Eisen-Suppressed-Inventions-and-other-Discoveries-True-Stories-of ...

The Suppression of Alternative Medical Therapies 57
of the facilities at America's premier biological weapons testing center,
Fort Detrick in Frederick, Maryland. 12 Perhaps not coincidentally, the Cell
Tumor Biology Laboratory's output increased in 1971 as measured by the
publication of eight scientific articles by Gallo and co-workers compared
to at most four in previous years. These reports included Gallo's discovery
that by adding a synthetic RNA and feline (i.e., cat) leukemia virus
(FELV) "template" to "human type C" viruses (associated with cancers of
the lymph nodes), the rate of DNA production (and subsequent provirus
synthesis) increased as much as thirty times. The NCI researchers reported
that such a virus may cause many cancers besides leukemias and lymphomas
including sarcomas. 13
In this 1971 report Gallo et al. also reported modifying simian (i.e.,
monkey) viruses by infusing them with cat leukemia RNA to make them
cause cancers as seen in AIDS patients. 13
Furthermore, Fujioka and Gallo concluded from studies conducted in
late 1969 or early 1970 that they would need to further "evaluate the functional
significance of tRNA changes in tumor cells," by designing an
experiment in which "specific tumor cell tRNAs" would be "added directly
to normal cells." They explained that one way of doing this was to use
viruses to deliver the foreign cancer producing tRNA to normal cells. The
viruses which were then employed to do this, the researchers noted, were
the simian virus (SV40) and the mouse parotid tumor (polyoma) virus. 14
Such experiments clearly advanced immunodeficiency virus technology
and even provided a model for the development of HIV, the AIDS
virus—allegedly of simian virus descent—which similarly delivered
unique enzymes and a foreign RNA to normal cells necessary to cause an
acquired immunodeficiency syndrome in animals and humans.
DEVELOPING MORE AIDS-LIKE VIRUSES
In 1972, Gallo, his superiors and inferiors studied portions of simian
viruses to determine differences in RNA activity between infected versus
uninfected cancer cells, and whether the differences could be ascribed to
the infection and related DNA alterations. 15 They stated that "by studying
viral or cellular mutants or cell segregants . . . which have conditional
variations in virus-specific cellular alterations, it should be possible to
more precisely determine the biological significance of the RNA variation
reported here."
Clearly, the group was working to determine the relevance of various
viral genes on the development of human cancers and immune system collapse.
They reported their desire to use this information to find a cure for
cancer, but at this time their activity was more focused on creating various
cancers as well as new carcinogenic viruses which could infect humans.