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Annals of Oncology pemetrexed (PEM) maintenance therapy produced a high magnitude of PFS improvement. The OLCSG 0903 phase 2 trial investigated efficacy and safety of cisplatin (CDDP)-docetaxel (DOC)-BEV induction therapy followed by BEV-PEM maintenance therapy in patients with advanced nonsquamous non-small cell lung carcinoma. Methods: In this trial, 40 patients with good PS (0 or 1) participated in the induction phase, specified as four cycles of induction CDDP (80 mg/m 2 ), DOC (60 mg/m 2 ) and BEV (15 mg/kg) on day 1 of a 21-day cycle. Patients who had not progressed during CDDP-DOC-BEV induction received maintenance BEV (15 mg/kg) and PEM (500 mg/m 2 ) on day 1 of a 21-day cycle until disease progression. The primary endpoint was PFS, and the secondary endpoints included toxicity, OS and response rate. Results: Patient characteristics were as follows: median age: 62 years; 78% male; 100% Japanese; 30% PS 0; 73% stage IV; and 70% adenocarcinoma. At the time of this analysis, 23pts (58%) discontinued the treatment, and the proportion of discontinuations to AEs was 35% (8/23). The principal toxicity was myelosuppression (grade 4 hematological: 20 patients [50%]), and grade 3/4 febrile neutropenia was observed in 10 (25%) despite no treatment-related deaths. The objective response rate and disease control rate (% patients with CR/PR/SD) was 82.5% and 97.5%, respectively. The median PFS time was 10.2 months, and the 6-month PFS rate was 63.2% (95% confidence interval: 44.9-76.9 %). Conclusions: CDDP-DOC-BEV followed by BEV-PEM maintenance seems an effective and moderately tolerated treatment for patients with advanced nonsquamous non-small cell lung carcinoma. Disclosure: K. Hotta: Eli Lilly Japan sanofi-aventis Chugai Pharmaceutical Co. Ltd Nippon Kayaku. N. Takigawa: Eli Lilly Japan sanofi-aventis Chugai Pharmaceutical Co. Ltd. K. Kiura: Eli Lilly Japan sanofi-aventis Chugai Pharmaceutical Co. Ltd Nippon Kayaku. All other authors have declared no conflicts of interest. 1328 META-ANALYSIS OF RELATIONSHIP BETWEEN SKIN RASH AND OUTCOME IN NON-SMALL-CELL LUNG CANCER (NSCLC) PATIENTS TREATED WITH ERLOTINIB (E) AND GEFITINIB (G) K.F. Borgonovo, F. Petrelli, M. Cabiddu, M. Ghilardi, M. Cremonesi, F. Maspero, S. Barni Medical Oncology Division, Azienda Ospedaliera Treviglio-Caravaggio, Treviglio, ITALY Introduction: Dermatological toxicity in the form of acneiform rash is a common event in NSCLC patients being treated with anti-EGFR TKIs. An association between clinical benefit by EGFR-targeted therapy and development of this form of skin toxicity has been noted. The objective of this meta-analysis was to assess the predictive value of skin rash for outcome in patients with NSCLC treated with E or G. Materials and methods: Prospective clinical trials or retrospective case series with reported survival (OS), progression (PFS/TTP) and response rate (RR) as a function of skin rash were serched in PubMed until January 2012. The selected studies have to include adult patients with histologically confirmed NSCLC treated with G or E, alone or in combination with other approved agents. Hazard ratios with 95% confidence intervals (HRs) for PFS/TTP and OS and risk ratios (RRs) for response rate in patients with rash were pooled in a meta-analysis. Results: Twenty-four publications were included in this meta-analysis (17 prospective trials and 7 retrospective case series) for a total of 3032 patients. For the primary endpoint (OS) the occurrence of skin rash was significantly associated with reduced risk of death in patients treated with E or G (HR 0.30, p 3 14/7; non-smoker/smoker: 13/8, with EGFR mutations/without EGFR mutations – 12/9. Results: Median PFS was 7 months overall, ORR was 75% (9/12, 8 partial responses, 1 complete response) and the MoS was not reached in patients with EGFR mutations. Median PFS was 2 months overall, no objective responses and the MoS was 4 months in patients without EGFR mutations. Differences are statistically significent in ORR and PFS (p < 0.05). Conclusions: EGFR TKIs showed high effectiveness and good disease control in patients with brain metastases from NSCLC with EGFR mutations. Disclosure: All authors have declared no conflicts of interest. 1331 COMPARISON OF SURVIVAL IN PATIENTS WITH ADVANCED LUNG ADENOCARCINOMA TREATED BEFORE AND AFTER GEFITINIB APPROVAL IN CHINA Y. Shi 1 ,Y.Liu 1 , X. Hao 1 ,J.Li 1 ,X.Hu 1 , Y. Wang 1 , Z. Wang 2 , H. Wang 1 , X. Han 1 , X. Zhang 1 1 Oncology, Cancer Hospital-China Academy of Medical Sciences, Beijing, CHINA, 2 Medical Oncology, Cancer Hospital-Chao Yang District BeijingCAMS and PUMC, Beijing, CHINA Objective: This study compared the overall survival (OS) between advanced lung adenocarcinoma patients of before and after gefitinib approval in China. Methods: The clinical data of 558 advanced lung adenocarcinoma patients who ever received palliative chemotherapy were reviewed retrospectively. According to a matched-pair case-control study design, 255 patients of before gefitinib approval who only received palliative chemotherapy and 255 patients of after gefitinib approval who received gefitinib treatment were matched by age, sex and smoking history. Volume 23 | Supplement 9 | September 2012 doi:10.1093/annonc/mds409 | ix435
Results: The median survival time (MST) of 510 advanced lung adenocarcinoma patients from the date of first-line palliative therapy was 22.8 months. MST was significantly longer among the patients treated with gefitinib after gefitinib approval compared with the patients treated before gefitinib approval (33.5 vs. 14.1months, p < 0.001). Multivariate analysis showed that the independent prognostic factors to significantly improve OS of 510 patients included gefitinib treatment (hazard ratio 0.175, P
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Annals of Oncology www.annonc.oxfor
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subscriptions A subscription to Ann
