Insomnia Insomnia

Recommendations

Info

Medical and Neurological Causes 119 Table 1 (continued) Congestive heart failure Myocardial infarction Vasodilating medications Digoxin Diuretics Disorders of the gastrointestinal system Gastroesophageal reflux disorder Gastric and peptic ulcer disease Infectious, inflammatory, allergic intestinal disorders Neoplasia of the gastrointestinal tract Milk allergy/food allergies Urological disorders Prostatic hypertrophy Prostatic carcinoma Prostatitis Interstitial cystitis Bladder infection Nephrolithiasis Neurogenic bladder Chronic renal disease Hemodialysis Rheumatoid/musculoskeletal disorders Rheumatoid arthritis Ankylosing spondylitis Systemic lupus erythematosis Vasculitis Polymyalgia rheumatica Fibromyalgia Pain Chronic fatigue syndrome Osteoarthritis Degenerative change of the spine Scoliosis Scleroderma Endocrine changes Hyperthyroidism Hypothyroidism Adrenal dysfunction Menses Menopause Diabetes mellitus Pregnancy Neoplastic or hematologic disorders Disorders of the nervous system Alzheimer’s dementia Parkinson’s disease 119 (continued)
120 Plotkin Table 1 (continued) Lewy body dementia Progressive supranuclear palsy Multiple systems atrophy Huntington’s disease Fatal Familial <strong>Insomnia</strong> Creutzfeldt-Jakob disease Neuropathy/radiculopathy Muscular dystrophy Inflammatory myopathy Myelopathy Amyotrophic lateral sclerosis Stroke Multiple sclerosis Antoparkinsonian medications Cholinergic agonists Interferon therapy Cerebral mass lesions Cerebral palsy Headache Head injury Encephalitis/meningitis (including Lyme disease and HIV) Epileptic syndromes and bladder dysfunction that contribute to secondary insomnia. Restriction of body movement by severe neuropathy or myelopathy can cause discomfort due to the development of soreness over bony prominences, pressure sores, or arthralgia due to malpositioned extremities. For this reason, insomnia becomes especially prominent with the symptomatic progression of amyotrophic lateral sclerosis, worsening as bulbar involvement becomes prevalent (65). Stroke can produce impaired sleep due to structural changes in the cerebrum (56,66), especially when ischemia has affected the thalamus, anterior hypothalamus, or the central pons (67). A stroke can also produce insomnia by way of its associated physical symptoms, such as movement restriction due to paresis, sensory disruption causing parasthesia/dysesthesia, or impaired pharyngeal function causing aspiration and obstructive breathing. Multiple sclerosis may cause insomnia as demyelinated plaques involve more of the cortical mantle or the diencephalon, and may also be aggravated by motor, sensory, or urological symptoms that increase with the progression of the disease. <strong>Insomnia</strong> has also been reported with various forms of interferon therapy (68). It has been seen with lesions involving the pituitary (69) and pineal gland (70). Cerebral palsy may significantly affect sleep
Page 1 and 2:
Clinical Handbook of Insomnia Edite
Page 3 and 4:
C URRENT CLINICAL NEUROLOGY Daniel
Page 5 and 6:
© 2004 Humana Press Inc. 999 River
Page 8:
Series Editor’s Introduction The
Page 11 and 12:
x Foreword nervous system hypersomn
Page 14 and 15:
Contents Series Editor’s Introduc
Page 16:
Contributors HRAYR P. ATTARIAN, MD
Page 19 and 20:
2 Attarian et al.
Page 21 and 22:
4 Attarian et al. until the 1970s t
Page 23 and 24:
6 Attarian et al. Table 2 Diagnosti
Page 25 and 26:
8 Attarian et al. Duration of Illne
Page 27 and 28:
10 Attarian et al. REFERENCES 1. Ma
Page 29 and 30:
12 Attarian Another study in Austri
Page 31 and 32:
14 Attarian even less well studied.
Page 33 and 34:
16 Attarian communities. A structur
Page 35 and 36:
18 Attarian 5. Sutton, D. A., Moldo
Page 37 and 38:
20 Attarian 53. Kageyama, T., Kabut
Page 39 and 40:
22 Attarian
Page 41 and 42:
24 Bonnet and Arand Table 1 Reporte
Page 43 and 44:
26 Bonnet and Arand On the day spen
Page 45 and 46:
28 Bonnet and Arand SUMMARY These m
Page 47 and 48:
30 Bonnet and Arand chronic caffein
Page 49 and 50:
32 Bonnet and Arand response that a
