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New trends in physics teaching, v.4; The ... - unesdoc - Unesco

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Colour Difference<br />

Colour<br />

<strong>The</strong> ability of the normal eye to assess differences of colour is not uniform over the CIE diagram.<br />

McAdam performed a series of experiments with several observers, lead<strong>in</strong>g to the results shown <strong>in</strong><br />

figure 12. <strong>The</strong> ellipses represent the ability of the eye to detect differences. It is clear that the eye<br />

is most sensitive to changes <strong>in</strong> hue and saturation <strong>in</strong> the blue, Our eyes are not very sensitive to<br />

changes of saturation <strong>in</strong> the green, <strong>The</strong> axes of the ellipses have been multiplied by ten. Colour<br />

differences are notoriously difficult to specify, which is a nuisance s<strong>in</strong>ce so much depends upon<br />

them <strong>in</strong> the dye and pa<strong>in</strong>t <strong>in</strong>dustries. Many pa<strong>in</strong>t and dye companies depend upon <strong>in</strong>terpolations<br />

of the Munsell scheme of order<strong>in</strong>g <strong>in</strong> terms of hue, value and chroma to specify their pigments.<br />

To avoid the limitations of human observers, recently both analogue and digital computers have<br />

been employed to measure colour differences employ<strong>in</strong>g spectra obta<strong>in</strong>ed from a spectrophotometer.<br />

1 I bl<br />

n .2 .3 .4 .5 .E .7<br />

:<<br />

.I .2 .3 .A .5 .6 .7<br />

x<br />

Figure 13. Defective colour vision. Loci of colou~s confused by protanopes (a) and deuteranopes (b).<br />

‘Colour bl<strong>in</strong>dness’ - Defective Colour Vision<br />

<strong>The</strong> CIE chromaticity diagram is based on the fact that the response of normal <strong>in</strong>dividuals to<br />

match<strong>in</strong>g colour is closely the same. However, a small percentage of the population has a reduced<br />

ability to perceive colours. <strong>The</strong> mechanism of the ret<strong>in</strong>al process is so little understood that it is<br />

uncerta<strong>in</strong> whether the causes of abnormalities lies <strong>in</strong> the malfunction of the ret<strong>in</strong>a or the process<strong>in</strong>g<br />

that follows this. All forms seem to be <strong>in</strong>herited. Abnormalities are divided <strong>in</strong>to monochromatism<br />

(‘total colour bl<strong>in</strong>dness’), dichromatism (‘partial colour bl<strong>in</strong>dness’) <strong>in</strong> which only two<br />

dist<strong>in</strong>ct hues are sensed, and anomalous trichromatism, where the three colour response differs<br />

from the norm. <strong>The</strong> colour response of a monochromatic (a better word would be ‘achromatic’)<br />

<strong>in</strong>dividual is the same for photopic and scotopic vision i.e. high or low levels of illum<strong>in</strong>ation. This<br />

leads to the belief that only the rod cells, which respond to low level illum<strong>in</strong>ation, are present <strong>in</strong><br />

such eyes. Dichromatism takes several forms, the effects of two of which, deuteranopia and<br />

protanopia, are shown <strong>in</strong> figure 13. Colours ly<strong>in</strong>g along the straight l<strong>in</strong>es on these diagrams are<br />

confused by such <strong>in</strong>dividuals. Thus, both would be unable to dist<strong>in</strong>guish red from green, and the<br />

207

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