final program.qxd - Parallels Plesk Panel

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PP 3.21 Glucose intolerance and insulin-resistance found in HIV-infected subjects during their anti-HIV protease inhibitor treatment: a prospective, randomized comparison study of three oral hypoglycemic drugs Leonardo Calza, Roberto Manfredi Infectious Diseases, University of Bologna, Italy Introduction HAART-related dysmetabolic alterations recently emerged in their frequency and clinical correlates. When considering protease inhibitor (PI)-treated patients (p) a high prevalence (30-80%) of insulin-resistance and hyperinsulinemia have been found, versus a lower (
PP 3.22 Multiple subcutaneous lipomatosis prospectically observed in patients receiving highly active antiretroviral therapy (HAART). Possible links with concurrent metabolic abnormalities, and pathogenetic insights Roberto Manfredi, Leonardo Calza Infectious Diseases, University of Bologna, Italy Introduction As a consequence of the availability of highly active antiretroviral therapy (HAART), the lipodystrophy syndrome and a broad spectrum of metabolic abnormalities progressively emerged in the last decade (1997-2006). Localized fat accumulation may occur as visceral adipositiy, increased breast size, gynecomastia, lipomastia, and the so-called buffalo hump, although very limited reports are to date available on the role of lipomatosis during HIV infection. Patienys and Methods Nineteen patients (p) out of over 1,000 treated with HAART as of end of December, 2005 (>1.5%) (15 males and four females, aged 36-58 years), experienced multiple ultrasonography-confirmed subcutaneous lipomas (three to over 20 lesions), predominantly involving the trunk and upper and lower limbs, usually associated with local discomfort. Results Among involved p, the duration of HIV seropositivity at lipomatosis onset varied between 32-116 months, and no p had developed full-blown AIDS. Our 19 p experienced 5-14 different anti-HIV therapeutic lines: almost all available protease inhibitors (PI) and nucleoside analogues had been used previously or during the occurrence of lipomatosis, while a NNRTI was used by in five p only. All p were given a PI-based HAART since 16-74 months (mean 28.2±13.9 months). While the virological-immunological situation of HIV disease remained favorable, a broad spectrum of concurrent lipodistrophy syndrome- and dysmetabolic-related alterations were found. In detail, an evident lipoatrophy was present in 15 p out of 19, associated with central adiposity in 12 p. Hypertriglyceridemia, hypercholesterolemia, and hyperglycemia were detected at in 15, 10, and three p, respectively. The subsequent follow-up (8-70 months), was characterized by the occurrence of further lesions in 10 p (with five p undergoing plastic surgery with satisfactory results), and a substantially stable disease in the remaining 9 p, in absence of spontaneous regression, even when the initial HAART regimen was changed. POSTERS Conclusions Sucutaneous lipomas have not been reported with increased frequency during HIV infection, including the HAART era. An accurate diagnostic workup (ultrasonography and eventually fine-needle biopsy) is needed to prevent the rare occurrence of malignant degeneration. The frequent association of lipomatosis with other clinical-metabolic disturbances related to HAART, should deserve further epidemiological and pathogenetic studies, to better investigate their eventual, mutual relationship, and to identify and plan eventual prevention strategies. “ Focusing FIRST on PEOPLE “ 163 w w w . i s h e i d . c o m
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EDITORIAL Dear Madam, Dear Sir, It
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PRACTICAL INFORMATION - INFORMATION
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PROGRAM AT A GLANCE “ Focusing FI
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WEDNESDAY JUNE 21, 2006 - MERCREDI
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WEDNESDAY JUNE 21, 2006 - MERCREDI
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THURSDAY JUNE 22, 2006 - JEUDI 22 J
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THURSDAY JUNE 22, 2006 - JEUDI 22 J
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FRIDAY JUNE 23, 2006 - VENDREDI 23
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POSTERS PRESENTATIONS - PRESENTATIO
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The hospital reform in progress may
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OP 1.3 Public Health and Social Sci
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OP 2.1 Research Priorities in HIV :
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OP 2.3 Research Priorities In HIV I
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OP 3.2 Interactions between HSV and
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OP 3.4 Fertility options in HIV-inf
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OP 3.5 Non-AIDS defining cancers in
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OP 4.3 Update on TMC114 - Actualit
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OP 4.5 Recent data on PA-457 Matura
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OP 5.3 Immunology - Summary of adva
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OP 6.2 Molecular Epidemiology of HI
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In 1995 HIV started to spread rapid
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Given the high HIV prevalence in ID
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OP 6.4 The Epidemiology of HIV & ST
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OP 7.3.1 We must switch early patie
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OP 7.4.2 Entry inhibitors have to b
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OP 8.2 West Nile Virus Infection in
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First, the origin of primary introd
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To reduce the amount of virus that
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OP 9.1 New Developments in the Trea
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OP 9.3 Future options in non respon
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the consensus interferon dose (25,2
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interferon maintenance therapy in d
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26) Kaiser S, Hass HG, Gregor M. Su
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OP 10.1 Progress in Microbicide Dev
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OP 10.3 Current Advances in HIV Vac
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dysmetabolic, functional, or organi
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Orphans Forty-three percent of the
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PP 1.4 Pradip Timilsena, Laxmi Pras
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PP 1.6 Epidemiological aspects of M
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PP 1.8 HIV transmission in The Gamb
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PP 1.10 Evaluation of Prevalence an
