Perchè gli animali non sono soggetti ad attacchi cardiaci -Dr. M.Rath

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12 DOCUMENTAZIONE Opie LH. (1979) Review: Role of carnitine in fatty acid metabolism of normal and ischemic myocardium (Recensione: il ruolo della carnitina nel metabolismo degli acidi grassi del miocardio normale e ischemico). American Heart Journal 97: 375-388. Paolisso G, et al. (1993) Pharmacologic doses of vitamin E improve insulin action in healthy subjects and in noninsulin-dependent diabetic patients (Le dosi farmacologiche di vitamina E migliorano l’azione dell’insulina nei soggetti sani e nei pazienti diabetici non insulino-dipendenti). American Journal of Clinical Nutrition 57: 650-656. Paterson JC. (1941) Canadian Medical Association Journal 44:114-120. Some factors in the causation of intimal hemorrhages and in the precipitation of coronary thrombi (Alcuni fattori che causano le emorragie intimali e la precipitazione dei trombi coronarici). Pauling L. (1986) How to Live Longer and Feel Better (Come vivere a lungo e sentirsi meglio). WH Freeman and Company, New York. Pfleger R, Scholl F. (1937) Diabetes und vitamin C (Il diabete e la vitamina C). Wiener Archiv für Innere Medizin 31: 219-230. Psaty BM, Heckbert SR, Koepsell TD, et al. (1995) The risk of myocardial infarction associated with antihypertensive drug therapies (Il rischio di infarto miocardico associato a terapie farmacologiche antiipertensive). Journal of the American Medical Association 274: 620-625. Rath M, Niendorf A, Reblin T, Dietel M, Krebber HJ, Beisiegel U. (1989) Detection and quantification of lipoprotein (a) in the arterial wall of 107 coronary bypass patients (Rilevazione e quantificazione della lipoproteina(a) nella parete arteriosa di 107 pazienti con bypass coronarico). Arteriosclerosis 9: 579-592. Rath M, Pauling L. (1990a) Hypothesis: Lipoprotein (a) is a surrogate for ascorbate (Ipotesi: la lipoproteina(a) è un surrogato dell’ascorbato). Proceedings of the National Academy of Sciences USA 87: 6204-6207. Rath M, Pauling L (1990b) Immunological evidence for the accumulation of lipoprotein (a) in the atherosclerotic lesion of the hypoascorbemic guinea pig (Evidenza immunologica dell’accumulo di lipoproteina(a) nella lesione aterosclerotica della cavia di laboratorio con carenze di ascorbato). Proceedings of the National Academy of Sciences USA 87: 9388- 9390. Rath M, Pauling L. (1991a) Solution to the puzzle of human cardiovascular disease: Its primary cause is ascorbate deficiency, leading to the deposition of lipoprotein (a) and fibrinogen/fibrin in the vascular wall (Soluzione all’enigma della malattia cardiovascolare nell’uomo: la sua 333
PERCHÉ GLI ANIMALI NON SONO SOGGETTI AD ATTACCHI CARDIACI E GLI UOMINI SÌ! causa primaria è la carenza di ascorbato che porta al deposito di lipoproteina(a) e fibrinogeno/fibrina nella parete vascolare). Journal of Orthomolecular Medicine 6: 125-134. Rath M, Pauling L. (1991b) Apoprotein(a) is an adhesive protein (L’apoproteina(a) è una proteina adesiva) . Journal of Orthomolecular Medicine 6: 139-143. Rath M., Pauling L. (1992a) A unified theory of human cardiovascular disease leading the way to the abolition of this disease as a cause for human mortality (Una teoria unificata della malattia cardiovascolare umana verso l’eliminazione di questa malattia come causa di mortalità nell’uomo). Journal of Orthomolecular Medicine 7: 5-15. Rath M, Pauling L. (1992b) Plasmin-induced proteolysis and the role of apoprotein(a), lysine, and synthetic lysine analogs (La proteolisi indotta da plasmina e il ruolo di apolipoproteina(a), lisina e analoghi sintetici della lisina). Journal of Orthomolecular Medicine 7: 17-23. Rath M. (1992c) Lipoprotein-a reduction by ascorbate (Riduzione della lipoproteina-a mediante ascorbato). Journal of Orthomolecular Medicine 7: 81-82. Rath M. (1992d) Solution to the puzzle of human evolution (Soluzione all’enigma dell’evoluzione umana). Journal of Orthomolecular Medicine 7: 73-80. Rath M. (1992e) Reducing the risk for cardiovascular disease with nutritional supplements (La riduzione del rischio di malattia cardiovascolare grazie agli integratori alimentari). Journal of Orthomolecular Medicine 7: 153-162. Rath M. (1993c) A new era in medicine (Una nuova era della medicina). Journal of Orthomolecular Medicine 8: 134-135. Rath M. (1996) The Process of Eradicating Heart Disease Has Become Irreversible (Il processo di debellamento della cardiopatia è ormai irreversibile). Journal of Applied Nutrition 48: 22-33. Rath M., Niedzwiecki A. (1996) Nutritional Supplement Program Halts Progression of Early Coronary Atherosclerosis Documented by Ultrafast Computed Tomography (Il programma di integratori alimentari blocca la progressione dell’aterosclerosi coronarica precoce documentata mediante tomografia computerizzata ultraveloce). Journal of Applied Nutrition. 48: 68-78. Rhoads GG, Dahlen G, Berg K, Morton NE, Dannenberg AL. (1986) Lp(a) Lipoprotein as a risk factor for myocardial infarction (Lipoproteina Lp(a) 334
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Dr. Matthias Rath, M.D. Perché gli
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Tutti gli eserciti del mondo messi
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Prima edizione © 2007 Dr. Rath Edu
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4 Ipertensione 103 Il Programma vit
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8 Dr. Rath aiuta a diminuire i fatt
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Perché gli animali non sono sogget
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Perché gli animali non sono sogget
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PERCHÉ GLI ANIMALI NON SONO SOGGET
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Perchè gli animali non sono soggetti ad attacchi cardiaci -Dr. M.Rath

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