Muscarinic M1, M3, Nicotinic,GABAA and GABAB Receptor ...

Arison RN, Ciaccio EI, Glitzer MS, Cassaro JA, Pruss MP. (1967). Light and electron
microscopy of lesions in rats rendered diabetic with streptozotocin. Diabetes, 16:
51–56.
Asada H, Kawamura Y, Maruyama K, Kume H, Ding R, Ji FY, Kanbara N, Kuzume
H, Sanbo M, Yagi T, Obata K. (1996). Mice lacking the 65 kDa isoform of
glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and
GABA in their brains but are susceptible to seizures. Biochem Biophys Res
Commun, 229: 891–895.
Ashkenazi A, Winslow JW, Peralta EG. (1987). An M2 muscarinic receptor subtype
couple to both adenylyl cyclase and phosphoinositide turnover. Science, 238:
672–675.
Auer RN, Hugh J, Cosgrove E, Curry B. (1989). Neuropathologic findings in three
cases of profound hypoglycemia. Clin Neuropathol, 8: 63–68.
Auer RN, Kalimo H, Olsson Y, Siesjö BK. (1985). The temporal evolution of
hypoglycemic brain damage. II. Light– and electron–microscopic findings in the
hippocampal gyrus and subiculum of the rat. Acta Neuropathol, 67: 25–36.
Auer RN, Siesjo BK. (1988). Biological differences between ischemia,
hypoglycaemia, and epilepsy. Ann. Neural, 24: 699–707.
Auer RN, Siesjo BK. (1993) Hypoglycaemia: brain neurochemistry and
neuropathology. Baillieres Clin Endocrinol Metab, 7: 611–625.
Auer RN. (2004). Hypoglycaemic brain damage. Forensic Science International, 146:
105–110.