Download Booklet - Diabetes in Asia Study Group

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E-mail: drzamanshaikh@yahoo.com Prof. M. Zaman Shaikh, MBBS, FCPS, MRCP, MSc, FRCP Prof. M Zaman Shaikh is Professor of Medicine at Dow University of Health Sciences and the Director at the National Institute of Diabetes and Endocrinology, Karchi, Pakistan. He is also involved with the following professional associations as President, in Heath Times Academy, Vice President of Pakistan Society of Endocrinology, Vice President of Pakistan Society of Physicians and a Member of Editorial Board “Asian Journal of Andrology” published form China. Abdullah H Ben Nakhi MD Kuwait Diabetes Society (KDS) PO Box 68039 71961 Kifan Kuwait Tel +965-4819838 / +965-4819838 Fax +965-4819885 E-mail : kds@kma.org.kw / ahnakhi@yahoo.com Vice president of the Kuwait Diabetes Society Deputy Director General, and consultant diabetologist, Dasman Diabetes Institute, Kuwait Board member of the Gulf Group for the Study of Diabetes Member of the Gulf Collaborative Council Committee (GCC) for Diabetes 26
Prof. Abdul Basit Speakers & Topics Aetio-pathology of Type 2 Diabetes; Genetics vs Environment Gene-environment interactions play a major role in the aetio-pathology of type 2 Diabetes (t2DM). Genetic involvement pattern is complicated as it is a polygenic disorder with multiple genes located on different chromosomes contributing to its susceptibility. thrifty Genotype hypothesis suggested that the presence of gene or genes persist at a high level in the population because they somehow confer a survival advantage in times of adequate or over nutrition. these genes determine increased fat storage. several genes have already been identified as candidate for thrifty genotype like those encoding protein of the insulin signaling and leptin pathways. role of genetics in determining insulin resistance is supported by the findings of decreased insulin activity and hyperinsulinemia among first degree, on-diabetic relatives. some studies also suggests that both the maternal and fetal Glucokinase genotypes interact to influence birth weight. it has also been proposed that a genetically programmed insulin effect during embryogenesis determines fetal growth and provides a possible molecular link between birth weight and susceptibility to t2DM. it is shown that the ability of low birth weight to predict later diabetes in offspring appears to depend on the presence of paternal diabetes. these observation appear to strengthen the case for a genetic link between low birth weight and t2DM. reduced fitness in people with diabetes may also be a genetic tendency. several epidemiological and metabolic studies of twins and first degree relatives of t2DM have demonstrated that diabetes has an important genetic component. the fact that t2DM is a genetically heterogeneous disorder implies that several primary defects contribute to the susceptibility to the disease. environmental factors like high calorie diets and reduced physical activity definitely contributes to the increasing prevalence of obesity and t2DM.the thrifty phenotype hypothesis proposed the concept of environmental ‘programming’ suggesting the existence of developmental windows during which exposures ‘set’ physiological systems and hence long-term consequences. extremes of maternal age is also found to be a contributing factor to low birth weight. the protective effect of breastfeeding for childhood obesity and t2DM has also been observed. high prevalence of the disease in the off springs of gestational diabetic mothers or concordance rates of t2DM in identical twins further suggests intrauterine environmental influences. predictive adaptive response hypothesis proposes that the fetus dynamically interacts and reads the environment which it will be born into and adapts to gain a future survival advantage. environmental pollution and infectivity has also been proposed in certain studies to stimulate the fat cells to secrete molecules that promote insulin resistance, endothelial dysfunction, coagulation disturbances and 27
Page 3 and 4: Diabetes in asia stuDy Group (DasG)
Page 5 and 6: PreFace First up all I am particula
Page 7 and 8: Future perspective in type 2 Diabet
Page 9 and 10: LiSt of frEE PAPErS Papers and Spea
Page 11 and 12: SPEAKErS 11
Page 13 and 14: Prof. Abdul Basit, F.R.C.P (Lon) Pr
Page 15 and 16: Prof. Noorjahan Samad MBBS, D.R.C.O
Page 17 and 18: Prof. Anoop Presently the director
Page 19 and 20: Prof. Edwin Gale Edwin Gale qualifi
Page 21 and 22: Prof. Muhammad Yakoob Ahmedani Prof
Page 23 and 24: Dr. Arun K Baksi Secretary General
Page 25: Program Manager Qatar Diabetes Asso
Page 29 and 30: with increasing diabetes prevalence
Page 31 and 32: With its tendency to have accelerat
Page 33 and 34: ORGANIZING DIABETES CARE IN A RESOU
Page 35 and 36: Dr.Nabil Isseh Diabetes In the Midd
Page 37 and 38: Patient Education in preventing Hyp
Page 39 and 40: Abstract Title Living with Diabetes
Page 41 and 42: Abstract Title INVENTORY OF DIABETE
Page 43 and 44: Abstract Title Knowledge, Skills an
Page 45 and 46: 2) Medical history of the patients:
Page 47 and 48: nephropathy, followed by peripheral
Page 49 and 50: Abstract Title HiGH BLooD GLuCose a
Page 51 and 52: Abstract Title Prevalence figures f
Page 53 and 54: Abstract Title Final Height Achieve
Page 55 and 56: Abstract Title Diabetes Mellitus du
Page 57 and 58: Abstract Title Predictors of Outcom
Page 59 and 60: Abstract Title PROJECT KADAM Knowle
Page 61 and 62: Abstract “Diabetes Care for Schoo
Page 63 and 64: 13:30-14:00 14:00-14:30 14:30-15:00
Page 65 and 66: 12:00-13:00 Secondary prevention: W
Page 67 and 68: DASG Journal Journal of Diabetology

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