equired to produce health benefits, long-term efficacy, and the relative effect of legume foods, including ofsoy-based foods themselves, as opposed to food components such as isoflavones.Cardiovascular disease, type 2 diabetes and excess weight• Cardiovascular disease: Recent evidence suggests that consumption of soy foods is associated with reducedtotal cholesterol and LDL cholesterol levels, as markers for coronary heart disease risk (Grade C; EvidenceReport, Section 7.4). 392• Type 2 diabetes: No recent studies of the relationship between legumes/beans and type 2 diabetes were identified.• Excess weight: No recent studies of the relationship between legumes/beans and weight loss were identified. 393-395Cancer• Colorectal cancer: Evidence suggests that consuming legumes is associated with reduced risk of colorectalcancer (Grade C; Evidence Report, Section 7.3). 396-400 However, in one study the effect was only significantfor women, 399 as also seen in the recent analysis of the European Prospective Investigation into Cancer andNutrition (EPIC) database. 401 However no evidence of an association between consumption of legumes andcolorectal cancer was described in the WCRF report (see Appendix F). 43Guideline 2• Other cancers: Recent evidence is limited and/or inconclusive for an association regarding legume/beanconsumption and breast or prostate cancer (Evidence Report, Sections 7.1 and 7.2). Similar limited associationshave been described in the WCRF report (see Appendix F). 43 An insufficient number of studies were availableto form an evidence statement on legume/bean consumption and gastric cancer. However the WCRF reportfound limited evidence of a relationship between the consumption of legumes and a decreased risk of gastriccancer (see Appendix F). 43Other conditionsRecent evidence is limited and/or inconclusive regarding an association between consumption of soy foods andbone fracture in post-menopausal women, cerebral and myocardial infarction, and mortality due to cardiovasculardisease and hypertension.2.2.4 The evidence for consuming ‘fruit’Evidence for the health advantages of including fruit in the diet has been strong for decades, but has strengthenedconsiderably recently, particularly for cardiovascular disease. There is also increasing evidence of a protectiveeffect against a number of chronic diseases for consumption of vegetables and fruit when considered together(see Appendix J). Protective effects are increasingly described in quantitative terms, although different serve sizeshave been used in different studies, which make comparison difficult, while findings about dose response are notalways consistent across studies.Table 2.4: Evidence statements for consuming ‘fruit’Evidence statementConsumption of each additional daily serve of fruit is associated with a reduced risk of coronary heart disease.Consumption of at least one and a half serves of fruit a day, ideally two and a half or more, is associated withreduced risk of stroke.Consumption of fruit is associated with a reduced risk of obesity and weight gain.Consumption of fruit is associated with a reduced risk of oral and nasopharyngeal cancerGradeBBCCNotes: Grades – A: convincing association, B: probable association, C: suggestive associationIncludes evidence statements and gradings from the Evidence Report (literature from years 2002–2009). Does not include evidence fromother sources, such as the 2003 edition of the dietary guidelines (in which individual studies were classified according to their design aslevel I, II or III but overall grades for relationships were not derived), although these sources have been used to inform these <strong>Guidelines</strong>.Grade C evidence statements showing no association and all Grade D statements can be found in Appendix E.Enjoy a wide variety of nutritious foodsNational <strong>Health</strong> and Medical Research Council39
The following studies relate primarily to whole fruit, although some included dried fruit and/or fruit juice in theirdefinitions of fruit intake. The evidence regarding fruit juice and excess weight is included under drinks in Section3.3.2. Although serve sizes differed between studies, the evidence statements presented below are based onstandard serve sizes of 150g.Cardiovascular disease, type 2 diabetes and excess weightGuideline 2• Cardiovascular disease: It is probable that consumption of each additional daily serve of fruit is associatedwith a reduced risk of coronary heart disease (Grade B; Evidence Report, Section 1.1). 362-364 Increased protectionof at least 7% was gained from each additional serve of fruit consumed per day. It is probable that consumingat least one and a half serves of fruit a day, ideally two and a half or more, is associated with a reduced risk ofstroke (Grade B; Evidence Report, Section 1.2). 