hysiological foundations...fJJHT(JIll )' ,1002 3 ,5 6 7 I,r1""/rgIf L1..lD7 129 -- s,-,ZJt /tItJ,'. 117 7.45SJOr N-"- ~1!11l ",.l~~( N-1OP/Iz-.1 1-4 N-I,JTht i1crtfJsei11hr P/Iz-a1 IIJ8b lilt beller, the 11I0rt exactly Ihe 1Mpalienl R1IIDMS flit nstr"dion ofcomilllJinr doily t'Xef'&bt 1rtJining.'tJ A~tiliJn oIH1t' tIItropy is usually only 'SSOI"y ~1Itn astlll'rtstressful i1l!umae.g.'IIiJ, tpt'roliJn) ((JUst. flJt Po; tTl to,sinKIrJ6050l:N 10ortJUgN,y !Ittpre-hropy Itvtl.- 2 0 2 4 6 8 J) 12 14 16 18 20 22 24 26 28 30 32 .34 36 38 40 42 44 46 48 50 52 wrebt ..43Onlfdconlinutfus/y dro)pi/W.rofptl/;ens29complied wllh thertf/IIesi ltJra regtit1r hi/ow-upo 2 3 5 6t -15I10-7 8 9 10 11 12 I1KJflIIIsFig.41 Measurements showing the lasting, almost unchanged increase of the arterial P02 by means of the discoveredfunctional regeneration procedure for the lung-heart system on initially 53 subjects (groups of 43 and 10 witha mean age of 65.3 years and 62.6 years) without pronounced chronic unspecific lung diseases. Sx =standarddeviation; the 02-art increase is statistically significant at the 1% level; study made by H. G. Lippmann and H. H.Wiemuth in our Institute 1979/801.160;n ftlctors ~16 '23 ~.J) 1,,25POrirf10013mmIfJ'" e IrPa70 F-~=--+---t----t----i600 9 If 27 3617f ~ '7~~,gluessloIistf('(]l!y sgnliCllnl. rp-O,D5).'o,lJ,c,d,t: difftlMlZS !IIltIttn tk POrt!tFig. 42 Effect of the 02MT regeneration procedureon the arterial re ting P02 in 30 patients aged5 9 V a with roughlv normal blood pressur .i n from non pecialized cure institutions12"9This increase only gains considerable medicalsignificance with the fact, which now has beenstatistically proven, that it lasts for up to morethan a year, provided severe stressful events donot lead to a lasting reduction (switching).Figure 41 shows the results of a special investigationinto the duration of the P02_art elevationafter 3, 6, 9 and 12 months. The resultof a further study into the gradual rise in theP0 2 increase with an increasing number oftreatment hours, as well as into the duration ofthe effect after the end of the procedure anbe seen in Fig. 42.The regulating and wit hing m hani m f thmicrocirculation, th effe t of whi h in th pulnaryy tern i chara t tiz d in th pr . ufigure, i actually nothing but th r al fnatural pro e which ha b n kn n f rlong tim and ha be n larg I pI .n del ctron mi ro op [89 90]. r rrito th pr f th sw lling of th nd th l' Icells in O 2 deficiency. B u thi 1.
Basic mechanisms anunctions5511111;tfellz«4SB50+----_~I40~----......JI I IOzHT IIfItr ilion f£1ff ok--0oס1 ....1' , IP lIz « under inho/a/ion- 120mmHg ~8lrPaArterial resting levels of P02 (A) and the2 (B) before and after 30 hrs 02MT cure prore,represented as x + 1s. N = 13. (Therapynders with a mean age of 59 ± 9.4 years) [92]est capillary cross-section extends again,the microcirculation increases. Our diserytherefore refers to the reversibility offundamental, natural mechanism whichoccurs in all capillaries of the human body,and also to the recognition and proof that thebasis of this mechanism is formed by a feedbacksystem with switching properties, whichleads to the persistence of the bioenergeticallytriggered effect.As has been mentioned above, the ba ic effect,the increase in the P02-art or in the oxygentransport to the body tissue, has unfortunatelybeen called into question by two studie madein foreign institutes with small numbers of patients,e.g. [91]. Since these negative findingsmet with an extraordinary response in variousmedical journals, centers in Dresden andelsewhere, which had been practising the 02MTfor some time, carried out studies with largenumbers of patients into the question of theincrease or non-increase in the P0 2 _ artby meanof 02MT. The positive and evidential result ofthese studies are given in Fig. 43 and Table 4We should refer here to the works of Caspe.[93, 94], in which it is established using 108cure patients that 18 months after 02MT amean increase in the resting P02_art of 6 mmHg(0.7 kPa) still exists, as compared with the pretherapylevel.e 4Results of 12 studies into the lasting increase of the arterial P0 2(tP02-ar ), at rest, by means of 02MTdies, authors no. of patients toP02-art [mmHg] time(mean age)3rd German editionof this bookp.35.37p.37.38p.38p.39p.44p.67p.317f rther studiesCaspers 1985olf 1985olte 1980von ArdenneLippmannseveral sanatoriavon ArdenneWolf10 clinicsvon Ardennevon ArdenneSchnizer (Fulgum)19811982 (!)8 (72 years)43 (64 years)30 (59 years)13 (59 years)108 (67 years)1407 (69 years)5 (- 60 years)15 (58 years)3827938 (67 years)88 (62 years)22 (62 years)22 (also after 30 days)14 (after 90 days)13 (after 50 days)18 (after some days)19 (after some days)12 (after> 30 days)16 (after some days)4 (after> 1 day) 1o9,4 (after> 1 day)13 (after> 1 day)14 (after> 1 day) 1o (failure to reach switching threshold):ti:',.E!!E!r:!!i~on!!.:2682 (I) toPa02 = 14 mmHg (!) or 1,8 kPa11 studies1 study (Nolte) 22 ~ = 0 mmHgverage lowering of the P0 2- ven(flP02_ven), at rest, in these studies was -10,3 mmHg (v.A.) or -15 mmHgment of the effectiveness of the 02MT without including measurements of the changes occuringvenous Po is not possible I This basic principle was ignored by some authors (Nolte, Hendrik,in false assessments of effectiveness.ulti~
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Exholationtubeloop aroundcmebroncll
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