terminology and guidelines for glaucoma ii - Kwaliteitskoepel

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$Refractiechirurgie - Nederlands Oogheelkundig Gezelschap$

2.2.4 - PRIMARY OPEN-ANGLE GLAUCOMA SUSPECT (POAG/HPG SUSPECT) See also Ch. Introduction and Ch. 2.2 Etiology: Unknown Pathomechanism: Unknown Features: Visual field and/or optic disc and/or nerve fiber layer normal or suspicious, with at least one being suspicious Peak IOP > 21 mm Hg < 30 mm Hg without treatment (diurnal tension curve) Gonioscopy: open anterior chamber angle Risk factors to be considered: IOP difference > 4 mm Hg between the two eyes Peak IOP value Any other vascular risk factor for glaucomatous optic neuropathy (See Ch. 2.2) POAG in fellow eye AION in fellow eye Note Pseudoexfoliation and pigment dispersion are risk factors for secondary open-angle glaucoma. High IOP is associated with, but not proven to be a causal factor of vein occlusion, especially in patients with high blood pressure, hypercholesterolemia or obesity. 2.2.5 - PRIMARY OPEN-ANGLE GLAUCOMA/Normal-Pressure Glaucoma (POAG/NPG) See Ch. Introduction and Ch. 2.2 Etiology: Unknown Pathomechanism: Unknown. Optineurin mutation has been found in families with NPG Features: Onset: from the 35th year onwards Signs and symptoms: Asymptomatic until field loss advanced Peak IOP < 22 mm Hg without treatment (diurnal tension curve) Optic nerve head damage typical of glaucoma Disc hemorrhage Visual field defect typical of glaucoma; common paracentral defects Gonioscopy: open anterior chamber angle (exclude intermittent angle-closure; see Ch. 2.4.3) No history or signs of other eye disease or steroid use. Consider central corneal thickness if findings do not match (see Ch. 1.1) 2.2.6 - POAG/Normal-Pressure Glaucoma Suspect (POAG/NPG-SUSPECT) Etiology: Unknown Pathomechanism: Unknown Features: Signs and symptoms: Visual field: normal or suspicious Optic disc and/or nerve fibre layer: findings not diagnostic of, but compatible with, the diagnosis of glaucoma Peak IOP: < 22 mm Hg without treatment (diurnal tension curve or refeated measurements) Gonioscopy: open anterior chamber angle (exclude intermittent angle-closure see Ch. 2.4.3) Risk factors to be considered: IOP difference > 4 mm Hg between the two eyes Any other vascular risk factor for glaucomatous optic neuropathy (See Ch. 2.2) NPG in fellow eye Consider corneal thickness if findings do not match (see Ch. 1.1) Ch. 2 - 7 EGS
2.2.7 - OCULAR HYPERTENSION (OH) Etiology: Unknown Pathomechanism: Unknown Features: Signs and symptoms: Peak IOP > 21 mm Hg without treatment (diurnal curve) Visual field: normal Optic disc and retinal nerve fibre layer: normal Gonioscopy: open anterior chamber angle (exclude intermittent angle-closure. See Ch. 2.4.3) No history or signs of other eye disease or steroid use. Other risk factors: none High IOP is associated with, but not proven to be causal of vein occlusion, especially in patients with high blood pressure, hypercholesterolemia or obesity. Consider corneal thickness if findings do not match. See Ch. 1.1 Although in the past it has been used as a diagnosis and still is usually separated for research and classification purposes, the term ocular hypertension (OH) should be used just to indicate that the IOP is consistenly outside two standard deviations from the normal mean, with all other ocular findings within normal limits. Ch. 2 - 8 EGS
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ISBN: 88-87434-13-1 Editrice DOGMA
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ACKNOWLEDGEMENT This work was made
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2.2.5 - Primary Open-Angle Glaucoma
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Foreword These Definitions and Guid
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INTRODUCTION The aim of the book is
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Since resources are limited worldwi
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II - RANDOMIZED CONTROLLED TRIALS F
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II.3 - COLLABORATIVE NORMAL TENSION
