C - Michigan Technological University

41. Chowdhury, I., B. Tharakan, and G.K. Bhat, Current concepts in apoptosis: Thephysiological suicide program revisited. Cell Mol Biol Lett, 2006. 11(4): p. 506-25.42. Pagliari, L.J., et al., The multidomain proapoptotic molecules Bax and Bak aredirectly activated by heat. Proc Natl Acad Sci U S A, 2005. 102(50): p. 17975-80.43. Oltvai, Z.N., C.L. Milliman, and S.J. Korsmeyer, Bcl-2 heterodimerizes in vivowith a conserved homolog, Bax, that accelerates programmed cell death. Cell,1993. 74(4): p. 609-19.44. Burlacu, A., Regulation of apoptosis by Bcl-2 family proteins. J Cell Mol Med,2003. 7(3): p. 249-57.45. Fleck, C.C. and H.V. Carey, Modulation of apoptotic pathways in intestinalmucosa during hibernation. Am J Physiol Regul Integr Comp Physiol, 2005.289(2): p. R586-R595.46. Lesauskaite, V., et al., Apoptosis of cardiomyocytes in explanted andtransplanted hearts. Comparison of results from in situ TUNEL, ISEL, and ISOLreactions. Am J Clin Pathol, 2004. 121(1): p. 108-16.47. Hedgecock, N.L., et al., Quantitative regional associations between remodeling,modeling, and osteocyte apoptosis and density in rabbit tibial midshafts. Bone,2007. 40(3): p. 627-37.48. Aguirre, J.I., et al., Osteocyte apoptosis is induced by weightlessness in miceand precedes osteoclast recruitment and bone loss. J Bone Miner Res, 2006.21(4): p. 605-15.49. Bellido, T., et al., Proteasomal degradation of Runx2 shortens parathyroidhormone-induced anti-apoptotic signaling in osteoblasts. A putative explanationfor why intermittent administration is needed for bone anabolism. J Biol Chem,2003. 278(50): p. 50259-72.50. Basso, N. and J.N. Heersche, Effects of hind limb unloading and reloading onnitric oxide synthase expression and apoptosis of osteocytes and chondrocytes.Bone, 2006. 39(4): p. 807-14.51. Basso, N., et al., The effect of reloading on bone volume, osteoblast number, andosteoprogenitor characteristics: studies in hind limb unloaded rats. Bone, 2005.37(3): p. 370-8.52. Ishijima, M., et al., Osteopontin is associated with nuclear factor kappaB geneexpression during tail-suspension-induced bone loss. Exp Cell Res, 2006.312(16): p. 3075-83.53. Sakai, A., et al., Disruption of the p53 gene results in preserved trabecular bonemass and bone formation after mechanical unloading. J Bone Miner Res, 2002.17(1): p. 119-27.139