Thursday-Abstracts

Recommendations

Info

Official Journal of the American College of Sports Medicine rEsuLTs: On-ice practice time gradually increased as the season progressed from 102.0 ± 0 minutes preseason to 131.2 ± 23.4 minutes during the final month. Off-ice training time decreased from 141.4 ± 66.4 minutes preseason to 31.0 ± 27.8 minutes during the final month. Intensity of both on and off-ice training decreased from preseason into the season and as the season progressed (percentage of on-ice practices at 90-100% of maximal HR: 15.6 ± 10.6 preseason vs. 1.2 ± 1.7 final month, percentage of off-ice training sessions from 90-100% of maximal HR:10.1 ± 9.3 preseason vs. 1.3 ± 4.2 final month). Percentage of game time spent in each HR zone indicated similar intensity for each period, including 9% of game time spent at 90-100% of maximal HR. Preseason off and on-ice training/practices most closely resembled game HR zone averages. CONCLusION: Intensity of preseason training closely simulates game intensity. Inseason training is less intense than games and decreases as the season progresses. 1765 May 30, 4:30 PM - 4:45 PM Tracking Changes In Body Composition Within Elite Wheelchair Basketball Players Mhairi Keil1 , Alan M. Batterham, FACSM2 , Vicky Goosey- Tolfrey3 . 1English Institute of Sport/Loughborough University, Birmingham, United Kingdom. 2Teesside University, UK, Middlesborough, United Kingdom. 3Loughborough University, Loughborough, United Kingdom. (No relationships reported) Practical methods for monitoring body composition changes are important when preparing wheelchair basketball players for competition. PurPOsE: To evaluate the tracking of within-athlete changes in criterion measures of body composition (dual energy X-ray absorptiometry; DXA) with simple skinfold thickness measures over a 15-month competitive training period. METhOds: Participants were male Great Britain Wheelchair Basketball players (n=16) who underwent body composition assessments at 5 time points over 15 months. The primary outcome was DXA-derived body fat percentage, with sum of 4, 6, or 8 skinfolds (mm) as the predictor variable. Data were analysed using a linear mixed model (random slope and intercept) to derive the within-athlete typical error of the estimate in predicting criterion body fat percentage from sum of skinfolds. This typical error allowed us evaluate how well a simple measure of the sum of skinfolds could track criterion changes in body fat percentage; that is, we derived the change in sum of skinfolds that would have to be observed in an individual athlete to conclude that a substantial change in body fat percentage had occurred (greater than the minimum important difference given by a standardised mean difference of 0.2 standard deviations). All data were log-transformed prior to analysis. rEsuLTs: The sum of 8 skinfolds was the most precise practical measure for tracking changes in body fat percentage. The within-subject correlation (r) between the sum of 8 skinfolds and DXA body fat percentage was 0.86 (95% confidence interval, 0.81 to 0.90). For the monitoring of an individual male wheelchair basketball player, a change in sum of 8 skinfolds by a factor of greater than 1.13 (multiply or divide by 1.13) is associated with a practically meaningful change in body fat percentage. For example, an athlete whose sum of 8 skinfolds decreased from 114 mm to 100 mm (a reduction greater than 114/1.13) could be said to have become leaner. Reductions in sum of skinfolds lower than this value would be considered trivial, given the typical prediction error observed. CONCLusIONs: A measure of the sum of 8 skinfolds can track changes in body fat percentage within individual wheelchair athletes with adequate precision, providing a useful field monitoring tool in the absence of often impractical criterion measures. 1766 May 30, 4:45 PM - 5:00 PM differences In skeletal Muscle Contractile Properties among Power and Endurance Elite athletes Irineu Loturco1 , Hamilton Roschel2 , Carlos Ugrinowitsch2 , Cristiano Laurino3 , Saulo Gil1 , Ronaldo Kobal1 , Valmor Tricoli2 . 1Pão de Açúcar Group - Nucleus of High Performance in Sports, São Paulo, Brazil. 2University of São Paulo, São Paulo, Brazil. 