14th ICID - Poster Abstracts - International Society for Infectious ...

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When citing these abstracts please use the following reference: Author(s) of abstract. Title of abstract [abstract]. Int J Infect Dis 2010;14S1: Abstract number. Please note that the official publication of the International Journal of Infectious Diseases 2010, Volume 14, Supplement 1 is available electronically on http://www.sciencedirect.com Final Abstract Number: 25.004 Session: Clinical Bacterial Infections Date: Wednesday, March 10, 2010 Time: 12:30-13:30 Room: Poster & Exhibition Area/Ground Level Type: Poster Presentation Salivary IgA responses in newborn against pathogen of oral cavity. Influence of prematurity in this response R. D. Nogueira, M. L. T. Sesso, M. C. L. Borges, L. R. Roberti, V. P. L. Ferriani University of Sao Paulo - Faculty of Medicine of Ribeirao Preto, Ribeirao Preto, SP, Brazil Background: Analysis of the mucosal immune system represents an interesting way to understand the microbial colonization in early life, particularly the response of SIgA (Secretory IgA) present in saliva because it represents the first line of defense. Previous study showed in children with 6 months of age, high complexity of SIgA response to antigens of Streptococcus mutans (MS), main pathogen of the dental caries, but little is known about the ontogeny of the mucosal immune system in the first day of life especially in preterm newborn (below 37 weeks of gestation). Thus, we compared the levels and specificity of SIgA to MS and others species enrolled with initial infection in fullterm (FT) and preterm (PT) early in life. Methods: Stimulate saliva from 160 children, with 0 day of life, were enrolled in this study. Salivary IgA and IgM levels were determined by ELISA. Subsets of 24 fullterm (FT) and 24 Preterm (PT) children showing similar salivary IgA levels were paired and matched for gender, racial background, breastfeeding. SIgA antibody reactivity to MS, Streptococcus sanguinis (SSA), Streptococcus mitis (SMI) and Streptococcus gordonii (SGO) Ags was determined in Western blot assays. Results: Levels of SIgA were statistically different (Whitney test, p
When citing these abstracts please use the following reference: Author(s) of abstract. Title of abstract [abstract]. Int J Infect Dis 2010;14S1: Abstract number. Please note that the official publication of the International Journal of Infectious Diseases 2010, Volume 14, Supplement 1 is available electronically on http://www.sciencedirect.com Final Abstract Number: 25.005 Session: Clinical Bacterial Infections Date: Wednesday, March 10, 2010 Time: 12:30-13:30 Room: Poster & Exhibition Area/Ground Level Type: Poster Presentation Infectious complications of venomous snakebite: 2 cases from Eastern Nepal S. K. Sharma, S. Shrestha, B. Badhu, C. S. Agrawal, B. Khanal BP Koirala Institute of Health Sciences, Dharan, Nepal Background: Venomous snakebite is a common and deadly disease throughout the tropics, but there are few reports of infections associated with snake envenomation in Asia.We describe two patients with distinct infectious syndromes following venomous snakebites in Eastern Nepal. Methods: Two case reports. Results: Case 1: Endopthalmitis and necrotizing fasciitis. A 21 year old female victim of snake bite (common cobra) to the foot presented within 30 minutes and received 24 ampoules of antivenom (Bharat Serum, India) over 2 days. After 18 hrs she developed dusky, ascending wound discoloration associated with (pain, blisters, fevers, and leukocytosis). Necrotizing fasciitis was diagnosed and treated with antibiotics and surgical debridement. On hospital day 2nd, the patient developed right eye pain and blindness. Analysis of vitreous humor aspirate revealed gram negative bacilli, and intravenous and ocular antibiotics were administered; aspirate cultures revealed growth of Serratia marcescens. The patient was discharged after 21 days, with moderate foot dysfunction and total eye blindness. Case 2: Local soft tissue infection with Enterococcus. A 62 years old alcoholic male presented within 1 hour after snakebite to the right hand from an unknown species. He had applied two tourniquets (to right forearm and arm) that were loosened in the emergency ward. He developed mild local swelling and neurological symptoms (ptosis, opthalmoplegia) within two hours of the bite, and received 200 ml of antivenom (Bharat serum, India) over 2 hours. (Although the neurologic symptoms resolved) the patient subsequently developed fevers and the hand swelling increased over 2 days. The wound developed gangrene and purulence over requiring surgical debridement. Wound cultures grew Enterococcus species, and he received gentamicin, metronidazole, and vanomycin. He was discharged on day 18th with moderate loss of hand functions. Conclusion: Snakebite-associated infection in the tropics can manifest variously, with local, regional and remote effects on injured tissues. More studies are warranted for this important complication of envenomation.
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