Psychedelic-information-theory-Shamanism-in-the-age-of-Reason

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which facilitates extended dream-like states should also facilitatememory compression, LTP, and neuroplasticity.In programmatic terms the hallucinogenic interrupt can be thoughtof as a back-door or reboot mechanism that allows the subject to enter avisually driven ego programming and debugging matrix; this statewould be similar to hypnosis mixed with an element of lucid dreamingor creative visualization. To stretch the computer metaphor further, inthe absence of hypnotic suggestion or shamanic control, the psychedelicdebugging matrix will naturally drop into a maintenance mode whereanxieties are brought to the fore like a screen-saver programmed tobrowse through repressed salient patterns arising within chaotic noise. 8All of these programmatic metaphors for psychedelics are accurate, andall imply a spontaneous cataloging, compression, or re-organization ofexisting synaptic memory via nonlinear eidetic emotional cues; thisimplies synaptic testing, strengthening, and neuroplasticity.Physiology of Psychedelic NeuroplasticityHallucinogens which target the 5-HT 2A receptor can influencecellular functioning via the activation of G proteins and intracellularsecondary messengers. The signaling pathways mediated by the 5-HT 2Areceptor include the activation of PKC and MAPK, protein kinaseswhich energize enzymes to perform complex cellular maintenance. Theactivation of PKC undoubtedly plays a role in the production andmaintenance of long term memory. Evidence shows that inhibiting PKCactivation in the cortex for as little as a few hours can cause the rapiderasure of long-term memory associations. 11 It is obvious that PKC is afundamental part of memory formation and retention, and it isreasonable to assume that drugs which stimulate PKC activity mayenhance or alter the processes of memory formation, recall, retention,and plasticity.The processes by which PKC mediates memory associations is stillunknown, but the primary assumption is that PKC interacts withdiglycerides (DAG) at the intracellular membrane to mark energeticsignaling areas for receptor formation and synaptic strengthening. Thesecondary signaling cascade goes like this: The 5-HT 2A receptor is
stimulated, activating phospholipase C (PLC) in the cell membrane,which then chops a phospholipid (PIP 2 or PI) at the membrane into anIP 3 group and DAG. The DAG stays near the membrane while IP 3activates the release of Ca 2+ from the endoplasmic reticulum, whichthen activates PKC, which allows PKC to carry energy back to the cellmembrane near the DAG site before activating other enzymes andintracellular substrates. PKC performs its job by moving phosphategroups around the cytoplasm and activating cellular enzymes such asadenosine which forms into AMP, ADP, and ATP by carrying phosphategroups in chains of up to three at once, allowing metabolic energy tomove to other sites throughout the cell. The addition and removal ofphosphates to and from proteins is a fundamental part of all organicmetabolic processes; 5-HT 2A agonists stimulate this phosphorylationprocess through PLC, IP 3, Ca 2+ , and subsequent PKC activation.The fact that 5-HT 2A agonists stimulate PKC and fundamentalmetabolic processes indicates a strong case for psychedelicneuroplasticity. It is interesting to note that Salivinorin A, from Salviadivinorum, also appears activate these same phosphorylation pathwaysthrough the kappa-Opiod receptor. 12 It is also interesting to note that ofall the hallucinogenic compounds that occur in nature, psilocybin(found in magic mushrooms) is the only one that comes with its ownphosphate group (which is stripped to make psilocin), and also appearsto be the weakest tryptamine agonist at the 5-HT 2A receptor. 13 While itis tempting to assert that PKC phosporylation is at the root of allhallucinogenesis and psychedelic effect, it has been demonstrated that5-HT 2A mediated PI hydrolosis is not always a good indicator ofpsychedelic potency. 14 Although there are multiple factors responsiblefor hallucinogenesis, psychedelic stimulation of PKC activityundoubtedly plays a role in perturbing and stimulating persistentmemory functions and promoting potential potent neuroplasticity.Positive and Negative PlasticityShamanic transformation may stimulate neuroplasticity by helpingthe subject realize a more transcendent or spiritually integrated visionof themselves. The logic follows that transformation of the vision of
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PsychedelicInformation TheoryShaman
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Figure 2. Images of internally gene
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Figure 3. The eight-pointed star is
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Chapter 02The Value of PsychedelicI
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Cultural information is considered
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this does not necessarily make bumm
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Chapter 03Psychedelic InformationTh
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of the information in a psychedelic
