national-clinical-guidelines-for-stroke-fourth-edition
national-clinical-guidelines-for-stroke-fourth-edition
national-clinical-guidelines-for-stroke-fourth-edition
Create successful ePaper yourself
Turn your PDF publications into a flip-book with our unique Google optimized e-Paper software.
C Consensus<br />
D Consensus; Sato et al 2005<br />
6.10 Impaired tone – spasticity and spasms<br />
There is considerable debate on the definition, physiological nature and importance of<br />
spasticity. Increased tone, abnormal posturing and involuntary spasms may cause<br />
discom<strong>for</strong>t <strong>for</strong> the patient and difficulties <strong>for</strong> carers, and are associated with activity<br />
limitation. Their close association with other impairments of motor control makes it<br />
difficult to determine the extent to which spasticity is a specific cause of disability.<br />
Spasticity is common, especially in the non-functional arm. Estimates of prevalence vary<br />
from 19% at 3 months after <strong>stroke</strong> (Sommerfeld et al 2004) to 38% of patients at 12<br />
months (Watkins et al 2002).<br />
Evidence to recommendations<br />
The spasticity literature contains moderately sized RCTs, two recent larger RCTs<br />
(McCrory et al 2009; Shaw et al 2011) related to botulinum toxin, systematic reviews and<br />
a Cochrane review related to upper limb splinting and stretching. These studies and<br />
reviews show that improvements after botulinum toxin administration are identified in<br />
spasticity (modified Ashworth scale), range of movement and ease of care (also referred<br />
to as passive function). In RCTs, significant group differences have not been<br />
demonstrated in activities per<strong>for</strong>med by the patient, although a small minority of<br />
individuals have reportedly made these changes. Inability to demonstrate improvements<br />
in activity indicates that these changes are unlikely in the majority of patients receiving<br />
botulinum toxin, but may also reflect limitations in some of the measurement tools used.<br />
Improvements in activity <strong>for</strong> lower limb spasticity require further evaluation.<br />
6.10.1 Recommendations<br />
6 Recovery phase from impairments and limited activities: rehabilitation<br />
A Any patient with motor weakness should be assessed <strong>for</strong> the presence of spasticity as a<br />
cause of pain, as a factor limiting activities or care, and as a risk factor <strong>for</strong> the<br />
development of contractures.<br />
B For all the interventions given below, specific goals should be set and monitored using<br />
appropriate <strong>clinical</strong> measures (eg numerical rating scales <strong>for</strong> ease of care (eg Arm<br />
Activity measure (ArmA)) or pain (eg 10-point numerical rating scale), the modified<br />
Ashworth scale, and range of movement).<br />
C In any patient where spasticity is causing concern, the extent of the problem should<br />
be monitored and simple procedures to reduce spasticity should be started. This may<br />
include positioning, active movement and monitoring range of movement <strong>for</strong><br />
deterioration of function, passive movement and pain control.<br />
D Patients with persistent or progressing troublesome focal spasticity affecting one or<br />
two joints and in whom a therapeutic goal can be identified (usually ease of care also<br />
referred to as passive function) should be given intramuscular botulinum toxin. This<br />
should be in the context of a specialist multidisciplinary team service accompanied by<br />
rehabilitation therapy or physical maintenance strategies (eg splinting or casting) over<br />
the next 2–12 weeks following botulinum toxin injection. Functional assessment<br />
© Royal College of Physicians 2012 87