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Annals of Oncology Official Journal
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The European Society for Medical On
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Audit Committee Chair: Fortunato Ci
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Familial cancer Judith Balmaña, Ba
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ESMO 2012 Congress Travel Grants 47
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Exhibitors The following companies
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ESMO Fellowships, 1989-2011 The Eur
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Ondrej Gojis, Czech Republic 2008-2
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abstracts hpv biology and implicati
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abstracts the characterization of l
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abstracts description of intra-tumo
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prevent cell death. It is anticipat
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22IN TARGETING THE HOST IN TNBC G.
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abstracts a paradigm shift in early
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abstracts how can medical oncologis
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feasibility), but by how high the c
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abstracts re-inventing the medical
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abstracts emerging diagnostic and t
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abstracts biologically based treatm
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abstracts molecular targets in mali
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abstracts melanoma therapy: from fr
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diagnosis and can also be used for
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analysis at 11 months median follow
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mouse model of Kras mutant NSCLC. I
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abstracts subtyping soft tissue sar
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abstracts key topics in supportive
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ESMO-CSCO Joint Symposium: Building
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ESMO-EANM-ESR Joint Symposium: Imag
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ESMO-ESTRO Joint Symposium: Innovat
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ESMO-MASCC Joint Symposium: Integra
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ESO Society Symposium: Celebrating
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Results: TIMP-1 expression was incr
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will benefit from this targeted the
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cytomegalovirus (CMV). Analysis of
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functional consequences of Endoglin
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elationship between the ROR score,
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183P EPIDERMAL GROWTH FACTOR RECEPT
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Plasma adiponectin level on the pre
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Table: 198P PFS OS Median, mo HR Me
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204P EVALUATION OF PTEN AND PIK3CA
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Material and methods: We searched P
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Results: The median concentration o
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Table: 226P Methods: Pts were rando
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However, additional analysis sugges
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number of biomarkers have been trie
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Table: 248O 249PD PROTEOMIC PREDICT
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Conclusions: BW and serum Ctrough o
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261P BREAST CANCER SUBTYPES AND OUT
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90 mg/m2-cyclophophamide 600 mg/m2
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to 9 weeks after dosing. Trastuzuma
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Patients and methods: We reviewed d
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sector patients. The aim of this st
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Linear Logistic Model with Relaxed
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Results: The median CTC fold change
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312: Table: Chemotherapy uptake wit
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abstracts breast cancer, locally ad
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Results: 8 trials (2092 pts) with 1
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absolute difference of median value
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Novartis, Genentech, and sanofi-ave
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Results: Three RCTs with 1023 patie
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collected from all patients for det
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355P FIRST REPORT OF LONG-TERM RESP
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361P A RETROSPECTIVE ANALYSIS OF PL
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publications and aggregated in a me
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Thus the primary aim to target BCSC
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383 WHY DO WOMEN WITH BREAST CANCER
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Results: We evaluated 76 pts with M
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more than 6 cycles of capecitabine.
