C:\Eco-SSLs\Contaminant Specific Documents\Lead\November ...

No Dose Fowler, B. A., Kimmel, C. A., Woods, J. S., McConnel, E. E., and Grant, L. D. 1980. Chronic
low-level lead toxicity in the rat. iv. an integrated assessment of long-term toxicity with special
reference to the kidney. Toxicol. Appl. Pharmacol. 56(1): 59-77.
No Dose Fowler, B. A., Kimmel, C. A., Woods, J. S., McConnell, E. E., and Grant, L. D. 1980. Chronic
low-level lead toxicity in the rat. iii. an integrated assessmentof long-term toxicity with special
reference to the kidney. Toxicol. Appl. Pharmacol. 56(1): 59-77.
Rev Fowler, B. A. and Mahaffey, K. R. 1978. Interactions among lead, cadmium, and arsenic in
relation to porphyrin excretion patterns. Environ Health Perspect. 25: 87-90.
No Dose Fox, D. A. 1979. Physiological and neurobehavioral alterations during development in lead
exposed rats. Neurobehav. Toxicol. 1 ((Suppl 1)): 193-206.
No Dose Fox, D. A., Campbell, M. L., and Blocker, Y. S. 1997. Functional alterations and apoptotic cell
death in the retina following developmental or adult lead exposure. Neurotoxicology. 18(3):
645-664.
No Dose Fox, D. A. and Chu, L. W. 1988. Rods are selectively altered by lead: ii. ultrastructure and
quantitative histology. Exp. Eye Res. 46(4): 613-625.
Fate Fox, D. A. and Farber, D. B. 1988. Rods are selectively altered by lead: i. electrophysiology and
biochemistry. Exp Eye Res. 46(4): 597-611.
No Oral Fox, D. A., He, L., Poblenz, A. T., Medrano, C. J., Blocker, Y. S., and Srivastava, D. 1998. Leadinduced
alterations in retinal cgmp phosphodiesterase trigger calcium overload, mitochondrial
dysfunction and rod photoreceptor apoptosis. Toxicol Lett. 102-103: 359-61.
Phys Fox, D. A. and Rubinstein, S. D. 1989. Age-related changes in retinal sensitivity, rhodopsin
content and rod outer segment length in hooded rats following low-level lead exposure during
development. Exp Eye Res. 48(2): 237-49.
In Vit Fox, D. A., Rubinstein, S. D., and Hsu, P. 1991. Developmental lead exposure inhibits adult rat
retinal, but not kidney, na+,k(+)-atpase. Toxicol Appl Pharmacol. 109(3): 482-93.
In Vit Fox, D. A. and Srivastava, D. 1995. Molecular mechanism of the lead-induced inhibition of rod
cgmp phosphodiesterase. Toxicol Lett. 82-83: 263-70.
No Dose Fox, D. A., Srivastava, D., and Hurwitz, R. L. 1994. Lead-induced alterations in rod-mediated
visual functions and cgmp metabolism: new insights. Neurotoxicology 15(3): 503-12.
No Dose Fox, DF, Lewkowski, JP, and Cooper, GP. 1979. Persistent visual cortex excitability alterations
produced by neonatal lead exposure. Neurobehav. Toxic. 1: 101-6.
Surv Franson, J., Christian, I., Schmutz, J. A., Creekmore, L. H., and Fowler, A. C. 1999.
Concentrations of selenium, mercury, and lead in blood of emperor geese. Environ. Toxicol.
Chem. 18(5): 965(5).
Rev Franson J C. 1996. Interpretation of tissue lead residues in birds other than waterfowl. .
Environmental Contaminants in Wildlife: Interpreting Tissue Concentrations. 265-279.
Surv Franson J.C., Augspurger T.P., Converse K.A., Spitzer P.R., Miller E.A., Bachmann R.W., Jones
J.R., Peters R.H., and Soballe, D. M. (eds.). 1995. Necropsy results and concentrations of
Eco-SSL for Lead 114
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