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220<br />
ABDOMINAL ULTRASOUND<br />
peak incidence before 5 years of age. It occurs in<br />
various sites throughout the body.<br />
Other causes of focal liver lesions<br />
Liver metastases may occur from most paediatric<br />
malignancies, particularly neuroblastoma, rhabdomyosarcoma<br />
and Wilms’ tumour (p. 229).<br />
Leukaemia and lymphoma may also cause focal<br />
defects in the liver. Liver involvement may be manifested<br />
by hepatomegaly with normal liver texture,<br />
a non-specific sign, or by diffuse coarsened liver<br />
texture with or without hepatomegaly.<br />
Haemangioendothelioma<br />
Vascular tumours account for most benign liver<br />
tumours in childhood, with haemangioendotheliomas<br />
being seen more frequently than cavernous<br />
haemangiomas. Although haemangioendothelioma<br />
may be asymptomatic, infants generally present<br />
before the age of 6 months with an abdominal<br />
mass, respiratory distress, anaemia and cardiac failure,<br />
caused by the shunting of blood from the<br />
aorta through the tumour. Large tumours may<br />
bleed spontaneously, resulting in haemoperitoneum.<br />
They may present with jaundice and<br />
increased transaminase levels and 50% of children<br />
also have cutaneous haemangioma. 10<br />
These tumours are generally multiple, of varying<br />
echogenicity and may have a complex echotexture<br />
due to thrombus, calcifications and internal septations<br />
(Fig. 9.3B). The vascular nature of these<br />
lesions is demonstrated by a large coeliac axis and<br />
marked decrease in the size of the aorta below the<br />
origin of the coeliac axis. The main differential<br />
diagnosis of multiple haemangioendothelioma is<br />
from metastatic liver disease, particularly from disseminated<br />
neuroblastoma.<br />
Although most asymptomatic paediatric haemangioendotheliomas<br />
regress spontaneously, those<br />
complicated by cardiac failure require active treatment.<br />
Steroids may be administered and serial<br />
ultrasound scans may be used to monitor the gradual<br />
resolution of the lesion. Angiographic<br />
embolization or surgical ligation of the major<br />
feeding vessels of the hepatic artery may be necessary<br />
in severe cases that fail to respond to steroid<br />
therapy.<br />
PANCREAS<br />
Normal appearances<br />
The acoustic characteristics of the pancreas vary<br />
with age. Pancreatic echogenicity is quite variable<br />
and is occasionally hypoechoic in neonates compared<br />
with the adult gland. In older children<br />
echogenicity is equal to or slightly greater than<br />
that of the liver. The pancreas is relatively larger in<br />
young children than in adults, gradually increasing<br />
with age, reaching adult size in late teens. 11 The<br />
pancreatic duct is often visualized but should not<br />
be greater than 2 mm in width. The relative<br />
hypoechogenicity and relatively larger size of the<br />
normal pancreas in childhood should not be misinterpreted<br />
as a sign of probable pancreatitis when<br />
scanning a child with abdominal pain (Fig. 9.4).<br />
Pathology of the pancreas<br />
Pancreatic abnormalities are relatively uncommon<br />
in childhood. Most ultrasound abnormalities are<br />
the result of infiltrative processes associated with<br />
other syndromes or diseases (Table 9.1). Focal<br />
pancreatic lesions are rare.<br />
Ultrasound is an ideal investigation for evaluating<br />
the paediatric pancreas, as a high-frequency<br />
Figure 9.4 Normal pancreas in a 13-year-old girl—<br />
relatively hypoechoic and bulky in comparison with the<br />
adult gland.