368,369• Type 2 diabetes: The recent evidence suggests that consumption of fruit is not associated with risk of type 2diabetes (Grade C; Evidence Report, Section 1.4). However, as there is a strong relationship between type 2diabetes and body weight (see Chapter 1), the association between consumption of fruit and reduced risk ofexcess weight gain (see below) suggests longer-term studies may be required to investigate potential effects.• Excess weight: The recent body of evidence suggests that consumption of fruit is associated with a reducedrisk of obesity and weight gain (Grade C; Evidence Report, Section 1.3). 200-208Cancer• Alimentary tract cancer: There is emerging evidence that fruit consumption is associated with reduced riskof several types of cancer along the alimentary tract. The recent body of evidence suggests that consumptionof fruit is associated with a reduced risk of oral and nasopharyngeal cancer (Grade C; Evidence Report, Section1.10), 373,374,376,377,402 consistent with findings of a convincing effect on reduced risk of cancers of the mouth, pharynxand larynx and a limited effect on nasopharyngeal cancers described in the WCRF report (see Appendix F). 43• Breast cancer, ovarian cancer and endometrial cancer: Expanding on previous reports, 36 recent evidencenow suggests that consumption of fruit is not associated with risk of breast cancer (Grade C; Evidence Report,Section 1.6), 378,403-407 ovarian cancer (Grade C; Evidence Report, Section 1.11) 384,385 or endometrial cancer (GradeC; Evidence Report, Section 1.12). 386-388,408• Colorectal cancer: Recent evidence suggests that consumption of fruit is not associated with risk of colorectalcancer (Grade C; Evidence Report, Section 1.8). 378,389,402,407,409-411 Further, there is limited evidence to suggest anassociation between the consumption of most fruits by specific type and colorectal cancer (Evidence Report,Section 1.14), 389,411 which expands on earlier studies by the WCRF (see Appendix F). 43• Other cancers: Recent evidence is limited and/or inconclusive for an association regarding fruit consumptionand gastric, lung, oesophageal and pancreatic cancers (Evidence Report, Sections 1.5, 1.7, 1.9 and 1.13).2.2.5 How consuming plenty of vegetables, including different types and colours,and legumes/beans, and fruit may improve health outcomesVarious mechanisms may explain the different health benefits of diets high in vegetables, legumes/beans andfruit. These include potential synergies between the foods as well as the action of specific components foundat high levels in these foods, including vitamins and minerals, various phytochemicals including carotenoids andbioflavonoids (such as anthocyanins and flavonols), as well as dietary fibre and other specific characteristics ofthese foods such as low energy (kilojoules) density.Cardiovascular disease, type 2 diabetes and excess weight mechanismsFood components with anti-oxidant activity including vitamins (vitamin C and E) and phytochemicals in thesefoods may reduce the risk of inflammation and haemostasis, and of cholesterol becoming oxidised and depositedin blood vessels to form the atherogenic plaques that underlie many cardiovascular conditions. 392,412-414 Severalstudies have shown that consumption of vitamin C is associated with reduced risk of cardiovascular disease andstroke, however, other studies have shown no protective effect. 8,415 Plant foods including vegetables and fruitprovide potassium and magnesium, both of which have been linked to lower blood pressure. 8 Importantly, reviewsof the effect of beta-carotene on coronary heart disease suggest that benefits may be related to the componentsof the foods, various antioxidants and micronutrients in these foods or other confounding factors, rather than tothe beta-carotene alone. 41640EAT FOR HEALTH – australian dietary guidelinesNational <strong>Health</strong> and Medical Research Council
- Page 1 and 2: EAT FOR HEALTHAustralianDietaryGuid
- Page 3 and 4: © Commonwealth of Australia 2013Pa
- Page 6: Australian Dietary GuidelinesGuidel
- Page 9 and 10: GUIDELINE 2• Enjoy a wide variety
- Page 11 and 12: guideline 5• Food safety 975.1 Se
- Page 13 and 14: Table A2: Mean daily intakes of ene
- Page 15 and 16: More recent evidence from Western s
- Page 17 and 18: Relationship between the documents
- Page 19 and 20: In this way, the Evidence Report wa
- Page 21 and 22: Challenges for adoption of the Guid
- Page 23 and 24: Australian Guide to Healthy EatingA
- Page 25 and 26: Guideline 11.1 Setting the sceneA h
- Page 27 and 28: Guideline 1UnderweightWhile the gre
- Page 29 and 30: Guideline 1Figure 1.2: Mean energy
- Page 32 and 33: Evidence statementBabies born to mo
- Page 34 and 35: • Childhood weight gain: There is
- Page 36 and 37: Given the scope of the Guidelines,
- Page 38 and 39: Appropriate steady weight gain duri
- Page 40 and 41: Dietary restriction beyond prudent
