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PATHOLOGY OF THE GALLBLADDER AND BILIARY TREE 51<br />

A<br />

Figure 3.16 (A) The gallbladder lumen is filled with stones, causing dense shadowing in the gallbladder fossa. The<br />

thickened gallbladder wall can be demonstrated separately (arrows) from the reflective surface of the stones.<br />

(B) A small layer of bile is visible between the stones and the anterior gallbladder wall.<br />

B<br />

Figure 3.17<br />

Microgallbladder in cystic fibrosis.<br />

Figure 3.18 TS of a porcelain gallbladder<br />

demonstrating a calcified wall with strong acoustic<br />

shadowing.<br />

HYPERPLASTIC CONDITIONS OF THE<br />

GALLBLADDER WALL<br />

Adenomyomatosis<br />

This is a non-inflammatory, hyperplastic condition<br />

which causes gallbladder wall thickening. It may be<br />

mistaken for chronic cholecystitis on ultrasound.<br />

The epithelium which lines the gallbladder wall<br />

undergoes hyperplastic change, extending diverticula<br />

into the adjacent muscular layer of the wall.<br />

These diverticula, or sinuses (known as<br />

Rokitansky–Aschoff sinuses), are visible within the<br />

wall as fluid-filled spaces (Fig. 3.19), which can<br />

bulge eccentrically into the lumen, and may contain<br />

echogenic material or even (normally<br />

pigment) stones.<br />

The wall thickening may be focal or diffuse, and<br />

the sinuses may be little more than hypoechoic

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