Page 51 and 52:
34 Bonnet and Arand The Development
Page 53 and 54:
36 Bonnet and Arand ACKNOWLEDGMENT
Page 55 and 56:
38 Bonnet and Arand 45. Bonnet, M.
Page 57 and 58:
40 Attarian Table 1 Types of Insomn
Page 59 and 60:
42 Attarian 15 to 40 seconds. PLMs
Page 61 and 62:
44 Fig. 1. Example of a 1-week slee
Page 63 and 64:
46 Fig. 3. One-week printout of an
Page 65 and 66:
48 Attarian Fig. 4. Algorithm. (Use
Page 67 and 68:
50 Attarian 27. Polo-Kantola, P., E
Page 69 and 70:
52 Garcia
Page 71 and 72:
54 Garcia association is known as s
Page 73 and 74:
56 Fig. 1. A sleep log used in chil
Page 75 and 76:
58 Garcia the reality that the tend
Page 77 and 78:
60 Fig. 2. Actigraph recording in a
Page 79 and 80:
62 Garcia neurodevelopmental disabi
Page 81 and 82:
64 Garcia 35. Czeisler, C., kronaue
Page 83 and 84:
66 Garcia
Page 85 and 86: 68 Attarian showed similar degrees
Page 87 and 88: 70 Attarian agitation and hence the
Page 89 and 90: 72 Attarian Affective disorder is s
Page 91 and 92: 74 Attarian and Drug Administration
Page 93 and 94: 76 Fig. 2. Sleep log after treatmen
Page 95 and 96: 78 Attarian 18. Bonnet, M. H. and A
Page 97 and 98: 80 Attarian 65. Perlis, M., Aloia,
Page 99 and 100: 82 Attarian was confirmed by resear
Page 101 and 102: 84 Attarian times worrying about th
Page 103 and 104: 86 Attarian insomnia or for other s
Page 105 and 106: 88 Attarian
Page 107 and 108: 90 Duntley tion systems found that
Page 109 and 110: 92 Duntley Case 1 A 36-year-old fem
Page 111 and 112: 94 Fig. 2. The objective amount of
Page 113 and 114: 96 Duntley PROGNOSIS AND COMPLICATI
Page 115 and 116: 98 Duntley
Page 117 and 118: 100 Attarian EPIDEMIOLOGY There are
Page 119 and 120: 102 Attarian CLINICAL MANIFESTATION
Page 121 and 122: 104 Attarian DIAGNOSTIC WORKUP The
Page 123 and 124: 106 Attarian 13. Hauri, P. J. (1998
Page 125 and 126: 108 Plotkin
Page 127 and 128: 110 Plotkin quality of sleep. Ultim
Page 129 and 130: 112 Plotkin of the therapeutic effi
Page 131 and 132: 114 Plotkin months or years. The ef
Page 133 and 134: 116 Plotkin associated somatic disc
Page 135: 118 Plotkin Table 1 Causes of Secon
Page 139 and 140: 122 Plotkin REFERENCES 1. Foley, D.
Page 141 and 142: 124 Plotkin 48. Marchetti, F., Rome
Page 143 and 144: 126 Plotkin
Page 145 and 146: 128 Karaz Table 1 DSM-IV Diagnoses
Page 147 and 148: 130 Karaz poor. Focusing only on th
Page 149 and 150: 132 Karaz He felt hopeless that his
Page 151 and 152: 134 Karaz Table 4 Diagnostic Criter
Page 153 and 154: 136 Karaz complained of feeling anx
Page 155 and 156: 138 Karaz other physicians avoided
Page 157 and 158: 140 Karaz 17. Attarian, H. P. (2000
Page 159 and 160: 142 Duntley disorders has been conf
Page 161 and 162: 144 Duntley itance. A recent study
Page 163 and 164: 146 Duntley The patient’s primary
Page 165 and 166: 148 Duntley return to sleep. He tos
Page 167 and 168: 150 Duntley Treatment of RLS should
Page 169 and 170: 152 Duntley 34. Cartwright, R. (197
Page 171 and 172: 154 Perlis et al.
Page 173 and 174: 156 Perlis et al. Fig. 1. A schemat
Page 175 and 176: 158 Perlis et al. ditioned arousal
Page 177 and 178: 160 Perlis et al. subjected to empi
Page 179 and 180: 162 Perlis et al. effect), (3) may
Page 181 and 182: 164 Perlis et al. Cognitive Therapy
Page 183 and 184: 166 Perlis et al. Table 2 Cognitive
Page 185 and 186: 168 Perlis et al. Perhaps more impo
Page 187 and 188:
170 Perlis et al. 28. Carskadon, M.
Page 189 and 190:
172 Perlis et al.
Page 191 and 192:
174 Attarian Secondary Insomnias Re
Page 193 and 194:
176 Attarian 50 years of age who sl
Page 195 and 196:
178 Attarian Antidepressants Becaus
Page 197 and 198:
180 Attarian The use of benzodiazep
Page 199 and 200:
182 Attarian to placebo. Zaleplon a
Page 201 and 202:
184 Attarian 3. Wyatt, R. J., Engel
Page 203 and 204:
186 Attarian residual sedation foll
Page 205 and 206:
188 Index Clonidine, 183 CNS arousa
Page 207 and 208:
190 Index Proton pump inhibitors, 1
Page 209:
Current Clinical Neurology CLINICAL
show all

Insomnia Insomnia

You also want an ePaper? Increase the reach of your titles

Delete template?

Save as template?