TiHo Bibliothek elib - Tierärztliche Hochschule Hannover

Untersuchung des Chemokins MIP-3 β/CCL19
SCAPINI, P., C. LAUDANNA, C. PINARDI, P. ALLAVENA, A. MANTOVANI, S.
SOZZANI u. M. A. CASSATELLA (2001):
Neutrophils produce biologically active macrophage inflammatory protein-3α (MIP-
3α)/CCL20 and MIP-3β/CCL19.
Eur. J. Immunol. 31, 1981–1988
SCHWARTZ, M., P. F. MOORE u. A. TIPOLD (2008b):
Disproportionally strong increase of B cells in inflammatory cerebrospinal fluid of
dogs with Steroid-responsive Meningitis-Arteritis.
Vet. Immunol. Immunopathol. 125, 274-283
SCHWARTZ, M., C. PUFF, V. M. STEIN, W. BAUMGARTNER u. A. TIPOLD (2011):
Pathogenetic factors for excessive IgA production: Th2-dominated immune response
in canine steroid-responsive meningitis-arteritis.
Vet. J. 187, 260-266
SERAFINI, B., R. MAGLIOZZI, S. COLUMBA-CABEZAS, E. AMBROSINI u. F.
ALOISI (2001):
Recruitment of dendritic cells and occurrence of lympoid- like features in the central
nervous system during autoimmune encephalomyelitis.
J. Neuroimmunol. 118, 5
SHIBUYA, M., N. MATSUKI, K. FUJIWARA, S. IMAJOH- OHMI, N. T. PHAM, S.
TAMAHARA u. K. ONO (2007):
Autoantibodies against glial fibrillary acidic protein (GFAP) in cerebrospinal fluids
from
Pug dogs with necrotizing meningoencephalitis.
J. Vet. Med. Sci. 69, 241–245
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