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Brian P. Jacob, David C. Chen, Bruce Ramshaw, Shirin Towfigh (eds.) - The SAGES Manual of Groin Pain-Springer International Publishing (2016)

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7. <strong>Groin</strong> <strong>Pain</strong> Etiology: Athletic Pubalgia Evaluation…<br />

69<br />

Induced by exercise, the pain has a characteristic clinical pattern <strong>of</strong><br />

medial thigh pain commencing in the region <strong>of</strong> the adductor muscle<br />

origin and radiating distally along the medial thigh, with strenuous<br />

exercise. An anatomic study on cadaver limbs by Harvey and Bell<br />

reinforced the concept that obturator neuropathy is caused by an entrapment<br />

syndrome due to the angle that the nerve pierces the adductor<br />

muscles and travels between the adductor fascial compartments [ 20 ].<br />

In athletes, congenital anatomic nerve variants combined with physical<br />

training that augments adductor muscle development may be the<br />

main mechanism for obturator nerve entrapment syndrome in athletic<br />

pubalgia.<br />

In addition to the sensory abnormalities, motor deficits can be found<br />

in advanced cases <strong>of</strong> obturator nerve entrapment. Physical exam may<br />

reveal asymmetry between affected and non-affected sides, with weakness<br />

and atrophy <strong>of</strong> adductor muscles on the affected. MRI will rarely<br />

show nerve- related injury, but is helpful to rule out adductor or gracilis<br />

tendonitis involvement. In cases where the MRI is normal, but clinical<br />

suspicion is high, the best test to confirm obturator neuropathy is needle<br />

electromyography (EMG). Kimura et al. noted that fibrillation potentials<br />

or high-amplitude, long-duration complex motor unit potentials were<br />

consistent with chronic denervation <strong>of</strong> the hip adductor muscle group,<br />

but not in other lower extremity muscles [ 21 ].<br />

Treatment <strong>of</strong> Nerve Entrapment Syndromes<br />

Nerve pain is generally as hard to treat as it can be to diagnose. <strong>The</strong><br />

longer the pain symptoms have persisted, the more difficult it is to<br />

achieve adequate cessation <strong>of</strong> pain. In fact, treatment strategies can be<br />

divided into acute and chronic/recurring pain categories. For all three<br />

neuralgia syndromes, acute pain is best treated with activity cessation<br />

and NSAIDs for 3–6 weeks, followed by a strength and flexibility rehabilitation<br />

program that leads to competitive activity resumption. Regional<br />

nerve blocks that are diagnostic <strong>of</strong> nerve entrapment can be modified<br />

with long-acting local anesthetics (bupivacaine) and a corticosteroid<br />

(prednisolone tebutate) for sustained pain relief. For chronic and recurring<br />

pain that prevents full return to training and competition, nerve<br />

surgery will be required. For the ilioinguinal and genit<strong>of</strong>emoral nerves,<br />

neurectomy, <strong>of</strong>ten together, achieves good results. Since these nerves are<br />

mostly sensory in function, resection at the level <strong>of</strong> the transversus

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