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Descriptive Psychopathology: The Signs and Symptoms of ...

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180 Section 3: Examination domainsMany other classic symptoms <strong>of</strong> hysteria are shown to result from specific disease.At one time, Parkinson’s disease, St Vitus’ dance, tetanus <strong>and</strong> eclampsia wereconsidered neurotic disorders. 111 Denial <strong>of</strong> illness, la belle indifference, <strong>and</strong>hemianesthesia,classic signs <strong>of</strong> conversion, are associated with thalamic disease. Manynon-epileptic fits are in fact partial complex seizures identified by laboratory studies.Anatomically inconsistent pain patterns are <strong>of</strong>ten prominent early signs in multiplesclerosis, <strong>and</strong> astasia–abasia, the “classic” hysterical gait <strong>and</strong> walking difficulty, isassociated with dementia, 112 <strong>and</strong> midline cerebellar <strong>and</strong> corpus callosum lesions. 113Many examples <strong>of</strong> motor conversions <strong>and</strong> psychogenic movement disorder areunderstood as features <strong>of</strong> catatonia or specific neurologic disease. Patient 1.7, the78-year-old woman who was said to have psychogenic “confusion” <strong>and</strong> catatonia(diagnosed by a university hospital neurology team, <strong>and</strong> then a psychiatrist), wasfound to be in non-convulsive status epilepticus. Patient 4.1, the 56-year-old nursewho was said to have a psychogenic movement disorder (diagnosed by a psychiatricresident, his faculty supervisor, a neurologist, <strong>and</strong> several other psychiatry facultymembers), was found to have familial cerebellar–pontine degeneration.Patient 7.5 also illustrates the value <strong>of</strong> not assuming symptoms to be “psychogenic”even if they appear associated with so-called primary or secondary gain(psychological or tangible, respectively). 114Patient 7.5 115A 30-year-old woman was referred to a psychoanalyst by her primary carephysician because <strong>of</strong> neck <strong>and</strong> head pain <strong>of</strong> six months’ duration keeping herfrom going to work, <strong>and</strong> for which no medical cause was determined in acursory evaluation. She was pain-free most <strong>of</strong> the time, except when riding thecommuter train to work. <strong>The</strong> train ride took about 25min, <strong>and</strong> her pain beganshortly after she boarded <strong>and</strong> worsened as the train approached the terminus<strong>and</strong> her workplace, forcing her to return home. She had been at the job forabout six months, <strong>and</strong> found it “stressful”.Obtaining a careful history (the story <strong>of</strong> the illness), the psychoanalystdiscovered that by the time the patient boarded the train all the seats weretaken <strong>and</strong> she had to st<strong>and</strong> on the platform near the car exit, receiving the fullenergy <strong>of</strong> the car bouncing over the tracks. He surmised that the car’s up <strong>and</strong>down movement affected a cervical spine abnormality leading to nerve rootinflammation <strong>and</strong> the pain. This was confirmed on CT scan <strong>and</strong> a neck braceworn during the commute resolved the patient’s difficulties.Patient 7.6 also illustrates the dangers <strong>of</strong> automatically accepting the patient’ssymptoms as “psychogenic” if they do not fit the examiner’s underst<strong>and</strong>ing <strong>of</strong>neuroanatomy <strong>and</strong> function.

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