Untitled

262 EFFECTS OF STRESSbasolateral amygdala and footshock stimulation on neurons of the central amygdala.Biological Psychiatry, 59: 801–811.Rosenkranz, J. A., & Grace, A. A. (1999). Modulation of basolateral amygdala neuronalfiring and afferent drive by dopamine receptor activation in vivo. Journal of Neuroscience,19, 11027–11039.Rosenkranz, J. A., & Grace, A. A. (2001). Dopamine attenuates prefrontal cortical suppressionof sensory inputs to the basolateral amygdala of rats. Journal of Neuroscience,21, 4090–4103.Rosenkranz, J. A., & Grace, A. A. (2002a). Cellular mechanisms of infralimbic andprelimbic prefrontal cortical inhibition and dopaminergic modulation of basolateralamygdala neurons in vivo. Journal of Neuroscience, 22, 324–337.Rosenkranz, J. A., & Grace, A. A. (2002b). Dopamine-mediated modulation of odourevokedamygdala potentials during Pavlovian conditioning. Nature, 417, 282–287.Rosenkranz, J. A., Moore, H., & Grace, A. A. (2003). The prefrontal cortex regulates lateralamygdala neuronal plasticity and responses to previously conditioned stimuli.Journal of Neuroscience, 23, 11054–11064.Sapolsky, R. M. (1996). Stress, glucocorticoids, and damage to the nervous system: Thecurrent state of confusion. Stress, 1, 1–19.Sapolsky, R. M. (1999). Glucocorticoids, stress, and their adverse neurological effects:Relevance to aging. Experimental Gerontology, 34, 721–732.Sapolsky, R. M., Krey, L. C., & McEwen, B. S. (1985). Prolonged glucocorticoid exposurereduces hippocampal neuron number: Implications for aging. The Journal of Neuroscience,5, 1222–1227.Sapolsky, R. M., Uno, H., Rebert, C. S., & Finch, C. E. (1990). Hippocampal damageassociated with prolonged glucocorticoid exposure in primates. Journal of Neuroscience,10, 2897–2902.Shanks, N., Zalcman, S., Zacharko, R. M., & Anisman, H. (1991). Alterations of centralnorepinephrine, dopamine and serotonin in several strains of mice following acute stressorexposure. Pharmacology, Biochemistry & Behavior, 38, 69–75.Thierry, A. M., Javoy, F., Glowinski, J., & Kety, S. S. (1968). Effects of stress on themetabolism of norepinephrine, dopamine and serotonin in the central nervous systemof the rat. I. Modifications of norepinephrine turnover. Journal of Pharmacology &Experimental Therapeutics, 163, 163–171.Thompson, J. L., Pogue-Geile, M. F., & Grace, A. A. (2004). The interactions amongdevelopmental pathology, dopamine, and stress as a model for the age of onset of schizophreniasymptomatology. Schizophrenia Bulletin, 30, 875–900.Valentino, R. J., & Foote, S. L. (1986). Brain noradrenergic neurons, corticotropinreleasingfactor, and stress. In T. W. Moody (Ed.), Neural and endocrine peptides andreceptors (pp. 101–120). New York: Plenum Press.Valentino, R. J., & Foote, S. L. (1988). Corticotropin-releasing hormone increases tonicbut not sensory-evoked activity of noradrenergic locus coeruleus neurons in unanesthetizedrats. Journal of Neuroscience, 8, 1016–1025.Veening, J. G., Swanson, L. W., & Sawchenko, P. E. (1984). The organization of projectionsfrom the central nucleus of the amygdala to brainstem sites involved in centralautonomic regulation: A combined retrograde transport-immunohistochemical study.Brain Research 303(2), 337–357.Walker, E. F., & Diforio, D. (1997). Schizophrenia: A neural diathesis-stress model. PsychologicalReview, 104, 667–685.Weinberger, D. R., Zigun, J. R., Bartley, A. J., Jones, D. W., & Torrey, E. F. (1992).