final program.qxd - Parallels Plesk Panel

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Upon 60 minutes contact of BVDV spiked human blood with Whatman FTA cards, the virus was reduced to non-detectable levels in all FTA card samples resulting in virus Log10 reductions greater than 4.09 depending on the lot tested (95% confidence interval was calculated according to the ICH Topic Q5A). Conclusions Whatman FTA cards inactivate human immunodeficiency virus type 1 (HIV-1) and BVDV (model for human Hepatitis C virus) at room temperature and therefore could be safely used for collection, transport, archiving and isolation of nucleic acids from patient samples infected with HIV-1 or human Hepatitis C virus. “ Focusing FIRST on PEOPLE “ 130 w w w . i s h e i d . c o m
PP 2.12 Cellular immune response to Cryptosporidium parvum in Cryptosporidium-HIV co-infected patients Kirti Kaushik, Sumeeta Khurana, Ajay Wanchu*, Nancy Malla Kirti Kaushik, Sumeeta Khurana, Ajay Wanchu*, Nancy Malla Dept.of Parasitology and Internal Medicine*, PGIMER, Chandigarh, India Acquired Immuno Deficiency Syndrome (AIDS) is a global emergency with far reaching effects. The gastrointestinal tract is a major target organ in HIV related opportunistic infections, which persists, commonly as diarrhoea. Cryptosporidia are increasingly being recognized as important enteric pathogens in AIDS and other immuno-suppressed patients. The host immune responses that prevent the initial infection by Cryptosporidium, limit its spread and ultimately facilitate its clearance are poorly understood .Since data on immune response to Cryptosporidia in HIV patients co-infected with Cryptosporidium are lacking, the present study was designed to assess the cellular immune responses in the HIV patients infected with Cryptosporidia. For this PBMCs separated from the patients were proliferated in the presence of Cryptosporidium parvum crude soluble antigen (CCA) and proliferation was measured by counting the thymidine incorporation into the cells. PBMCs from the Cryptosporidium infected patients (both HIV infected and non-infected) showed high proliferation against CCA as compared to Cryptosporidium non- infected subjects. POSTERS “ Focusing FIRST on PEOPLE “ 131 w w w . i s h e i d . c o m
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EDITORIAL Dear Madam, Dear Sir, It
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PRACTICAL INFORMATION - INFORMATION
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PROGRAM AT A GLANCE “ Focusing FI
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WEDNESDAY JUNE 21, 2006 - MERCREDI
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WEDNESDAY JUNE 21, 2006 - MERCREDI
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THURSDAY JUNE 22, 2006 - JEUDI 22 J
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THURSDAY JUNE 22, 2006 - JEUDI 22 J
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FRIDAY JUNE 23, 2006 - VENDREDI 23
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POSTERS PRESENTATIONS - PRESENTATIO
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The hospital reform in progress may
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OP 1.3 Public Health and Social Sci
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OP 2.1 Research Priorities in HIV :
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OP 2.3 Research Priorities In HIV I
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OP 3.2 Interactions between HSV and
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OP 3.4 Fertility options in HIV-inf
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OP 3.5 Non-AIDS defining cancers in
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OP 4.3 Update on TMC114 - Actualit
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OP 4.5 Recent data on PA-457 Matura
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OP 5.3 Immunology - Summary of adva
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OP 6.2 Molecular Epidemiology of HI
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In 1995 HIV started to spread rapid
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Given the high HIV prevalence in ID
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OP 6.4 The Epidemiology of HIV & ST
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OP 7.3.1 We must switch early patie
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OP 7.4.2 Entry inhibitors have to b
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OP 8.2 West Nile Virus Infection in
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First, the origin of primary introd
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To reduce the amount of virus that
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OP 9.1 New Developments in the Trea
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OP 9.3 Future options in non respon
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the consensus interferon dose (25,2
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interferon maintenance therapy in d
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26) Kaiser S, Hass HG, Gregor M. Su
Page 80 and 81: OP 10.1 Progress in Microbicide Dev
Page 82 and 83: OP 10.3 Current Advances in HIV Vac
Page 84 and 85: dysmetabolic, functional, or organi
Page 86 and 87: Orphans Forty-three percent of the
Page 88 and 89: PP 1.4 Pradip Timilsena, Laxmi Pras
Page 90 and 91: PP 1.6 Epidemiological aspects of M
Page 92 and 93: PP 1.8 HIV transmission in The Gamb
Page 94 and 95: PP 1.10 Evaluation of Prevalence an
Page 96 and 97: PP 1.12 Co-morbidity of HIV, Hepati
Page 98 and 99: PP 1.14 Screening for HIV-infection
Page 100 and 101: PP 1.16 Low performance of HIV-1 se
Page 102 and 103: PP 1.18 The spread of HIV-1 resista
Page 104 and 105: PP 1.20 Study of the anti-HIV recom
Page 106 and 107: PP 1.22 Comparative investigation o
Page 108 and 109: PP 1.24 Foreign Citizens Admitted t
Page 110 and 111: PP 1.26 Morbidity pattern of PLWA r
Page 112 and 113: PP 1.28 Incidence of Atypical Mycob
Page 114 and 115: PP 1.29 Developing Partnerships Bet
Page 116 and 117: PP 1.31 Pakistani Youth and their R
Page 118 and 119: PP 1.33 Model based on a quantum al
Page 120 and 121: PP 2.2 Analysis of Full-length HIV-
Page 122 and 123: PP 2.4 The Cobas Ampliprep/Cobas Ta
Page 124 and 125: PP 2.6 Mutations and Polymorphisms
Page 126 and 127: PP 2.8 Differences in the phenotypi
Page 128 and 129: PP 2.10 Biochemical Characterizatio
Page 132 and 133: PP 2.13 Assessing the Contribution
