Insomnia Insomnia
Insomnia Insomnia
Insomnia Insomnia
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Idiopathic <strong>Insomnia</strong> 83<br />
report that these patients slept much less, or required less sleep, than their siblings<br />
when they were infants. Sleep latency is long. Sleep is riddled with many awakenings<br />
and may show sleep-stage abnormalities, such as rapid eye movement (REM)<br />
sleep with few eye movements or ill-formed sleep spindles during stage 2 sleep (5).<br />
Paradoxically, idiopathic insomniacs may show fewer body movements per unit of<br />
sleep than do normal sleepers (14). The spectrum of severity of insomnia in this<br />
condition varies from mild (essentially a light sleeper) to severe and incapacitating,<br />
as the presumed underlying neurological abnormality varies from mild to severe<br />
(14). Daytime features typically include decreased attention and vigilance, low levels<br />
of energy and concentration, and a deterioration of mood commonly described<br />
as grim and subdued, rather than obviously depressed or anxious. In mild or moderate<br />
idiopathic insomnia, psychological functioning is remarkably intact. In severe<br />
cases, daytime functioning may be severely disrupted and affected patients may be<br />
unable to hold a job. During childhood and adolescence, idiopathic insomnia is<br />
often associated with soft neurological signs (i.e., as dyslexia or hyperactivity).<br />
Many cases show diffuse nonspecific abnormalities on the EEG (1).<br />
The ICSD’s diagnostic criteria for idiopathic insomnia are (1) a complaint of<br />
insomnia combined with a complaint of decreased functioning during wakefulness;<br />
(2) long-standing insomnia, typically beginning in early childhood or soon after<br />
birth; (3) relentless insomnia during periods of both poor and good emotional<br />
adjustment; (4) one or more of the following polysomnography: increased sleep<br />
latency, reduced sleep efficiency, and increased number and duration of awakenings<br />
often a reversed first-night effect (best sleep on the first night); (5) the diagnosis<br />
cannot be made if medical or psychiatric disease or stress can explain the early<br />
Case 1<br />
Mr. T. is a 53-year-old man with lifelong history of insomnia. Mr. T. stated<br />
that his mother told him that he had trouble sleeping even during his infancy.<br />
He stated that as far back as he could remember he has had insomnia. Around<br />
age 10 or 11 he started having some anxiety associated with his inability to<br />
sleep. He stated that when he has a decent night’s sleep for several nights in a<br />
row, he really does not feel depressed and anxious; but not getting a good<br />
night’s sleep creates anxiety over the inability to sleep, which then becomes<br />
reinforced when he does not sleep easily the following night.<br />
For reasons unknown, he did not seek medical attention for his problem<br />
until about 1 year ago when he mentioned it to his primary care physician. He<br />
was tried on Xanax, Ambien, Trazodone, Remeron, and over-the-counter<br />
Tylenol PM. Few medications had any impact on his sleep. Even with medication,<br />
he had only one good night every 2–3 weeks, during which he would<br />
sleep 6–7 solid hours.<br />
Most of the time, Mr. T. goes to bed between 11 PM and midnight feeling<br />
sleepy and tired, but when he lays down he is awake for at least 1 hour, some-