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406TiP PHASE II TRIAL OF PRIMARY SY
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abstracts CNS tumors 410O CONDITION
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Conclusions: Our data suggested tha
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Conclusions: As in other cancer typ
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432 GAMMA KNIFE RADIOSURGERY AS A M
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abstracts developmental therapeutic
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1PR), but no responses were seen in
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RO and Cd, as well as PD data for c
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and vulvar Ca), and 11 SDs were obs
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knowing HLA-A status double-blindly
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Acquired-resistance to EGFR inhibit
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474P PHASE 1 STUDY OF THE SELECTIVE
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TST is active in breast and gastric
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Treatment-induced shedding of circu
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Methods: Either co-cultures of peri
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501 PRECLINICAL DATA INVESTIGATING
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509TiP LUX-LUNG 8: A RANDOMIZED, OP
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(P = 0.64). Synchronous BBC was sig
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abstracts gastrointestinal tumors,
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Disclosure: M. Lee: I am an employe
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plus intravenous Bev(7.5mg/kg q 21d
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anti-EGFR treatment. The present st
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patients harboring a T/T genotype t
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551P ADJUVANT CHEMOTHERAPY (AC) USE
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experienced significantly more ≥
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functioning scales. Exploratory ana
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oxaliplatin (100 mg/m 2 )/raltitrex
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572P PANERB STUDY: WHICH CATEGORY O
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Results: 92/114 patients were preop
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585P CHANGES IN THE INTESTINAL ENVI
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592P PHASE II TRIAL OF COMBINED CHE
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minutes. In a previous study, 30-mi
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Patients and methods: Chemotherapy-
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612P ARE PATIENTS WITH RESECTABLE R
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Conclusions: AMPK and ACC activatio
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of surgical monotherapy in patients
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Table: 632 633 AFLIBERCEPT/FOLFIRI
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factors play the determining role i
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Abstract withdrawn in exceptional c
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were respectively 10.6 vs 8.5 vs 3.
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Results: 20 gastrointestinal cancer
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abstracts gastrointestinal tumors,
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Day 8. A second identical course wa
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proven in stage I GC. The aim of th
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Conclusions: Longer OS to FOLFOX wa
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692P PRELIMINARY SAFETY DATA FROM A
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subgroup. Taking into consideration
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Results: Three years of adjuvant im
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considered sufficient to give an 80
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721P FIRST-LINE TREATMENT WITH FOLF
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after Gem-based therapy. The additi
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735P RADIOGRAPHIC PARAMETERS IN PRE
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validate the revised AJCC 7th stagi
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Results: Among 862 MM we identified
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Results: Frequently reported advers
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gastric cancer patients with CNS me
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discriminate the prognosis of HCC p
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prospective, non-interventional stu
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abstracts genitourinary tumors, non
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787O EXTERNAL VALIDATION OF THE ASS
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patient preference for P over S, an
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798PD OPEN-LABEL PHASE II TRIAL OF
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Hb, PS and LM. Median PFS for patie
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may have led to reduced dose and sh
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Results: 1,622 patients were identi
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RCC) was designed to address an unm
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Patients and methods: This is a sin
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treatment at week 4, and week 12 by
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effective in second or further line
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Conclusions: In pts with RCC treate
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using Drug inCode ® pharmacogeneti
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Table: 857P standard, but is not av
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efused HDCT. Of 23 patients, 20 com
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we identified 49 and 220 patients w
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879 TREATMENT OF PATIENTS WITH META
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Median overall survival (OS) was 26
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abstracts Genitourinary tumors, pro
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and week 13 BPI-SF Q3 scores), 28%
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Working Group (PCWG)-2 guidelines r
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atio HR 0.39; CI 0.18, 0.83, p = 0.