3BM&F - Bovespa Track & Field Club, São Paulo, Brazil. (No relationships reported) Measurements of muscle belly responses to electrical stimuli using a displacement sensor provide valuable information about the skeletal muscle contractile properties. In this regard, it has been proposed that muscle contraction time (Tc) could be used as an accurate non-invasive predictor of fiber type composition. Additionally, the muscle radial displacement (Dm) is considered as a reliable measurement of muscle stiffness. Thus, it is conceivable that power and endurance elite athletes present significant differences between these skeletal muscle mechanical properties. PurPOsE: The purpose of this study was to evaluate if there are significant differences in muscle contractile properties among power and endurance elite athletes. METhOds: Thirty-four elite male track and field athletes were divided into two different groups: power athletes (PA) (sprinters and jumpers; n=18; age: 26 ± 3, 8 years; height: 179 ± 6, 8 cm; weight: 77, 8 ± 8, 2kg) and endurance athletes (EA) (endurance runners and triathletes; n=16; 26, 4 ± 7, 2 years; height: 168 ± 9, 2 cm; Vol. 45 No. 5 Supplement S341 weight: 61 ± 12, 2 kg). Dm and Tc were measured in both the rectus femoris (RF) and the biceps femoris (BF) muscles from the dominant leg, using a Tensiomyographer device (TMG Measurement System, TMG-BMC Ltd., Ljubljana, Slovenia). Unpaired t-tests were used to compare the differences between the groups. Statistical significance was set at P
<strong>Thursday</strong>, May 30, 2013 S342 Vol. 45 No. 5 Supplement D-59 Clinical Case Slide - Cardiovascular May 30, 2013, 3:15 PM - 5:15 PM Room: 103 1768 Chair: Aaron L. Baggish. Massachusetts General Hospital, Boston, MA. (No relationships reported) 1769 discussant: Sandra J. Hoffmann, FACSM. Idaho State University, Pocatello, ID. (No relationships reported) 1770 discussant: Warren B. Howe, FACSM. Bellingham, WA. (No relationships reported) 1771 May 30, 3:15 PM - 3:35 PM sudden Cardiac death--running Kenneth Leclerc, James Watts, Mark Peele, Kevin Steel, Michael Rose. San Antonio Military Medical Center, Fort Sam Houston, TX. (No relationships reported) hIsTOry: A 51-year-old male had sudden loss of consciousness while running on a treadmill at the gym. He had been evaluated 4 months prior for a neurocardiogenic syncopal event following a colonoscopy. Patient found to be without a pulse. Bystander CPR was performed and the use of an automated external defibrillator (AED) with successful restoration of a regular pulse. Interrogation of the AED revealed ventricular fibrillation appropriately defibrillated to normal sinus rhythm. PhysICaL EXaMINaTION: Heart rate 60, Blood pressure 130/81, Respiratory rate 12. His breath sounds were clear to auscultation. His heart was regular in rate and rhythm, normal S1 and S2 without murmur. He had no elevation in his jugular venous pulsation and was without lower extremity edema. PhysICaL EXaMINaTION: 1. Acute coronary syndrome 2. Arrhythmogenic right ventricular cardiomyopathy 3. Catecholaminergic polymorphic ventricular tachycardia 4. Brugada syndrome 5. Long QT syndrome 6. Torsades de Pointe TEsT aNd rEsuLTs: EKG --Normal sinus rhythm, right axis deviation, and right bundle branch block Echocardiogram --Normal left ventricular size, wall thickness and function with moderate dilation of the right ventricle and right atrium. Coronary Angiography --Luminal irregularities Cardiac MRI --Mildly dilated right ventricle with a mildly low right ventricular ejection fraction of 46.4% without other evidence for arrhythmogenic right ventricular dysplasia Electrophysiology Study --No accessory pathway found, no evidence of dual AV node physiology, and normal corrected QT interval after epinephrine infusion. A monomorphic ventricular tachycardia occurred after high dose (20mcg/min) isoproterenol infusion Genetic Testing --4 novel gene mutations coding for sarcomeric proteins (actinin, myosin, and titin). FINaL WOrKING dIaGNOsIs: Catecholamine sensitive ventricular tachycardia with aborted sudden death secondary to apparent RV cardiomyopathy TrEaTMENT aNd OuTCOMEs: 1. AICD placed for secondary prevention of sudden cardiac death 2. Suppressive long-acting beta-blocker therapy 3. Cardiac rehabilitation without recurrence of symptoms 4. Genetic counseling for the patient and first degree relatives 5. Further evaluation as to the role that the genetic variants may play into malignant arrhythmias 1772 May 30, 3:35 PM - 3:55 PM syncope and abnormal Electrocardiogram in an adolescent Basketball Player Jodi L. Zilinski, Miranda E. Contursi, Patricia A. Lowry, Caitlin H. O’Callaghan, Michael A. Fifer, Gregory D. Lewis, Aaron