• Childhood weight gain: There is convincing evidence that excess weight gain relative to height duringchildhood is associated with increased risk of being overweight later in life (Grade A; Evidence Report,Section 17.4). 58,241,242Guideline 1• Parental weight: There is convincing evidence that parental overweight or obesity is associated withincreased risk of child overweight or obesity. The risk is greater when both parents (rather than one) areoverweight or obese (Grade A; Evidence Report, Section 17.7). 49-62• Maternal smoking: There is evidence that babies born to mothers who smoke during pregnancy, as anindependent risk factor, probably have a higher risk of becoming overweight or obese in adolescence andadulthood (Grade B; Evidence Report, Section 17.5). 52,54,59-61,243• Television: Recent evidence suggests that hours spent watching television by children is associated withincreased risk of overweight or obesity (Grade C; Evidence Report, Section 17.3). 49,60,64-71,244-246 Media use,including television viewing, may displace time children spend in physical activities 246,247 and eating mealsand snacks in front of the television may also be associated with increased energy intake. 248• Socioeconomic status: There is evidence from developed countries to suggest that a low family incomeor socioeconomic status is associated with increased risk of overweight or obesity during childhood,adolescence and young adulthood (Grade C; Evidence Report, Section 17.9). 49,52,55,64,65,68,72-74 Similarly, theevidence suggests that low socioeconomic status is associated with an increased risk of overweight orobesity (Grade C; Evidence Report, Section 17.10). 59,62,239,249-252• Other factors: Although there were insufficient studies to make an evidence statement, other factorsassociated with increased risk of overweight and obesity throughout life included:––being overweight in adolescence 78––consuming takeaway food and low quality snacks 75-77––childhood smoking 78,83––increased price of fruit and vegetables 68,79––low self-esteem and/or depression 80-82––low locus of control score 84,85––stressful family life 86,87––food insecurity 65,81,253––self-reported dieting, 76,82,254 particularly in girls 255––inadequate sleep 60,244,256,257––low rates of breakfast consumption. 258The literature review to inform the revision of the US dietary guidelines found strong and consistent evidencethat children and adults who eat fast food, particularly those eating at least one fast food meal per week, are atincreased risk of weight gain, overweight and obesity. There was not enough evidence to evaluate any associationbetween eating at other types of restaurants and risk of weight gain, overweight and obesity. 198 The US reviewalso found moderate evidence suggesting that children who do not eat breakfast are at increased risk ofoverweight and obesity, with the evidence being stronger for adolescents. 198The US review also found a limited body of evidence showing conflicting results about whether liquid and solidfoods differ in their effects on energy intake and body weight, except that soup at a meal may lead to decreasedenergy intake and body weight. 198Finally, an emerging body of evidence documents the impact of the obesogenic food environment on body weightin children and adults. Current evidence indicates that the food environment is associated with dietary intake,especially lower consumption of vegetables and fruits, and resulting in higher body weight. 198 This is discussedfurther in Appendix A.Achieve and maintain a healthy weightNational <strong>Health</strong> and Medical Research Council21
Guideline 11.3 How dietary patterns can affect energy balance andweight outcomesEnvironmental and lifestyle factors resulting in overconsumption of energy in the diet and a decrease in physicalactivity are major contributors to the obesity epidemic. A small, persistent energy imbalance is enough tocause excess weight gain in both children and adults, which over time progressively increases the BMI. 135,139Available data from developed countries, including Australia, confirm an increase in energy intake concurrentwith the dramatic increase in the prevalence of overweight and obesity in children and adults since 1980. 139There has been much debate about the role of energy, carbohydrate and fat intake in the obesity epidemic. 236,259,260While reducing excess fat intake has been recognised as an important strategy to reduce energy intake in successfulweight loss interventions, 259 there is little evidence that population fat intake is associated with the obesity epidemicindependently of total energy intake. 260Foods with a higher energy density encourage energy intake above requirements. 198,261 Foods that are high inenergy density also tend to be more palatable, and high palatability is associated with increased food intake insingle-meal studies. 142,262 Fat and sugar are positively associated with energy density while water and dietaryfibre are negatively associated. Fat plays a role because of its high energy density compared to protein andcarbohydrates. Water and dietary fibre play a role through a dilution effect, although this is less for dietary fibrebecause of the much smaller range of fibre concentrations in food compared with both water and fat.It is plausible that the proportions of macronutrients (fat, carbohydrate, protein and alcohol) and types ofmacronutrients comprising total energy intake may affect an individual’s propensity to habitually overconsume.In this regard, dietary patterns that tend to be relatively low in total fat and moderate (not high) in carbohydrateare consistent with reduced risk of excess weight gain. Energy from drinks, in particular, may add to total energyintake without displacing energy consumed in the form of solid food, and it is plausible that sugar-sweeteneddrinks may contribute to excess energy intake through lack of impact on satiety. 216 The satiety value of foods mayalso be important in managing appetite and hunger. 263Energy-dense dietary patterns are associated with higher consumption of grain-based foods, fats and sweetsand lower consumption of vegetables and fruit. 