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We observed tumor responses and pro
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Here we report emerging data from t
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928P LHRH AGONIST-INDUCED SUPPRESSI
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Disclosure: S. Oudard: Has acted as
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939P NEOADJUVANT SIPULEUCEL-T IN LO
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945 BONE TURNOVER MARKERS AND POTEN
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952 SEQUENTIAL ANDROGEN DEPRIVATION
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managed in a multidisciplinary (MD)
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or hypercalcemia at screening; prio
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meeting. The prevalence of LGSC is
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Table: 975PD Continued AMG 386 + pa
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physicians in the U.S. and Europe.
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989P PHASE II STUDY OF COMBINATION
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elated with stage, nodal involvemen
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1004P CASE CONTROL STUDY ON TWO DIF
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eight patients who had infertility
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abstracts head and neck cancer 1016
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same CT/RT; Arm B2: 3 cycles of ind
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induction chemotherapy in the treat
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patients. We have developed a rando
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evaluated by the screening instrume
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morbidities that radiation would le
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Conclusions: Through adequate selec
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abstracts hematological malignancie
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anthracyclines in vitro. A favorabl
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1076P RITUXIMAB PLUS SUBCUTANEOUS C
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than the standard target of ≥2.5
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Patients: From November 2002 to May
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lymphadenopathy and multiple cutane
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prospective non-interventional stud
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Funding: GSK Biologicals Disclosure
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survival rates of 13-25% (Prieto et
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high response rates and prolonged p
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GlaxoSmithKline, Merck Sharp & Dohm
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advisor for Merck Sharp & Dohme, an
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or cutaneous melanoma. A survival u
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systemic therapy or were intolerant
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abstracts neuroendocrine tumors and
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morbidity, with G3 tumors behaving
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pts. Poorly differentiated endocrin
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Adenocarcinoma was present in 25 pa
Page 385 and 386: Method: Endobronchial ultrasound gu
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Page 389 and 390: disease after surgery were treated
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Page 393 and 394: 1200P CONCOMITANT CHEMORADIATION GI
Page 395 and 396: Patients and methods: The study was
Page 397 and 398: months (95% CI:13-25). The PFS and
Page 399 and 400: maintenance therapy had favourable
Page 401 and 402: abstracts non-small cell lung cance
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Page 405 and 406: 1234PD RANDOMIZED PHASE III TRIAL O
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Page 409 and 410: 1243P IDENTIFICATION OF MICRORNA (M
Page 411 and 412: N-arm n=34 P-arm n=32 All n=66 Male
Page 413 and 414: 1255P POPULATION BASED EVALUATION O
Page 415 and 416: 1262P DISTINCT SPREAD OF EGFR MUTAT
Page 417 and 418: 1268P VISUAL DISTURBANCES IN PATIEN
Page 419 and 420: MERCK SERONO: Advisor, speaker in s
Page 421 and 422: Conclusions: These data from SAiL a
Page 423 and 424: NSCLC diagnosis and time of BM diag
Page 425 and 426: 1291P PHASE I/II DOSE-FINDING STUDY
Page 427 and 428: Network utilization of WBCGF in the
Page 429 and 430: 1303P COST EFFECTIVENESS OF PEMETRE
Page 431 and 432: Results: The patients’ characteri
Page 433 and 434: EGFR mut at exons 18, 19, 21, and K
Page 435: Methods: EML4-ALK fusion was screen
Page 439 and 440: enefit from sustained EGFR blockade
Page 441 and 442: epresented by 19 pts (32%) female,
Page 443 and 444: and at least one prior course of ch
Page 445 and 446: Methods: NSCLC patients with radiog
Page 447 and 448: 1367TiP LONG-TERM ERLOTINIB THERAPY
Page 449 and 450: Austria, Czech Republic, Finland, G
Page 451 and 452: were every 6 weeks (wk) (S1), every
Page 453 and 454: Advantage enrollees (83% vs. 25%) [
Page 455 and 456: Results: Based on 10,000 simulation
Page 457 and 458: cancer tumors 12%, Germ cell tumor
Page 459 and 460: Material and methods: 50 lung cance
