <strong>for</strong> investigating common abnormalities<br />

with ultrasound.<br />

The liver has complex vascular arrangements and a good<br />

understanding of the anatomy and physiology is essential<br />

when per<strong>for</strong>ming a Doppler ultrasound assessment.<br />

Liver Doppler is a technically challenging examination<br />

due to the difficulties encountered in scanning the diseased<br />

liver and the abnormal flow states often present. Sound<br />

techniques utilising a thorough understanding of the technical<br />

parameters of Doppler ultrasound is necessary to obtain<br />

clinically useful in<strong>for</strong>mation.<br />

The most common pathological processes found in the<br />

portal venous system are portal vein thrombosis (PVT) and<br />

portal hypertension (PHT).<br />

PVT can be accurately diagnosed with ultrasound with a<br />

reported sensitivity of 93% and specificity of 99%.<br />

<strong>Ultrasound</strong> is also used extensively to diagnose PHT<br />

and a number of ultrasound findings have been described<br />

although these have very variable per<strong>for</strong>mances.<br />

While commonly used, both portal vein dilatation and<br />

reversal of flow in the portal vein are relatively poor indicators<br />

of PHT with sensitivities of only 40% and 1–8%<br />

respectively.<br />

The presence of portosystemic collaterals is the most<br />

sensitive indicator of PHT with a sensitivity of 70–83%.<br />

Identification of collaterals is aided by their predictable<br />

positions and clues from flow patterns found in the por-<br />

ASUM <strong>Ultrasound</strong> Bulletin 2008 <strong>February</strong> <strong>11</strong> (1)

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