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Cultural IntegrationBy conservative
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6. Gresch PJ, Smith RL, Barrett RJ,
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Chapter 04What is Consciousness?Sin
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function of recall is to analyze pa
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Modular ConsciousnessThe most drama
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consciousness, meta-consciousness,
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Chapter 05Limits of HumanPerception
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dark-adapted eye utilizes the cone
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Figure 6. The peripheral drift illu
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objects, people, and places in thei
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The Limits of Expanded Consciousnes
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7. Accounts of auras, electromagnet
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Figure 8. Stabilized and Destabiliz
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Constraint, Control, and Feedback I
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phosphenes, or strobing or flickeri
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longer release, and a slightly fast
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Page 58 and 59: least one amine group. The simplest
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Page 64 and 65: Figure 13. Layers of the neocortex.
Page 66 and 67: 5-HT2A receptor mechanicsThe 5-HT 2
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Page 70 and 71: 5-HT2A cross-agonism and organism r
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Page 78 and 79: Figure 16. Spatial-relationship map
Page 80 and 81: Entopic hallucinations fall into pr
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Page 86 and 87: Notes and References1. Williams JD,
Page 88 and 89: Chapter 11Eidetic HallucinationPsyc
Page 90 and 91: Figure 21. Acetylcholine (ACh) modu
Page 92 and 93: described. According to this model,
Page 94 and 95: 4. Accounts of eidetic hallucinatio
Page 96 and 97: Chapter 12Erratic HallucinationHall
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Page 102 and 103: Chapter 13PsychedelicNeuroplasticit
Page 106 and 107: inner self will then reinforce posi
Page 108 and 109: 4. Gresch PJ, Smith RL, Barrett RJ,
Page 110 and 111: Part IIShamanism inthe Age of Reaso
Page 112 and 113: Chapter 14What is Shamanism?The ori
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Page 118 and 119: alien pulsation; a guttural river o
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Page 122 and 123: consciousness unwinds from the recu
Page 124 and 125: Shamanic Information CompressionThe
Page 126 and 127: 3. Observations of shamanic rituals
Page 128 and 129: To apply simple psychedelic therapy
Page 130 and 131: ecently been demonstrated in scient
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Page 134 and 135: Chapter 17Hypnotic Entrainmentand I
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Page 140 and 141: Chapter 18Psychic Bonding and PsiPs
Page 142 and 143: The feedback of pheromonal response
Page 144 and 145: 2. Sheldrake R, 'A New Science of L
Page 146 and 147: testing and error correction allows
Page 148 and 149: group. In the ego-driven beta state
Page 150 and 151: Chapter 20Shamanic SorceryShamanic
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If shamanic sorcery is a kind of no
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ensure they will feel safe to appea
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The All One is subjectively perceiv
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Chapter 22Information Genesisand Co
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generated, and novelty creation in
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navigating the external world is ad
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transition is already underway and
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frequency and ADSR envelope of any
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13. Nonlinear forms are perceived t
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vague and based solely on subjectiv
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or input, but is instead is the res
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Bibliography and ReferencesBibliogr
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Behrendt RP; 'Hallucinations: synch
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Ermentrout, Cowen; 'A mathematical
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Luke D; 'Psi-verts and Psychic Pira
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Siegel JM; 'The stuff dreams are ma
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About this TextAbout PsychedelicInf
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About the AuthorJames L. Kent is a
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Psychedelic-information-theory-Shamanism-in-the-age-of-Reason

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