264 International data suggest that the major foods contributing toincreased energy intake include sweetened drinks, snack foods and fast food 261,265,266 and that increasing portionsize is also an important contributor. 198,267,268 The low cost of energy-dense nutrient-poor food relative to nutrientdensefood also has a major role. 261,264The three key lifestyle areas related to overweight and obesity are dietary pattern, physical activity and behaviouralchange. Multicomponent interventions that address all three areas are more effective than those that addressonly one or two. Positive outcomes have been described, at least in the short term, in clinical weight lossregimes that include a range of interventions. 269,270 <strong>For</strong> further information, see the NHMRC Overweight andObesity <strong>Guidelines</strong>. 1211.4 Practical considerations: achieve and maintain a healthy weightIntentional weight loss in overweight and obese individuals reduces the risk factors for mortality and morbidity,and alleviates the symptoms of many chronic conditions. 269,270 It is not necessary to lose large amounts of weightto achieve substantial health gains. <strong>For</strong> example, it is estimated that a weight loss of 5 kg in all people who areoverweight or obese would reduce the national prevalence of type 2 diabetes by 15%. 271 Improving nutritionand/or increasing physical activity also benefits health in areas beyond weight control, such as bone strength,mental health and immune function. 2,272There is a need to provide population guidance on promotion of healthy weight, primary prevention of overweightand obesity, weight maintenance, weight loss, and management of weight-related conditions, disorders anddiseases. A focus on healthy weight is a more positive way to address weight issues than focusing on obesityand overweight. It encourages those who are a healthy weight to maintain that weight. It also helps to reducethe risk of any unintended negative consequences, such as disordered eating. Promotion of healthy weightincorporates prevention and management of underweight, overweight and obesity in children and adults andpromotes healthy growth in children.22EAT FOR HEALTH – australian dietary guidelinesNational <strong>Health</strong> and Medical Research Council
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Bone healthEvidence suggesting an a
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As part of the consultation process
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Expert reviewThe Guidelines underwe
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1995 - The Core Food GroupsThe Core
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Appendix DQuestions for the literat
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Umbrella review questions1. What di
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Appendix ESummary of evidence state
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Evidence statementGradeFruitThe eff
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Limited - no conclusionEvidence is
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Appendix GFood, nutrition and envir
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Figure G1: Examples of environmenta
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G4Australia’s progress toward a s
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Appendix IPhysical activity guideli
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Appendix JStudies examining the hea
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Appendix KAlcohol and energy intake
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Cholesterol: Cholesterol, chemicall
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Limit: Limit is used to emphasise t
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Salt: Dietary salt is an inorganic
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Acronyms and abbreviationsADHDAIDSA
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References1. Rayner M, Scarborough
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37. National Health and Medical Res
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75. Taveras EM, Berkey CS, Rifas-Sh
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112. Olds TS, Tomkinson GR, Ferrar
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153. Rangan AM, Schindeler S, Hecto
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192. Stookey JD, Constant F, Gardne
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231. Hyson DA, Schneeman BO, Davis
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271. Marks GC, Coyne C, Pang G. Typ
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312. Cunningham J, O’Dea K, Dunba
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355. Burns C, Inglis A. Measuring f
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392. Harland JI, Haffner TA. System
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433. Northern Territory Government.
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473. Aune D, Chan DSM, Lau R, Vieir
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512. Ness A, Maynard M, Frankel S,
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550. Vislocky LM, Pikosky MA, Rubin
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588. Sellers TA, Vierkant RA, Djeu
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628. Rosner SA, Åkesson A, Stampfe
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820. Newcomb PA, Nichols HB, Beasle
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862. Australian Institute of Health
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903. Romero-Gutiérrez G, Vaca-Orti
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942. Käferstein F, Abdussalam M. F
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985. Centre for Epidemiology and Re
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1025. Lee A, Bonson A, Yarmirr D, O
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1066. Natural Resource Management M
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1106. Liu Y, Sobue T, Otani T, Tsug
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Notes
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www.nhmrc.gov.auwww.eatforhealth.go