FORMAL COMPLAINT - Sweden Confidential
FORMAL COMPLAINT - Sweden Confidential
FORMAL COMPLAINT - Sweden Confidential
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United Nations petition — incomplete report<br />
of ATP (a sedentary, 80 kilos officeholder normally expends 60–70 kg ATP in<br />
24 hours)!<br />
The cellulotoxic properties of Trilafon/Fentazine (perphenazine) adversely<br />
affects most of the 4–7 trillion reactions ordinarily taking place within each cell<br />
every second, and the destructive metabolic impact of this alienist praised morbificum<br />
is no way limited to oxidative subversion of amino acids, carbohydrates<br />
and fatty acids in the tricarboxylic acid cycle....; the overall efficaciousness of<br />
the ―Embden-Meyerhof pathway‖ (the pyruvate produced during the anaerobic<br />
breakdown of glucose [i.e glucolysis] is either converted to acetyl coenzyme A<br />
— and thus enters the tricarboxylic acid cycle — or to lactic acid, ethanol, carbon<br />
dioxide or a few other, organic products) and the ―urea cycle‖* (*this cycle<br />
is linked to the tricarboxylic acid cycle through the synthesis of fumarate**<br />
[**i.e butenedioic acid, HCOOHC:CHCOOH] hydrated to malate and thus<br />
oxidized to oxaloacetate. The oxaloacetate may undergo transamination to<br />
aspartate, be converted into glucose or pyruvate or condensate with acetyl<br />
coenzyme A to form citrate) are also deleteriously impeded!<br />
The poisonous influence of perphenazine will seriously hamper almost every<br />
step of the glycogenesis, glycogenolysis and gluconeogenesis* (*i.e the format-<br />
ion of glucose from noncarbohydrate sources) — multitudinous enzymecatalyzed<br />
reactions are desperately impaired, and the antiporter function of the<br />
transport protein ATP-ADP translocase* (*i.e the adenine nucleotide carrier —<br />
constituting about 15% of the inner mitochondrial membrane protein) as well as<br />
the specialized missions of many other, mitochondrial symporters/antiporters/<br />
carriers etc, consequentially corrupted.<br />
The adenylate kinase dependent hydrolyzation of ATP are curtailed, as perphenazine<br />
has messed up the optimal concentration of Mg 2+ and thus weakened<br />
the capacity of the magnesium ion to form a complex with ATPs terminal phos-<br />
phate groups — reducing the negative charge on the ion thereby becomes ardu-<br />
ous, and the whole substitution reaction are endangered (in addition the general<br />
stability of the carbonyl group in pyruvic acid has been fundamentally perverted,<br />
and later in the glycolysis it‘ll be burdensome for the reactive phosphate ester to<br />
transfer a phosphate group back to the adenosine diphosphate and thus regenerate<br />
ATP).<br />
Animadverted subversion of the ATP purveyance etc will also harm the ATPdriven<br />
―calcium pump‖ — the ―Ca 2+ –ATPase‖ — and a sodium–calcium exchanger<br />
using the Na + gradient across the plasma membrane as the main energy<br />
source (cf the ―Na + –K + pump‖).<br />
The distribution of body fluids is largely determined by osmotic forces, and the<br />
concentration of certain solutes in these fluids is decisive for the osmotic concentration*<br />
(*i.e the ―osmolality‖).<br />
Extracellular fluids (including blood plasma) — containing sodium as their pre-<br />
92<br />
<strong>FORMAL</strong> <strong>COMPLAINT</strong> VS THE KINGDOM OF NORWAY<br />
by Wilh. Werner WINTHER, Norway<br />
92