Page 134 and 135: PP 2.15 Correlation between circula
Page 136 and 137: PP 2.17 Interface targeting peptide
Page 138 and 139: PP 2.19 HIV-1 gene expression: less
Page 140 and 141: PP 2.21 Vesical Cancer and Papillom
Page 142 and 143: PP 3.2 Is Age a Complicating Factor
Page 144 and 145: PP 3.4 Reversible HIV associated en
Page 146 and 147: PP 3.6 Mycobacterium Ulcerans Cutan
Page 148 and 149: PP 3.8 A Clinical Study Of 60 Patie
Page 150 and 151: PP 3.9 HIV and Tuberculosis: Partne
Page 152 and 153: PP 3.11 Clinicopathological compari
Page 154 and 155: PP 3.13 Crohn's disease onset in a
Page 156 and 157: PP 3.15 Ocular manifestations occur
Page 158 and 159: PP 3.17 Bladder carcinoma observed
Page 160 and 161: PP 3.19 Increasing concerns related
Page 162 and 163: PP 3.21 Glucose intolerance and ins
Page 164 and 165: PP 3.23 Proviral DNA and plasma vir
Page 166 and 167: PP 3.25 Frequency, monitoring, clin
Page 168 and 169: PP 3.27 HIV-positive cases as part
Page 170 and 171: PP 3.29 The Effects of a Supervised
Page 172 and 173: PP 3.31 Prevalence of depression am
Page 174 and 175: PP 3.33 Rhinopharyngeal carcinoma w
Page 176 and 177: PP 3.35 Large vessel damage due to
Page 178 and 179: PP 3.37 No Interference between Syp
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PP 3.39 Faecal flora, diarrhoea ass
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PP 4.2 Triple therapy adherence in
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In a hierarchical multiple regressi
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PP 4.5 A Prospective Study of Antir
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PP 4.7 Telephone-Based Coping Impro
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PP 4.9 The significantly different
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PP 4.11 Self-Evaluation Investigati
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PP 4.13 Treatment of Kaposi's sarco
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PP 4.15 Pharmacokinetic Interaction
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PP 4.17 Human immunodeficiency viru
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PP 4.19 Much More Sure than 2 Years
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PP 4.21 No evidence of reduced viro
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PP 4.23 The increased liver toxicit
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PP 4.25 Rapid Oral Desensitization
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PP 5.1 Rational design of HCV antig
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PP 5.3 Immune restoration levels un
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PP 5.5 Fulminant Candida albicans p
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PP 5.7 Chronic hepatitic C-prompted
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PP 5.9 Chronic HBV infection associ
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PP 6.2 Broadly protective immunity
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PP 6.4 Chikungunya arthritis sequel
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PP 6.5 Experimental determination o
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PP 6.7 Emerging Arboviruses in Turi
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PP 6.9 Ocular LOA-LOA Infection in
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PP 6.11 Prevalence of Carriers of M
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PP 6.13 Evaluation of the Prevalenc
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PP 6.15 Assessment of need of Patie
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PP 6.17 Community-acquired septicem
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PP 6.19 Multiple sclerosis managed
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PP 6.21 A serious staphylococcal kn
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PP 6.23 Comparison between Secretor
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PP 6.25 Real- Time PCR and Flow Cyt
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FP 0.2 Preclinical Development of a
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FP 0.4 Primary Resistance of a Nove
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FP 1.1 In Vivo Emergence of RANTES-
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Conclusion:Patients with HIV-1 infe
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FP 1.4 V1V2 loop length variation d
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FP 1.6 Analysis of plasma cytokines
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FP 1.8 Detection of low frequency H
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FP 1.10 Replication-Competent Platf
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FP 2.1 Long-term non-progression in
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FP 2.3 HAART in Sub-Saharan countri
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FP 2.5 Morbidity and mortality by b
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FP 2.6 Tolerability of antiretrovir
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FP 2.8 Radata - An internet-based s
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FP 3.2 Glycosylated recombinant sim
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FP 3.4 Therapeutic Tat-Vaccination
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PL 2 HIV & AIDS Research: The Light
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SS 1.1 The importance of HIV drug r
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SS 1.3 Virtual phenotype and Clinic
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SS 2.1 The Lessons we have Learned
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SS 5.2 HIV-associated facial lipoat
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SS 6.1 How to improve the use of ne
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SS 6.3 Tipranavir is a Potent Prote
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ABBOTT FRANCE Abbott is a global, b
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BOEHRINGER INGELHEIM The Boehringer
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IVAGEN IVAGEN SA, a biotechnology c
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ROCHE Headquartered in Basel, Switz
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Putting knowledge into practice VIR
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NOTES “ Focusing FIRST on PEOPLE
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