Page 461 and 462: 1414TiP IMPLEMENTATION OF CANCER RE
Page 463 and 464: abstracts palliative care 1422O EFF
Page 465 and 466: Method and results: A total of 317
Page 467 and 468: 1436P SURVEY ON MODELS OF INTEGRATI
Page 469 and 470: care unit (PCU) at the National Can
Page 471 and 472: Characteristic No. % Total 63 1 st
Page 473 and 474: hysterectomised-9.10% and cervix ca
Page 475 and 476: abstracts psycho-oncology 1462PD IN
Page 477 and 478: events, tumour response, and surviv
Page 479 and 480: abstracts sarcoma 1477O ADHESION TO
Page 481 and 482: 1481PD A PHASE II STUDY OF DOVITINI
Page 483 and 484: 1488P ORGANISATION OF SARCOMA PATIE
Page 485 and 486: Patients and methods: From August 2
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chemotherapy respectively. At a med
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of this group Those with relapsed d
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1515 PREDICTORS OF SURVIVAL IN ADOL
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abstracts small cell lung cancer an
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emaining receiving either CAV or to
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1533P EXPRESSION OF WILM⍰TUMOUR G
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more than 3 months after first line
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lower recurrence rate of NE. Pegfil
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egimens (HR = 2.5, p < 0.001). Phys
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1561P PREDICTIVE DIAGNOSTICS FOR RE
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Methods: A prospective multicentre
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Table: 1574P Pharmacokinetic parame
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Novartis and unrestricted research
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studies have shown that chemotherap
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(Grade 1) and moderate to severe (G
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declined to relatively lower level
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1609P PHYSICAL ACTIVITY (PA) AND PH
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patients were admitted to the oncol
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Dicarbamin 100 mg/day on day 5 befo
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Results: Anemia was detected in 83.
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important to carry out randomized s
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abstracts translational research 16
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expression. Then, we analyzed PB sa
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Table: 1657P TH BV + TH HR (95% CI)
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BRAF mutations. Circulating free DN
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1671P PHASE I CLINICAL TRIAL OF CAN
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1678P PREDICTIVE VALUE OF TWO PHENO
Page 541 and 542:
theorized that the PI3K/AKT pathway
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Material and methods: 101 patients
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overexpressing and 232 had triple n
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author index author index A Aamdal
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author index Badran A. ix288 (874)
Page 551 and 552:
author index Bösmüller H. ix209 (
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author index Chen A. ix319 (966O) C
Page 555 and 556:
author index De Droogh E. ix452 (13
Page 557 and 558:
author index Esposito G. ix209 (618
Page 559 and 560:
author index Geiges G. ix306 (930P)
Page 561 and 562:
author index Hartono S. ix70 (155P)
Page 563 and 564:
author index Iwasaki H. ix542 (1694
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author index Kodaira M. ix90 (228P)
Page 567 and 568:
author index Lichtensztejn D. ix350
Page 569 and 570:
author index Martinez A. ix496 (153
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author index Morishita N. ix432 (13
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author index Okamoto N. ix432 (1320
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author index Piau S. ix456 (1401P)
Page 577 and 578:
author index Rodríguez Rodríguez
Page 579 and 580:
author index Scoggins C.R. ix371 (1
Page 581 and 582:
author index Stern L. ix272 (823P)
Page 583 and 584:
author index Trypaki M. ix429 (1308
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author index Whitmore J.B. ix310 (9
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subject index subject index A AAV-H
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subject index ix115 (316TiP), ix116
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subject index diffuse large B-cell
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subject index (1033P), ix340 (1036P
Page 595 and 596:
subject index medical information,
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subject index (612P), ix214 (633),
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subject index RCC, ix274 (830P) re-
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subject index TR-FRET, ix84 (206P)
Page 603 and 604:
drug index drug index 1248P, 1250P,
Page 605 and 606:
translational research index transl
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Page 609:
show all

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