L. Baggish. Massachusetts General Hospital, Boston, MA. (No relationships reported) hIsTOry: A 17 year old Caucasian male basketball player presented following a syncopal episode, which occurred during basketball practice. He had just completed MEDICINE & SCIENCE IN SPORTS & EXERCISE ® sprinting drills and was walking to the bench when he felt lightheaded and lost consciousness for approximately 30 seconds. He sustained no injuries. He had recently completed a course of azithromycin for an upper respiratory tract illness. He denied antecedent chest pain, palpitations, and prior syncope. PhysICaL EXaMINaTION: Vital signs: Ht 1.78 meters, Wt 68.0 kg, Heart rate 50 bpm, Blood Pressure 112/62 mmHg Healthy appearing. Lungs clear. Bradycardic regular rate, normal S1 and S2 without murmur or gallop. Nonfocal neurologic examination. PhysICaL EXaMINaTION: Neurally mediated syncope Cardiac arrhythmia Anomalous origin of coronary artery Structural heart disease (HCM, ARVC) Athletic remodeling Myocarditis TEsT aNd rEsuLTs: ECG: (Fig. a) - Sinus bradycardia - QTc 428 ms - Increased precordial voltage - Diffuse repolarization abnormalities Echocardiogram: - LV- asymmetric hypertrophy (LV mass 107 g/m 2 , wall measurements: interventricular septum 9 mm, posterior wall thickness 12 mm) - RV- normal structure and function - Coronary Arteries- well visualized, normal coronary origins and proximal courses Cardiac MRI: (Fig. B) -LV mass 115 g/m 2 - Asymmetric LV hypertrophy (max wall thickness = 15 mm in inferior region) - No T2 weighted edema, early or late gadolinium enhancement - No RV hypertrophy Cardiopulmonary Exercise Test (CPET): - Exercise time: 12:54 min - Peak workload 196 Watts - Maximal oxygen uptake (VO2) 33.4 ml·kg -1 ·min -1 (75% predicted) - Decreased peak O2 pulse consistent with abnormal stroke volume augmentation - No arrhythmias WOrKING dIaGNOsEs: Hypertrophic cardiomyopathy vs. Athletic remodeling with neurally mediated syncope TrEaTMENT aNd OuTCOMEs: 1. Prescribed detraining x 6 months 2. Repeat EKG (Fig. C): Persistent although less marked repolarization abnormalities 3. Repeat Cardiac MRI (Fig. d): Marked regression of LV wall thickness (max = 10 mm in inferolateral region) and LV mass 79 g/m 2 (Δ = -36 g/m 2 , % Δ = -31%) Discussion with patient and family regarding prognostic uncertainties surrounding “bizarre” ECG patterns in the absence of structural heart disease. Ultimately cleared for participation with close follow-up. 1773 May 30, 3:55 PM - 4:15 PM decreased Ejection Fraction - Baseball Peter H. Seidenberg, Wayne J. Sebastianelli. Penn State University, State College, PA. (Sponsor: Francis G. O’Connor, FACSM) (No relationships reported) hIsTOry: A 19 year old freshman baseball catcher underwent routine screening 2D echocardiography as part of his initial preparticipation evaluation. He reports no history of chest pain, syncope, dizziness, palpitations, or shortness of breath. His past medical history is unremarkable. There is no history of recent illness. There is a history of a severe flu-like illness 2-3 years ago, for which he did not seek medical attention. There is no significant family medical history. He has no current or prior history of smoking, alcohol use, drug use, or anabolic steroid use. ACSM May 28 - June 1, 2013 Indianapolis, Indiana
Page 1 and 2:
Thursday, May 30, 2013 S176 Vol. 45
Page 3 and 4:
Page 5 and 6:
Page 7 and 8:
Page 9 and 10:
Page 11 and 12:
Page 13 and 14:
Page 15 and 16:
Page 17 and 18:
Page 19 and 20:
Page 21 and 22:
Page 23 and 24:
Page 25 and 26:
Page 27 and 28:
Page 29 and 30:
Page 31 and 32:
Page 33 and 34:
Page 35 and 36:
Page 37 and 38:
Page 39 and 40:
Page 41 and 42:
Page 43 and 44:
Page 45 and 46:
Page 47 and 48:
Page 49 and 50:
Page 51 and 52:
Page 53 and 54:
Page 55 and 56:
Page 57 and 58:
Page 59 and 60:
Page 61 and 62:
Page 63 and 64:
Page 65 and 66:
Page 67 and 68:
Page 69 and 70:
Page 71 and 72:
Page 73 and 74:
Page 75 and 76:
Page 77 and 78:
Page 79 and 80:
Page 81 and 82:
Page 83 and 84:
Page 85 and 86:
Page 87 and 88:
Page 89 and 90:
Page 91 and 92:
Page 93 and 94:
Page 95 and 96:
Page 97 and 98:
Page 99 and 100:
Page 101 and 102:
Page 103 and 104:
Page 105 and 106:
Page 107 and 108:
Page 109 and 110:
Page 111 and 112:
Page 113 and 114:
Page 115 and 116: Thursday, May 30, 2013 S290 Vol. 45
Page 165: Thursday, May 30, 2013 S340 Vol. 45
show all

Thursday-Abstracts

You also want an ePaper? Increase the reach of your titles

Delete template?

Save as template?