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A B S T R A C T B O O K – A B S T R A C T S O F T A L K S FINE-TUNING OF PLANT IMMUNE RESPONSES TO HOST-ADAPTED PATHOGENS Jane Parker, Ana Garcia, Servane Blanvillain-Baufumé, Katharina Heidrich, Lennart Wirthmüller, Steffen Rietz Department of Plant-Microbe Interactions, Max-Planck Institute for Plant Breeding Research, Cologne, Germany Email: parker@mpipz.mpg.de 49 X X I V S P P S C O N G R E S S 2 0 1 1 Pleanary session 7 Plants have evolved a multi-layered innate immune system to recognize and respond to potentially destructive microbes in the environment. Resistance to host-adapted biotrophic pathogens often involves receptor-mediated transcriptional mobilization of defense pathways and programmed death of host cells at infection sites. However, these induced defenses are energetically costly and disturb normal metabolism and growth and therefore have to be tightly controlled. Fine-control is achieved via a stress signaling network which allows flexibility in responsiveness to pathogens and other environmental stimuli, depending on the prevailing conditions. While components of individual stress signaling pathways have been identified and to some extent characterized our understanding of how these pathways intersect at the molecular, cellular and tissue levels is relatively poor. We’re attempting to unravel resistance processes triggered by intracellular immune receptor-mediated recognition of a bacterial type III secreted effector and position them within the larger stress signaling network. I’ll describe our analysis of resistance signaling dynamics within host cells and how we think intracellular reconfigurations of protein complexes governing transcriptional reprogramming contribute to a balanced and flexible immune response.
Session 13.Pathogen defense and plant desease A B S T R A C T B O O K – A B S T R A C T S O F T A L K S PROTEIN PHOSPHATASE 2A AS A REGULATOR OF STRESS RESPONSES IN ARABIDOPSIS Andrea Trotta 1 , Grzegorz Konert 1 , Moona Rahikainen 1 , Michael Wrzaczek 2 , Mikko Tikkanen 1 , Eva-Mari Aro 1 , Saijaliisa Kangasjärvi 1 1 Department of Biochemistry and Food Chemistry, University of Turku, Turku, Finland 2 Department of Biosciences, University of Helsinki, Helsinki, Finland E-mail: saijaliisa.kangasjarvi@utu.fi Chloroplasts perform essential signaling functions in cellular networks that modulate various stress responses in plants. We have taken a reverse genetic approach to identify components in the cross-talk of chloroplast signaling. Mutants deficient in subunits of protein phosphatase 2A (PP2A) showed peculiar light-intensity-dependent phenotypes, and were chosen for further analysis. A PP2A regulatory subunit B was identified as a cytoplasmic component in the cross-talk between light acclimation and disease resistance in Arabidopsis thaliana. Under moderate light intensity, knock-down pp2a-b mutants show disintegration of chloroplasts, premature yellowing and constitutive activation of salicylic acid and jasmonic acid dependent defense responses, but no increases in hormone levels. Promoter::GUS analysis indicates activity of PP2A-B in patches that highly resemble the yellowings on pp2a-b leaves. Genetic analysis suggests that PP2A-B is functionally connected with CONSTITUTIVE EXPRESSION OF PR GENES 5 (CPR5), which acts as a negative regulator disease resistance and senescence in Arabidopsis leaves. Yeast twohybrid screening for candidate PP2A-B interacting proteins also identified components that operate on defense-associated pathways. Moreover, the constitutive defense response associates with hypomethylation of DNA and increased levels of methioninesalvage pathway components in pp2a-b'γ leaves. Studies are underway to specify the mechanisms of PP2A dependent signaling functions in plants. 50 X X I V S P P S C O N G R E S S 2 0 1 1
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Page 3 and 4: Foreword eword ord A B S T R A C T
Page 5 and 6: Program of the Congress gram of the
Page 7 and 8: A B S T R A C T B O O K - P R O G R
Page 9 and 10: A B S T R A C T B O O K - P R O G R
Page 11 and 12: Plenary session 1 A B S T R A C T B
Page 13 and 14: Plenary session 2 THE BIOLOGY OF AL
Page 15 and 16: Session 1. Biotechnology and synthe
Page 17 and 18: Session 1. Biotechnology and synthe
Page 19 and 20: Session 2. Systems biology and -omi
Page 21 and 22: Session 2. Systems biology and -omi
Page 23 and 24: A B S T R A C T B O O K - A B S T R
Page 27 and 28: Pleanary session 4 A B S T R A C T
Page 29 and 30: Invited talk 4 A B S T R A C T B O
Page 31 and 32: Session 5.Cell biology A B S T R A
Page 33 and 34: Session 6.Organelle biology A B S T
Page 35 and 36: Session 6.Organelle biology A B S T
Page 37 and 38: Session 7.Development A B S T R A C
Page 39 and 40: Session 8.Signalling A B S T R A C
Page 41 and 42: Pleanary session 6 A B S T R A C T
Page 49: A B S T R A C T B O O K - A B S T R
Page 53 and 54: Session 13.Pathogen defense and pla
Page 67 and 68: Sapporo Medical University, Sapporo
Page 91 and 92: SCF = A B S T R A C T B O O K - A B
Page 93 and 94: A B S T R A C T B O O K - L I S T O
Page 95 and 96: A B S T R A C T B O O K - L I S T O
Page 97 and 98: Sponsors onsors ors A B S T R A C T

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