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FORMAL COMPLAINT - Sweden Confidential

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United Nations petition — incomplete report<br />

of ATP (a sedentary, 80 kilos officeholder normally expends 60–70 kg ATP in<br />

24 hours)!<br />

The cellulotoxic properties of Trilafon/Fentazine (perphenazine) adversely<br />

affects most of the 4–7 trillion reactions ordinarily taking place within each cell<br />

every second, and the destructive metabolic impact of this alienist praised morbificum<br />

is no way limited to oxidative subversion of amino acids, carbohydrates<br />

and fatty acids in the tricarboxylic acid cycle....; the overall efficaciousness of<br />

the ―Embden-Meyerhof pathway‖ (the pyruvate produced during the anaerobic<br />

breakdown of glucose [i.e glucolysis] is either converted to acetyl coenzyme A<br />

— and thus enters the tricarboxylic acid cycle — or to lactic acid, ethanol, carbon<br />

dioxide or a few other, organic products) and the ―urea cycle‖* (*this cycle<br />

is linked to the tricarboxylic acid cycle through the synthesis of fumarate**<br />

[**i.e butenedioic acid, HCOOHC:CHCOOH] hydrated to malate and thus<br />

oxidized to oxaloacetate. The oxaloacetate may undergo transamination to<br />

aspartate, be converted into glucose or pyruvate or condensate with acetyl<br />

coenzyme A to form citrate) are also deleteriously impeded!<br />

The poisonous influence of perphenazine will seriously hamper almost every<br />

step of the glycogenesis, glycogenolysis and gluconeogenesis* (*i.e the format-<br />

ion of glucose from noncarbohydrate sources) — multitudinous enzymecatalyzed<br />

reactions are desperately impaired, and the antiporter function of the<br />

transport protein ATP-ADP translocase* (*i.e the adenine nucleotide carrier —<br />

constituting about 15% of the inner mitochondrial membrane protein) as well as<br />

the specialized missions of many other, mitochondrial symporters/antiporters/<br />

carriers etc, consequentially corrupted.<br />

The adenylate kinase dependent hydrolyzation of ATP are curtailed, as perphenazine<br />

has messed up the optimal concentration of Mg 2+ and thus weakened<br />

the capacity of the magnesium ion to form a complex with ATPs terminal phos-<br />

phate groups — reducing the negative charge on the ion thereby becomes ardu-<br />

ous, and the whole substitution reaction are endangered (in addition the general<br />

stability of the carbonyl group in pyruvic acid has been fundamentally perverted,<br />

and later in the glycolysis it‘ll be burdensome for the reactive phosphate ester to<br />

transfer a phosphate group back to the adenosine diphosphate and thus regenerate<br />

ATP).<br />

Animadverted subversion of the ATP purveyance etc will also harm the ATPdriven<br />

―calcium pump‖ — the ―Ca 2+ –ATPase‖ — and a sodium–calcium exchanger<br />

using the Na + gradient across the plasma membrane as the main energy<br />

source (cf the ―Na + –K + pump‖).<br />

The distribution of body fluids is largely determined by osmotic forces, and the<br />

concentration of certain solutes in these fluids is decisive for the osmotic concentration*<br />

(*i.e the ―osmolality‖).<br />

Extracellular fluids (including blood plasma) — containing sodium as their pre-<br />

92<br />

<strong>FORMAL</strong> <strong>COMPLAINT</strong> VS THE KINGDOM OF NORWAY<br />

by Wilh. Werner WINTHER, Norway<br />

92

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