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Treatment Regime: This varies with a number of factors, some of which will be more important than others in different individuals: 1. Site 88 1. Site 2. Affect on the horse 3. Affect to the owner (is it a show horse?) 4. Response (or lack of) to previous therapy 5. Extent (area) and severity (depth) of the lesions 6. Owners ability to treat / manage 7. Environmental conditions 8. Monies available for therapy I associate various sites with various possible underlying causes and as such, the treatment regime will include a treatment modality aimed at the presumed, or diagnosed underlying cause, as well as addressing the folliculitis. Tack areas: such as underneath bridle cheek straps, girths, saddles, cruppers. These areas have a naturally thicker skin than many other areas and so frequently the folliculitis +/- associated furunculosis will require a longer course of systemic antibiotics. I do not expect these infections to heal well without systemic antibiotics and also without rest from the presence of the overlying tack. Completely spelling a horse for the 3 to 4 weeks the folliculitis may require to resolve can be catastrophic from the point of view of a working horse (owner), however I expect that any external trauma to these areas during that time will extend the recovery period. Lambskin pads, utilising different tack, frequent surface washes may all help to minimise this trauma IF the horse must be worked. Pastern: frequently associated with environmental maceration, which needs to be corrected before one can honestly expect a recovery. However there are the other implicated factors such as UV-light and a contact hypersensitivity (whether that is mediated through a UV-sensitisation is still discussed). The lower leg swelling associated with a lymphadenopathy in “Draught horses” can also be associated initially with a pastern folliculitis. These are often deep, fissured and associated with a furunculosis which requires both topical and systemic therapy for the bacterial component. Generalised: may be associated with underlying stress or allergy factors, which will ned to be addressed concurrently. Due to the large body area affected, these will also require a systemic course, but typically respond fast. ACVSC Proceedings Dermatology Chapter Science Week 2005
2. Affect on the horse and 3. Affect to the owner The degree with which the dermatitis is interfering with work, the cosmetic appearance and the simple well being of the horse will all be factors which affect just how intensive the chosen treatment will be. These lesions are frequently painful and may turn a placid creature into a demon. If the horse is a work or show horse and the lesions are in the tack area, then the consequences are even more dramatic Owners will be affected emotionally, by the change in personality of their beloved pet, and by the potential inability of their horse to fulfil its “job”. ACVSC Proceedings Dermatology Chapter Science Week 2005 89
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Australian College of Veterinary Sc
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Australian College of Veterinary Sc
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3.00 Afternoon tea 3.30 How I treat
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Dr Jennifer A. Charles BVSc MVS Dip
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Scientists in Feline Medicine in 19
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IgE-bearing cells have been demonst
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years, median age 6.5 years, with a
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dusts, grain smuts, moulds, danders
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References: Evans AG, Paradis MR, &
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Comparatively simple, cheap and eff
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TABLE 1: CURRENT ALLERGENS SELECTED
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AmericanCockroach Cockroach mix
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Patient Preparation Drug Withdrawal
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Each injection site is visually exa
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References 1. Scott DW, Miller WH:
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Intradermal Testing (IDT) in Horses
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TABLE 2 Additional allergens used i
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Allergen Positive reactions Mosquit
Page 37 and 38: much higher and corresponded to the
Page 39 and 40: and this and other uncontrolled tri
Page 41 and 42: Introduction Diagnostic Approach to
Page 43 and 44: Wheals vary in size and shape and q
Page 45 and 46: References Evans AG, Paradis MR & O
Page 47 and 48: Introduction: Equine Chronic Urtica
Page 49 and 50: Acute treatment - Epinephrine 1:100
Page 51 and 52: anecdotal success with use of human
Page 53 and 54: an 80:20 mixture of evening primros
Page 55 and 56: Clinical approach to the horse with
Page 57 and 58: identify plants in a pasture that m
Page 59 and 60: Equine sarcoidosis Sonya Bettenay T
Page 61 and 62: described as a possible feature of
Page 63 and 64: others 2001), isotretinoin(Waldinge
Page 65 and 66: 12. LeGall, F., Loeuillet, L., Dela
Page 67 and 68: Introduction The equine sarcoid: an
Page 69 and 70: Clinical Presentation Small fibrous
Page 71 and 72: Assessment should be made on the fo
Page 73 and 74: (ix)Bone becomes devitalized if inc
Page 75 and 76: and mitomycin C have been used. All
Page 77 and 78: Owen LN & Barnett KC (1983) - Treat
Page 79 and 80: any anatomic location but commonly
Page 81 and 82: epidermis may be histologically nor
Page 83 and 84: References D.C. Knottenbelt, S. Edw
Page 85 and 86: indicated. Each sample for culture
Page 87: Proper dosage and proper duration a
Page 91 and 92: Affected horses usually scratch and
Page 93 and 94: • Cattle ear tags impregnated wit
Page 95 and 96: How I treat: equine pemphigus folia
Page 97 and 98: Treatment of pemphigus foliaceus in
Page 99 and 100: SCOTT, D.W., WALON, D.K., SLATER, M
Page 101 and 102: Diagnosis History of access of hors
Page 103 and 104: These vaccines are not manufactured
Page 105 and 106: References Alfaro AA & Mendoza L (1
Page 107 and 108: y a variety of fungal metabolites.
Page 109 and 110: Authors recommendations: 1. Both Im
Page 111 and 112: chorioptic mange by the author (Lit
Page 113 and 114: Clinical Disease The clinical resul
Page 115 and 116: treatment options have been used. P
Page 117 and 118: Notes ACVSC Proceedings Dermatology
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