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PATHOLOGY OF THE LIVER AND PORTAL VENOUS SYSTEM 101<br />

+34.9MM<br />

NORMAL<br />

1442 HZ<br />

C<br />

E<br />

Figure 4.21 cont’d (C) Balanced PV flow. Alternate<br />

forward and reverse low-velocity flow on the Doppler<br />

spectrum. The PV colour Doppler alternates red and blue.<br />

(D) PV thrombosis. The PV is dilated (arrows) and filled<br />

with thrombus. A collateral vessel is seen anterior to<br />

this—not to be confused with the PV—as this is a source<br />

of false-negative ultrasound results. (E) Non-dilated,<br />

thrombosed PV (arrow) with collaterals demonstrated on<br />

power Doppler.<br />

D<br />

other causes, including inflammatory or<br />

malignant conditions which may surround,<br />

compress or invade the portal and/or splenic<br />

veins (Box 4.1). The thrombosis may be total<br />

or partial.<br />

● hepatopetal main PV flow with hepatofugal<br />

peripheral flow may be a sign of HCC,<br />

requiring careful scanning to identify the<br />

lesion.<br />

● cavernous transformation. A network of<br />

collateral vessels may form around a<br />

thrombosed main portal vein at the porta,<br />

especially if the thrombosis is due<br />

to extrahepatic causes (for example

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