Autism Studies and Related Medical Conditions, January 2009 - TACA

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infection that provide new insights into mechanisms by which neurotropic agents and/or immune factors may impact developing or mature CNS circuitry to effect complex disturbances in movement and behavior. Note in press: Since this chapter was submitted, several manuscripts have been published that extend findings reported here and support the relevance of BDV infections of neonatal Lewis rats as models for investigating mechanisms of neurodevelopmental damage in autism. Behavioral abnormalities, including disturbed play behavior and chronic emotional overactivity, have been described by Pletnikov et al. (1999); inhibition of responses to novel stimuli were described by Hornig et al. (1999); loss of Purkinje cells following neonatal BDV infection has been demonstrated by Eisenman et al. (1999), Hornig et al. (1999), and Weissenböck et al. (2000); and alterations in cytokine gene expression have been reported by Hornig et al. (1999), Plata-Salaman et al. (1999) and Sauder et al. (1999). PMID: 11417134 [PubMed - indexed for MEDLINE] 25: J Autism Dev Disord. 2006 Nov;36(8):1039-51. Etiologies of autism in a case-series from Tanzania. Mankoski RE, Collins M, Ndosi NK, Mgalla EH, Sarwatt VV, Folstein SE. Tufts University School of Medicine and Sackler School of Graduate Biomedical Sciences, Boston, MA, USA. Most autism has a genetic cause although post-encephalitis cases are reported. In a case-series (N = 20) from Tanzania, 14 met research criteria for autism. Three (M:F = 1:2) had normal development to age 22, 35, and 42 months, with onset of autism upon recovery from severe malaria, attended by prolonged high fever, convulsions, and in one case prolonged loss of consciousness. In four other cases (M:F = 3:1), the temporal relationship between onset of autism and severe infection was close, but possibly spurious since malaria is common in Tanzania and there were indications of abnormal development in the child or a family member. In seven cases, (M:F = 6:1) autism onset was unrelated to malaria. The excess of non-verbal cases (N = 10) is related local diagnostic practice. Publication Types: Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Autism Studies & Related Medical Conditions – TACA © Page 122
PMID: 16897390 [PubMed - indexed for MEDLINE] 26: Lakartidningen. 1992 Jan 29;89(5):279-80. [Information-based adjustment of sex education clinics for adolescents] [Article in Swedish] Ruusuvaara L. Ungdomshälsan, kvinnokliniken, Akademiska sjukhuset, Uppsala. PMID: 1738248 [PubMed - indexed for MEDLINE] 27: J Autism Child Schizophr. 1977 Mar;7(1):49-55. Depressed lymphocyte responsiveness in autistic children. Stubbs EG, Crawford ML. Although there are associations linking autism with prenatal rubella, cytomegalovirus, syphilis, and varicella, the etiology of the autistic state remains obscure. Host defense against the etiologic agents postulated to be responsible for the autism-associated syndromes is believed to be primarily of the cell-mediated type. In this preliminary study, cellular immune function was assessed in vitro by phytohemagglutinin (PHA) stimulation of lymphocyte cultures. Twelve autistic children and 13 control subjects were compared. The autistic group exhibited a depressed lymphocyte transformation response to PHA when compared to the control subjects (p less than .01). Publication Types: Comparative Study In Vitro Research Support, U.S. Gov't, Non-P.H.S. PMID: 139400 [PubMed - indexed for MEDLINE] 28: Curr Psychiatr Ther. 1982;21:225-39. Psychiatric aspects of infectious diseases. Autism Studies & Related Medical Conditions – TACA © Page 123
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Autism Studies & Related Medical Co
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Values of free and total carnitine
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observation suggests that certain m
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with autism. These data suggest tha
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Markers rs2056202 and rs2292813 wer
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2- Autism and Gastrointestinal Infl
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than in nondisease controls (p
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Buie, T. M. (2005). "Gastroesophage
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control children and significant nu
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Horvath, K. and J. A. Perman (2002)
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components indicates a role of NFH
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patients with Crohn disease, one of
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analyzed by a registered pediatric
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oligonucleotide probes targeting pr
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would be effective in alleviating c
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demonstrate a focal CD8-dominated g
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Chlamydia pneumoniae and Streptococ
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vaccine (MCV), and once-exposed chi
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Wakefield, A. J., S. H. Murch, et a
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Walker SJ, Hepner K, Segal J, Krigs
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R, Maisawa S, Miki K. Guidelines fo
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approximately 73 and 37kDa in plasm
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Cook, E. H., Jr., B. D. Perry, et a
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evealed, that AAb level may serve a
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findings, which consisted of obstru
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angiography was performed in all fo
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matched non-autistic siblings had d
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Todd, R. D. and R. D. Ciaranello (1
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eaction was further confirmed by DO
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asic protein (MBP) and glial acidic
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exhibit higher than average levels
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Feasby, T., B. Banwell, et al. (200
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each group participated. Observatio
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Rossignol, D. A., L. W. Rossignol,
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Page 107 and 108: 7 - Autism & Infection - 22 citatio
Page 109 and 110: micro-organisms. The faecal flora o
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Page 115 and 116: disorder. Yamashita Y, Fujimoto C,
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Page 127 and 128: to contribute to metal-induced neur
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Huebner FR, Lieberman KW, Rubino RP
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Four aspects of clinical evidence f
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Dohan FC: Wheat "consumption" and h
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from 24 children with autism from o
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administration on certain disorders
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(significantly lower ratio of SAM t
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Fifty-three controlled trials of th
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scavenge reactive species, then the
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hyperactivity in patients with ADHD
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improvement may only become apparen
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Walsh WJ, Glab LB, Haakenson ML. Re
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use other internal or external stan
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increased in affected children comp
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