- Page 42: 1.5 Practice guide for Guideline 1T
- Page 45 and 46: 2.1 Enjoy a wide variety of nutriti
- Page 47 and 48: Pregnant and breastfeeding womenCon
- Page 49 and 50: 2.2 Enjoy plenty of vegetables, inc
- Page 51: • Prostate cancer: The evidence s
- Page 55 and 56: Dietary fibre from vegetables and f
- Page 57 and 58: Children and adolescentsThe recomme
- Page 59 and 60: Cancer• Colorectal cancer: There
- Page 61 and 62: Based on current consumption data,
- Page 63 and 64: disaggregate possibly different eff
- Page 65 and 66: CancerNo recent studies investigati
- Page 67 and 68: Based on most recent consumption da
- Page 69 and 70: 2.5 Enjoy milk, yoghurt, cheese and
- Page 71 and 72: Other conditionsThe traditional nut
- Page 73 and 74: The 1995 National Nutrition Survey
- Page 75 and 76: Reviews have also shown an associat
- Page 77 and 78: 2.7 Practice guide for Guideline 2T
- Page 80 and 81: GUIDELINE 3Limit intake of foods co
- Page 82 and 83: 3.1.2 The evidence for ‘limit int
- Page 84 and 85: 3.1.3 How limiting intake of foods
- Page 86 and 87: InfantsFor infants under the age of
- Page 88 and 89: Bone healthEvidence suggesting an a
- Page 90 and 91: 3.3.2 The evidence for ‘limit int
- Page 92 and 93: 3.3.4 Practical considerations: lim
- Page 94 and 95: 3.4.2 The evidence for ‘limit int
- Page 96 and 97: 3.4.3 How limiting intake of alcoho
- Page 98: 3.5 Practice guide for Guideline 3T
- Page 101 and 102: 4.1 Setting the sceneThe World Heal
- Page 103 and 104:
4.2.1 Breastfeeding incidence and d
- Page 105 and 106:
4.2.3 Cardiovascular disease and ex
- Page 107 and 108:
Lower socioeconomic status mothersW
- Page 109 and 110:
4.4 Practice guide for Guideline 4T
- Page 111 and 112:
5.1 Setting the sceneFoodborne illn
- Page 113 and 114:
5.4.2 InfantsThe immune system of i
- Page 115 and 116:
A1Social distribution of diet-relat
- Page 117 and 118:
• Consumption of milk and milk pr
- Page 119 and 120:
Food intake, diet and nutritional s
- Page 121 and 122:
The prevalence of health risk facto
- Page 123 and 124:
Table B2: Members of the Working Co
- Page 125 and 126:
The evidence was assessed according
- Page 127 and 128:
As part of the consultation process
- Page 129 and 130:
Expert reviewThe Guidelines underwe
- Page 131 and 132:
1995 - The Core Food GroupsThe Core
- Page 133 and 134:
Appendix DQuestions for the literat
- Page 135 and 136:
Umbrella review questions1. What di
- Page 137 and 138:
Appendix ESummary of evidence state
- Page 139 and 140:
Evidence statementGradeFruitThe eff
- Page 141 and 142:
Limited - no conclusionEvidence is
- Page 143 and 144:
Appendix GFood, nutrition and envir
- Page 145 and 146:
Figure G1: Examples of environmenta
- Page 147 and 148:
G4Australia’s progress toward a s
- Page 149 and 150:
It is suggested that by 2011, 125 c
- Page 151 and 152:
Appendix IPhysical activity guideli
- Page 153 and 154:
Appendix JStudies examining the hea
- Page 155 and 156:
Appendix KAlcohol and energy intake
- Page 157 and 158:
Cholesterol: Cholesterol, chemicall
- Page 159 and 160:
Limit: Limit is used to emphasise t
- Page 161 and 162:
Salt: Dietary salt is an inorganic
- Page 164 and 165:
Acronyms and abbreviationsADHDAIDSA
- Page 166 and 167:
References1. Rayner M, Scarborough
- Page 168 and 169:
37. National Health and Medical Res
- Page 170 and 171:
75. Taveras EM, Berkey CS, Rifas-Sh
- Page 172 and 173:
112. Olds TS, Tomkinson GR, Ferrar
- Page 174 and 175:
153. Rangan AM, Schindeler S, Hecto
- Page 176 and 177:
192. Stookey JD, Constant F, Gardne
- Page 178 and 179:
231. Hyson DA, Schneeman BO, Davis
- Page 180 and 181:
271. Marks GC, Coyne C, Pang G. Typ
- Page 182 and 183:
312. Cunningham J, O’Dea K, Dunba
- Page 184 and 185:
355. Burns C, Inglis A. Measuring f
- Page 186 and 187:
392. Harland JI, Haffner TA. System
- Page 188 and 189:
433. Northern Territory Government.
- Page 190 and 191:
473. Aune D, Chan DSM, Lau R, Vieir
- Page 192 and 193:
512. Ness A, Maynard M, Frankel S,
- Page 194 and 195:
550. Vislocky LM, Pikosky MA, Rubin
- Page 196 and 197:
588. Sellers TA, Vierkant RA, Djeu
- Page 198 and 199:
628. Rosner SA, Åkesson A, Stampfe
- Page 200 and 201:
669. Siri-Tarino PW, Sun Q, Hu FB,
- Page 202 and 203:
705. Mamalakis G, Kiriakakis M, Tsi
- Page 204 and 205:
743. Cook NR, Cutler JA, Obarzanek
- Page 206 and 207:
782. Balakrishnan M, Simmonds RS, T
- Page 208 and 209:
820. Newcomb PA, Nichols HB, Beasle
- Page 210 and 211:
862. Australian Institute of Health
- Page 212 and 213:
903. Romero-Gutiérrez G, Vaca-Orti
- Page 214 and 215:
942. Käferstein F, Abdussalam M. F
- Page 216 and 217:
985. Centre for Epidemiology and Re
- Page 218 and 219:
1025. Lee A, Bonson A, Yarmirr D, O
- Page 220 and 221:
1066. Natural Resource Management M
- Page 222 and 223:
1106. Liu Y, Sobue T, Otani T, Tsug
- Page 224 and 225:
Notes
- Page 226:
www.nhmrc.gov.auwww.eatforhealth.go