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PP 1.12 Co-morbidity of HIV, Hepati
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PP 1.14 Screening for HIV-infection
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PP 1.16 Low performance of HIV-1 se
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PP 1.18 The spread of HIV-1 resista
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PP 1.20 Study of the anti-HIV recom
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PP 1.22 Comparative investigation o
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PP 1.24 Foreign Citizens Admitted t
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PP 1.26 Morbidity pattern of PLWA r
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Page 116 and 117: PP 1.31 Pakistani Youth and their R
Page 118 and 119: PP 1.33 Model based on a quantum al
Page 120 and 121: PP 2.2 Analysis of Full-length HIV-
Page 122 and 123: PP 2.4 The Cobas Ampliprep/Cobas Ta
Page 124 and 125: PP 2.6 Mutations and Polymorphisms
Page 126 and 127: PP 2.8 Differences in the phenotypi
Page 128 and 129: PP 2.10 Biochemical Characterizatio
Page 130 and 131: Upon 60 minutes contact of BVDV spi
Page 132 and 133: PP 2.13 Assessing the Contribution
Page 134 and 135: PP 2.15 Correlation between circula
Page 136 and 137: PP 2.17 Interface targeting peptide
Page 138 and 139: PP 2.19 HIV-1 gene expression: less
Page 140 and 141: PP 2.21 Vesical Cancer and Papillom
Page 142 and 143: PP 3.2 Is Age a Complicating Factor
Page 144 and 145: PP 3.4 Reversible HIV associated en
Page 146 and 147: PP 3.6 Mycobacterium Ulcerans Cutan
Page 148 and 149: PP 3.8 A Clinical Study Of 60 Patie
Page 150 and 151: PP 3.9 HIV and Tuberculosis: Partne
Page 152 and 153: PP 3.11 Clinicopathological compari
Page 154 and 155: PP 3.13 Crohn's disease onset in a
Page 156 and 157: PP 3.15 Ocular manifestations occur
Page 158 and 159: PP 3.17 Bladder carcinoma observed
Page 160 and 161: PP 3.19 Increasing concerns related
Page 164 and 165: PP 3.23 Proviral DNA and plasma vir
Page 166 and 167: PP 3.25 Frequency, monitoring, clin
Page 168 and 169: PP 3.27 HIV-positive cases as part
Page 170 and 171: PP 3.29 The Effects of a Supervised
Page 172 and 173: PP 3.31 Prevalence of depression am
Page 174 and 175: PP 3.33 Rhinopharyngeal carcinoma w
Page 176 and 177: PP 3.35 Large vessel damage due to
Page 178 and 179: PP 3.37 No Interference between Syp
Page 180 and 181: PP 3.39 Faecal flora, diarrhoea ass
Page 182 and 183: PP 4.2 Triple therapy adherence in
Page 184 and 185: In a hierarchical multiple regressi
Page 186 and 187: PP 4.5 A Prospective Study of Antir
Page 188 and 189: PP 4.7 Telephone-Based Coping Impro
Page 190 and 191: PP 4.9 The significantly different
Page 192 and 193: PP 4.11 Self-Evaluation Investigati
Page 194 and 195: PP 4.13 Treatment of Kaposi's sarco
Page 196 and 197: PP 4.15 Pharmacokinetic Interaction
Page 198 and 199: PP 4.17 Human immunodeficiency viru
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Page 202 and 203: PP 4.21 No evidence of reduced viro
Page 204 and 205: PP 4.23 The increased liver toxicit
Page 206 and 207: PP 4.25 Rapid Oral Desensitization
Page 208 and 209: PP 5.1 Rational design of HCV antig
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PP 5.5 Fulminant Candida albicans p
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PP 5.7 Chronic hepatitic C-prompted
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PP 5.9 Chronic HBV infection associ
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PP 6.2 Broadly protective immunity
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PP 6.4 Chikungunya arthritis sequel
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PP 6.5 Experimental determination o
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PP 6.7 Emerging Arboviruses in Turi
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PP 6.9 Ocular LOA-LOA Infection in
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PP 6.11 Prevalence of Carriers of M
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PP 6.13 Evaluation of the Prevalenc
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PP 6.15 Assessment of need of Patie
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PP 6.17 Community-acquired septicem
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PP 6.19 Multiple sclerosis managed
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PP 6.21 A serious staphylococcal kn
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PP 6.23 Comparison between Secretor
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PP 6.25 Real- Time PCR and Flow Cyt
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FP 0.2 Preclinical Development of a
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FP 0.4 Primary Resistance of a Nove
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FP 1.1 In Vivo Emergence of RANTES-
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Conclusion:Patients with HIV-1 infe
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FP 1.4 V1V2 loop length variation d
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FP 1.6 Analysis of plasma cytokines
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FP 1.8 Detection of low frequency H
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FP 1.10 Replication-Competent Platf
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FP 2.1 Long-term non-progression in
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FP 2.3 HAART in Sub-Saharan countri
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FP 2.5 Morbidity and mortality by b
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FP 2.6 Tolerability of antiretrovir
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FP 2.8 Radata - An internet-based s
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FP 3.2 Glycosylated recombinant sim
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FP 3.4 Therapeutic Tat-Vaccination
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PL 2 HIV & AIDS Research: The Light
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SS 1.1 The importance of HIV drug r
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SS 1.3 Virtual phenotype and Clinic
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SS 2.1 The Lessons we have Learned
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SS 5.2 HIV-associated facial lipoat
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SS 6.1 How to improve the use of ne
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SS 6.3 Tipranavir is a Potent Prote
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ABBOTT FRANCE Abbott is a global, b
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BOEHRINGER INGELHEIM The Boehringer
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IVAGEN IVAGEN SA, a biotechnology c
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ROCHE Headquartered in Basel, Switz
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Putting knowledge into practice VIR
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NOTES “ Focusing FIRST on PEOPLE
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