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No stratification for stage of dise
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II. 6. 3 - from CNTG 1. Therapy tha
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References 1) The Ocular Hypertensi
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I - QUESTIONS TO ASK TO YOUR GLAUCO
Page 26 and 27: III - ABNORMAL THRESHOLD VISUAL FIE
Page 28 and 29: V - GONIOSCOPIC OPEN ANGLES SOME DI
Page 30 and 31: VII - TREATMENT STEPLADDER JUVENILE
Page 32 and 33: IX - MONOTHERAPY (in alphabetic ord
Page 34 and 35: XI - MANAGEMENT ACUTE ANGLE CLOSURE
Page 36 and 37: XIII - ANTIMETABOLITES FOR WOUND MO
Page 38 and 39: CHAPTER 1 PATIENT EXAMINATION
Page 40 and 41: Other methods 22-25 : Air-puff tono
Page 42 and 43: References 1) Martin XD. Normal int
Page 44 and 45: 1.2 - GONIOSCOPY Gonioscopy is a fu
Page 46 and 47: 1 2 3 4 Fig. 1 Dynamic indentation
Page 48 and 49: The Spaeth classification Insertion
Page 50 and 51: References 1) Palmberg P. Gonioscop
Page 52 and 53: small - size mid - size large - siz
Page 54 and 55: Fig. 5 A) Cirumlinear vessel, B) Ba
Page 56 and 57: 1.3.4.3 - OCT The Optical Coherence
Page 58 and 59: 31) Carpineto P, Ciancaglini M, Zup
Page 60 and 61: central 24 or 30 central degrees re
Page 62 and 63: A retinal sensitivity value worse t
Page 64 and 65: HODAPP CLASSIFICATION 31 EARLY GLAU
Page 66 and 67: References 1) Bengtsson B, Heijl A,
Page 68 and 69: 1.5 - BLOOD FLOW 1.5.1 - VASCULAR F
Page 70 and 71: 28) Flammer J, Orgul S, Costa VP, O
Page 72 and 73: All forms of glaucoma should be cla
Page 74 and 75: 2.2 - PRIMARY OPEN-ANGLE GLAUCOMAS
Page 78 and 79: 2.3 - SECONDARY OPEN-ANGLE GLAUCOMA
Page 80 and 81: 2.3.1.6 - Glaucoma due to intraocul
Page 82 and 83: 2.4 - PRIMARY ANGLE-CLOSURE Primary
Page 84 and 85: Systemic Risk factors for primary a
Page 86 and 87: OCCLUDABLE ANGLE: “Occludable”
Page 88 and 89: 2.5.3 - SECONDARY ANGLE-CLOSURE GLA
Page 90 and 91: CHAPTER 3 TREATMENT PRINCIPLES and
Page 92 and 93: Furthermore, blindness may occur de
Page 94 and 95: T A R G E T I O P Target IOP range
Page 96 and 97: Practical points for topical medica
Page 98 and 99: 3.3.1.1 - Category: ADRENERGIC AGON
Page 100 and 101: 3.3.1.2 - Category: ADRENERGIC ANTA
Page 102 and 103: 3.3.1.3 - Category: CARBONIC ANHYDR
Page 104 and 105: 3.3.1.4 - Category: PARASYMPATHOMIM
Page 106 and 107: 3.3.1.5 - Category: PROSTAGLANDIN D
Page 108 and 109: Drug interactions Precipitation occ
Page 110 and 111: COMBINATION THERAPY Starting* Addit
Page 112 and 113: 3.3.2.2 - Category: ADRENERGIC ANTA
Page 114 and 115: Pregnancy and nursing mothers Only
Page 116 and 117: 3.5 - LASER SURGERY 3.5.1 - LASER I
Page 118 and 119: 3.5.2 - LASER TRABECULOPLASTY 77-89
Page 120 and 121: 3.5.4 - CYCLOPHOTOCOAGULATION Indic
Page 122 and 123: Although medical therapy is still t
Page 124 and 125: 3.6.4 - COMPLEX CASES Complicated g
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Post-operative use Concentration: 0
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25) Janz NK, Wren PA, Lichter PR, M
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(Rescula TM ) in patients with glau
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in white patients with open-angle g
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CHAPTER 4 TREATMENT GUIDELINES
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4.2 - PRIMARY OPEN-ANGLE GLAUCOMAS
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4.2.7 - OCULAR HYPERTENSION (OH) Al
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4.3.1.7 - Glaucoma associated with
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• Withdrawal of aqueous from vitr
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4.5 - SECONDARY ANGLE-CLOSURE GLAUC
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4.5.3 - SECONDARY ANGLE-CLOSURE GLA
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NOTES
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NOTES
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NOTES
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