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Acute Aortic Disease.. - Index of

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36 Isselbacher<br />

deficits are most commonly detected in the arms, and since most type B aortic<br />

dissections arise just distal to the subclavian artery, they are less likely to compromise<br />

arterial flow to the arms. Not surprisingly, pulse deficits occur twice as<br />

<strong>of</strong>ten among those with type A aortic dissection than type B, at 30% versus 15%,<br />

respectively (5). It goes without saying that in order to detect a blood pressure<br />

differential, one must check blood pressure readings in both arms. However, quite<br />

<strong>of</strong>ten in emergency departments blood pressure readings are performed in one<br />

arm only. If one checks the blood pressure in only the arm that has a pulse deficit,<br />

one will obtain a falsely low blood pressure reading; this has <strong>of</strong>ten been referred<br />

to as “pseudohypotension,” and it sometimes leads to incorrect diagnosis or<br />

therapy. In order to avoid being misled in this way, and in order to identify pulse<br />

deficits as valuable clinical clues, blood pressure readings should be taken in both<br />

arms in all patients presenting with chest pain syndromes.<br />

In rare cases arterial supply to both arms is compromised, in which case<br />

comparing the blood pressure readings in the two arms will fail to reveal a<br />

difference, and yet both readings will be falsely low. One should suspect bilateral<br />

pseudohypotension if a patient appears to be well perfused despite low blood<br />

pressure readings in both arms. In such a case, one should check bilateral thigh<br />

blood pressure readings to detect evidence <strong>of</strong> a higher true arterial pressure.<br />

Even when a subclavian artery is completely occluded, in most cases there<br />

is still adequate perfusion to the arm to prevent any overt arm symptoms or signs<br />

at rest. However, occlusion <strong>of</strong> one <strong>of</strong> the common iliac arteries will typically<br />

result in a cold and pulseless lower extremity, which is readily evident on physical<br />

examination.<br />

It should also be noted that in some cases pulse deficits can be transient,<br />

as a result <strong>of</strong> acute decompression <strong>of</strong> the false lumen by distal reentry into the true<br />

lumen or movement <strong>of</strong> the intimal flap away from the occluded orifice.<br />

Other Signs<br />

Patients with aortic dissection may present with a fever, <strong>of</strong>ten accompanied<br />

by leukocytosis and an elevated erythrocyte sedimentation rate; the etiology <strong>of</strong><br />

the fever is uncertain. Rarely, aortic dissection may produce hoarseness, upper<br />

airway obstruction, hemoptysis (due to rupture into the tracheobronchial tree),<br />

hematemesis (due to rupture into the esophagus), a pulsating neck mass, or a<br />

continuous murmur (due to rupture <strong>of</strong> the aorta into the right atrium, the right<br />

ventricle, or the left atrium).<br />

THE GREAT MASQUERADER<br />

Many more patients present with symptoms <strong>of</strong> chest, back, or abdominal pain<br />

than have aortic dissection, and there is significant overlap among the symptoms<br />

and signs <strong>of</strong> aortic dissection and other acute syndromes. Indeed, most patients<br />

with aortic dissection do not present in a “classic fashion,” but rather with only a

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