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Vol. 60, 1909 - University of North Carolina at Chapel Hill

Vol. 60, 1909 - University of North Carolina at Chapel Hill

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EDITORIAL. 99<br />

logically wh<strong>at</strong> hitherto has been a puzzle cardia, much though they may differ in<br />

to clinicians. This theoiy assumes th<strong>at</strong> their an<strong>at</strong>omical found<strong>at</strong>ion. Any and<br />

inflamm<strong>at</strong>ory processes<strong>of</strong> the cardiac plexus every irrit<strong>at</strong>ion wiihin th<strong>at</strong> circle may cause<br />

are the cause <strong>of</strong> the affection, and this, in alarm in the heart. In this waj' spinal afmany<br />

cases, has been supported by the fact fections (tabes), injuries to the dorsal<br />

th<strong>at</strong> lesions were found in the nerves <strong>of</strong> the nerves and the rami communicantes, affeccardiac<br />

plexus. If we consider th<strong>at</strong> both tions <strong>of</strong> the nervus cardiacus magnus, neuvasoconstricting<br />

and vasodil<strong>at</strong>ing fibres <strong>of</strong> ritic affections <strong>of</strong> the cardiac and coronary<br />

the coronary area termin<strong>at</strong>e in the cardiac plexus, and finally embolism and tliromplexus<br />

and th<strong>at</strong> these fibres connect with bosis <strong>of</strong> the coronary arteries, may produce<br />

the spinal cord through the intermediary <strong>of</strong> the symptom complex <strong>of</strong> angina pectoris,<br />

the symp<strong>at</strong>hetic ganglia and the rami com- The result will be the same in all cases; in<br />

municantes; if we keep in mind th<strong>at</strong> the the center pain and anguish, in tlie periexcil<strong>at</strong>ioiis,<br />

which start from the centers phery vascular spasm with ischsemia <strong>of</strong> the<br />

for the vascuhir muscul<strong>at</strong>ure, proceed from myocardium. A healthy heart will overthe<br />

gray substance to the anterior roots and come such an <strong>at</strong>tack, even though it be<br />

from therethrough the rami communicantes serious, but if the heart muscle is weak and<br />

to the symp<strong>at</strong>hetic, the possibility suggests reduced in efficiency, whether from deficient<br />

itself th<strong>at</strong> the sensory fibres carry thesensa- nutrition or f<strong>at</strong>ty degener<strong>at</strong>ion, myocarditis<br />

tion <strong>of</strong> pain in a centripetal direction, or adhesions to tlie pericardium, then even<br />

whereas the excit<strong>at</strong>ion <strong>of</strong> the centrifugal a rudimentary vascular spasm in the heart<br />

fibres is manifested as a vascular spasm in muscle may lead to a c<strong>at</strong>astrophe, just as a<br />

the cardiac vessels. This would complete f<strong>at</strong>ty degener<strong>at</strong>ed heart may suddenly cease<br />

a circle, in which not only primary excita- to work without the presence <strong>of</strong> an acute<br />

tion, but also reflex processes may be mani- ischsemia.<br />

fested in the course <strong>of</strong> the rami communicantes<br />

through the connection <strong>of</strong> the spinal<br />

cord with the symp<strong>at</strong>hetic. This system<br />

B1.00D PRESSURE AND UIFC<br />

IMSIJRANOE.<br />

also includes the pneumogastric nerve. The The practice <strong>of</strong> ascertaining the blood<br />

investig<strong>at</strong>ions <strong>of</strong> Maass have shown th<strong>at</strong> pressure has come to be recognized as <strong>of</strong><br />

vasoconstrict(»r fibres for the coronary artery prime importance. Its significance in the<br />

termin<strong>at</strong>e in the pneumogastric nerve, and making up <strong>of</strong> prognoses has within the past<br />

since it is known th<strong>at</strong> the rami communi- few years been impressed very forcibly upcantes<br />

effect the connection <strong>of</strong> the sympa- on all genuinely scientific clinicians. Its<br />

theticus with the cervico-brachial plexus, a importance in this respect should be realized<br />

typical stenocardiac <strong>at</strong>tack would be ex- especially in considering the acceptance <strong>of</strong><br />

plained by any irrit<strong>at</strong>ion <strong>of</strong> th<strong>at</strong> system, applicants for life insurance. Dr. Ralph<br />

In order to bring about a stenocardiac <strong>at</strong>- (irace, in an article which appeared recenttack<br />

it would therefore be a m<strong>at</strong>ter <strong>of</strong> in- ly in the Xew York Medical Journal, emdifl'erence<br />

whether the caus<strong>at</strong>ive irrit<strong>at</strong>ion phasized this idea in forcible terms. To<br />

starts in the end appar<strong>at</strong>us—th<strong>at</strong> is the estim<strong>at</strong>e correctly the pressure <strong>of</strong> the blood<br />

sensitive organs <strong>of</strong> the coronary arteries—in in the vessels the most important evidence<br />

the transit st<strong>at</strong>ion—thalis the cardiac nerves is <strong>at</strong>tained by the use <strong>of</strong> the sphygmoma-<br />

—in the rami communicantes, or finally in nometer. Of this instrument there are<br />

the central appar<strong>at</strong>u.s—th<strong>at</strong> is the spinal many reliable makes, among which might<br />

cord. The irrit<strong>at</strong>ion <strong>of</strong> the sensitive con- be mentioned the Janeway, Riva-Rocci, &c.<br />

Jiections v.ould therefore produce pain, and As is well known, blood pressure is estith<strong>at</strong><br />

<strong>of</strong> the centrifugal connections ( vaso- tu<strong>at</strong>ed by the force necessary to compress<br />

motor I ischsemia <strong>of</strong> the myocardium. Ac- the artery and obliter<strong>at</strong>e the pulse wave becording<br />

lo wliich <strong>of</strong> these components pre- yond the point <strong>of</strong> compression. This force<br />

vails, tiie polymorphic character <strong>of</strong> the is measured in millimetres <strong>of</strong> mercury,<br />

stenocardiac paroxysms and the j)henomena In arteriosclerosis we have a condition in<br />

<strong>of</strong> radi<strong>at</strong>ion would be susceptible <strong>of</strong> inter- the vessel wall which is best described as<br />

prel<strong>at</strong>ion. The anguish could be exjilained diffuse permanent thickening, or a hyperas<br />

being due to the centripetal connections trophy<br />

and to the vagus, owing to an irrit<strong>at</strong>ion <strong>of</strong> artery.<br />

<strong>of</strong> the muscular elements <strong>of</strong> the<br />

There may be a thickening <strong>of</strong> the<br />

the oblong<strong>at</strong>a. F.et us keep the complete external co<strong>at</strong> or <strong>of</strong> the media, and should<br />

circle before our eyes; the spinal center <strong>of</strong> be distinguished from <strong>at</strong>heroma which is a<br />

innerv<strong>at</strong>ion, the dorsal nerves, the rami focal affection <strong>of</strong> the arterial wall affecting<br />

communicantes, the cardiac nerves, the separ<strong>at</strong>e limited areas, probably <strong>of</strong> microaortic<br />

plexus and the coronary plexus with organic origin and usually a disease <strong>of</strong> the<br />

the sensitive terniin.Tl organs in their vas- aged. In simple arteriosclerosis the blood<br />

cular walls. We shall be able to trace to pressure is not gre<strong>at</strong>ly raised as the increasone<br />

common basis all the types <strong>of</strong> steno- ed resistance <strong>of</strong> the thickened vessel wall is

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