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Recovery From Schizophrenia: Psychiatry And Political Economy

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NOTES 30333 Heston, L.L., “Psychiatric disorders in foster-home-reared children of schizophrenicmothers,” British Journal of <strong>Psychiatry</strong>, 112:819–25, 1966; Kety, S.S., Rosenthal, D.,Wender, O.H. and Shulsinger, F, “The types and prevalence of mental illness in thebiological and adoptive families of adopted schizophrenics,” in D.Rosenthal andS.S. Kety (eds), The Transmission of <strong>Schizophrenia</strong>, Oxford: Pergamon, 1968, pp. 345et seq.; Kety, S.S., Rosenthal, D., Wender, P.H. et al., “Mental illness in thebiological and adoptive families of adopted individuals who have becomeschizophrenic,” in R.R.Fieve, D.Rosenthal and H.Brill (eds), Genetic Research in<strong>Psychiatry</strong>, Baltimore: Johns Hopkins University Press, 1975, pp. 147 et seq.34 Gottesman, <strong>Schizophrenia</strong> Genesis, p. 103.35 Twin studies of the inheritance of schizophrenia are summarized in Gottesman,<strong>Schizophrenia</strong> Genesis, pp. 105–32.36 Kallman, F.J., “The genetic theory of schizophrenia: An analysis of 691schizophrenic twin index families,” American Journal of <strong>Psychiatry</strong>, 103:309–22, 1946.37 Kringlen, E., “An epidemiological-clinical twin study of schizophrenia,” inRosenthal and Kety (eds), The Transmission of <strong>Schizophrenia</strong>, pp. 49 et seq.38 Straub, R.E., MacLean, C.J., O’Neill F.A. et al., “Support for possibleschizophrenia vulnerability locus in region 5q22–31 in Irish families,” Molecular<strong>Psychiatry</strong>, 2:148–55, 1997; Schwab, S.G., Eckstein, G.N., Hallmayer, J. et al.,“Evidence suggestive of a locus on chromosome 5q31 contributing to susceptibilityof schizophrenia in German and Israeli families by multipoint affected sib-pairlinkage analysis,” Molecular <strong>Psychiatry</strong>, 2:156–60, 1997; Wang, S., Sun, C, Walczak,C.A., et al., “Evidence for a susceptibility locus for schizophrenia on chromosome6pter-p22,” Nature Genetics, 10:41–6, 1995; Freedman, R., Coon, H., Myles-Worsley, M. et al., “Linkage of a neurophysiological deficit in schizophrenia to achromosome 15 locus,” Proceedings of the National Academy of Sciences of the USA, 94:587–92, 1997.39 Meltzer, H.Y. and Stahl, S.M., “The dopamine hypothesis of schizophrenia: Areview,” <strong>Schizophrenia</strong> Bulletin, 2:19–76, 1976; Haracz, J.L., “The dopaminehypothesis: An overview of studies with schizophrenic patients,” <strong>Schizophrenia</strong>Bulletin, 8:438–69, 1982.40 Reynolds, G.P., “Beyond the dopamine hypothesis: The neurochemical pathologyof schizophrenia,” British Journal of <strong>Psychiatry</strong>, 155:305–16, 1989; Wyatt, R.J.,Alexander, R.C., Egan, M.F. and Kirch, D.G., “<strong>Schizophrenia</strong>: Just the facts. Whatdo we know, how well do we know it?” <strong>Schizophrenia</strong> Research, 1:3–18, 1988.41 Hirsch, S.R. and Weinberger, D.R. (eds) <strong>Schizophrenia</strong>, Oxford: Blackwell Science,1995; Seeman, P., “Dopamine receptors: Clinical correlates,” in F.E.Bloom andD.J.Kupfer (eds) Psychopharmacology: The Fourth Generation of Progress, New York:Ravens Press, 1995.42 Averback, P. “Lesions of the nucleus ansa peduncularis in neuropsychiatric disease,”Archives of Neurology, 38:230–5, 1981; Stevens, J.R., “Neuropathology ofschizophrenia,” Archives of General <strong>Psychiatry</strong>, 39:1131–9, 1982; Weinberger andKleinman, “Observations on the brain in schizophrenia,” pp. 52–5.43 Weinberger, D.R., DeLisi, L.E., Perman, G.P. et al., “Computer tomography inschizophreniform disorder and other acute psychiatric disorders,” Archives of General<strong>Psychiatry</strong>, 39:778–83, 1982; Van Horn, J.D. and McManus, I.C. “Ventricularenlargement in schizophrenia: A meta-analysis of studies of the ventricle: brain ratio(VBR),” British Journal of <strong>Psychiatry</strong>, 160:687–97, 1992; Weinberger and Kleinman,

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