Insomnia Insomnia
Insomnia Insomnia
Insomnia Insomnia
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Cognitive-Behavioral Therapy 159<br />
Objective Assessment<br />
In current clinical practice, the diagnosis of primary insomnia does not require<br />
an in-laboratory, polysomnographic study to substantiate the diagnosis. This is true<br />
for three reasons. First, there is enough of a general correspondence between the<br />
subjective complaint and objective measures so that polysomnographic assessment<br />
is not required to verify the sleep continuity disturbance. Second, traditional<br />
polysomnography does not reveal, or allow for the quantification of, the underlying<br />
sleep pathophysiologies that presumably give rise to the patient’s complaints. Third,<br />
and most pragmatically, third-party payers will not reimburse for sleep studies on<br />
patients with likely primary insomnia. However, sleep studies are indicated if the<br />
patient demonstrates symptoms consistent with other intrinsic sleep disorders and/<br />
or fails to respond to treatment.<br />
When assessed with polysomnography, patients with primary insomnia reliably<br />
exhibit increased SL, increased FNAs, increased WASO time, and decreased TST<br />
relative to good sleeper controls. Polysomnographic findings, however, do not<br />
correspond in a one-to-one fashion to patient perceptions of sleep continuity.<br />
Patients with insomnia routinely report more severe sleep disturbance than is evident<br />
on traditional polysomnographic measures (28–30). Some have argued that<br />
this discrepancy might be explained by the findings that patients with primary insomnia<br />
show a greater degree of psychopathology, including tendencies to somatize<br />
internal conflicts and exaggerate symptoms (31–33). Others have argued that the<br />
subjective–objective discrepancy findings reflect a cardinal feature of the disorder,<br />
that is, the persistence of sensory and information processing into NREM sleep. The<br />
continuance of such processes into polysomnographic-defined sleep are thought to be<br />
the basis for patient difficulties distinguishing between wakefulness and sleep (3).<br />
The extent to which one or both of these factors contributes to the discrepancies<br />
between subjective and objective measures of sleep in insomnia continues to be a<br />
matter of ongoing debate. (For additional information on these issues, please see the<br />
following section on the cognitive-behavioral perspective on insomnia.)<br />
When polysomnography is not feasible, the use of alternative, less costly objective<br />
devices can be particularly helpful when the clinician suspects a high degree of<br />
SSM. SSM is a term (as well as disorder) used to describe the common finding<br />
among patients with insomnia that there is a discrepancy between a patient’s subjective<br />
impression of sleep parameters and what is measured via objective recording<br />
methods. At the level of self-report, extreme values (gathered retrospectively or<br />
prospectively) may suggest that this is a component of the disorder (e.g., SLs >2<br />
hours, WASO >2 hours, or a TST of ≤4 hours). In the absence of a<br />
polysomnographic study, actigraphs may be used to obtain corroborating prospective<br />
data. Actigraphs are wristwatch-like devices that utilize sophisticated movement<br />
detectors to estimate the traditional sleep continuity parameters (e.g., SL,<br />
WASO, FNA, and TST). This information may, in turn, be compared to the selfreport<br />
data to assess the degree to which SSM is occurring. The extent to which<br />
subjective–objective discrepancies can be resolved using actigraphy has not been