GTMB 7 - Gene Therapy & Molecular Biology

Gene Therapy and Molecular Biology Vol 7, page 87by antisense cdk2 kinase oligonucleotides. J Clin Invest 93,1458-1464.Morishita, R, Gibbons, GH, Ellison, KE, Nakajima, M, Zhang, L,Kaneda, Y, Ogihara, T, and Dzau, VJ (1993). Singleintraluminal delivery of antisense cdc2 kinase andproliferating-cell nuclear antigen oligonucleotides results inchronic inhibition of neointimal hyperplasia. Proc Natl AcadSci USA 90, 8474-8478.Morishita, R, Gibbons, GH, Horiuchi, M, Ellison, KE, Nakajima,M, Zhang, L, Kaneda, Y, Ogihara, T, and Dzau, VJ (1995).A gene therapy strategy using a transcription factor decoy ofthe E2F binding site inhibits smooth muscle cell proliferationin vivo. Proc Natl Acad Sci USA 92, 5855-5859.Morishita, R, Gibbons, GH, Kaneda, Y, Ogihara, T, and Dzau,VJ (1994b). Pharmacokinetics of antisenseoligodeoxyribonucleotides (cyclin B1 and CDC 2 kinase) inthe vessel wall in vivo: enhanced therapeutic utility forrestenosis by HVJ-liposome delivery. Gene 149, 13-19.Nakamura, T, Morishita, R, Asai, T, Tsuboniwa, N, Aoki, M,Sakonjo, H, Yamasaki, K, Hashiya, N, Kaneda, Y, andOgihara, T (2002). Molecular strategy using cis-element'decoy' of E2F binding site inhibits neointimal formation inporcine balloon-injured coronary artery model. Gene Ther 9,488-494.Nobuyoshi, M, Kimura, T, Ohishi, H, Horiuchi, H, Nosaka, H,Hamasaki, N, Yokoi, H, and Kim, K (1991). Restenosis afterpercutaneous transluminal coronary angioplasty: pathologicobservations in 20 patients. J Am Coll Cardiol 17, 433-439.O'Brien, ER, Alpers, CE, Stewart, DK, Ferguson, M, Tran, N,Gordon, D, Benditt, EP, Hinohara, T, Simpson, JB, andSchwartz, SM (1993). Proliferation in primary and restenoticcoronary atherectomy tissue. Implications forantiproliferative therapy. Circ Res 73, 223-231.O'Brien, ER, Urieli-Shoval, S, Garvin, MR, Stewart, DK,Hinohara, T, Simpson, JB, Benditt, EP, and Schwartz, SM(2000). Replication in restenotic atherectomy tissue.Atherosclerosis 152, 117-126.Olson, NE, Kozlowski, J, and Reidy, MA (2000). Proliferation ofintimal smooth muscle cells. Attenuation of basic fibroblastgrowth factor 2-stimulated proliferation is associated withincreased expression of cell cycle inhibitors. J Biol Chem275, 11270-11277.Orekhov, AN, Andreeva, ER, Mikhailova, IA, and Gordon, D(1998). Cell proliferation in normal and atherosclerotichuman aorta: proliferative splash in lipid-rich lesions.Atherosclerosis 139, 41-48.Parkes, JL, Cardell, RR, Hubbard, FC, Hubbard, D, Meltzer, A,and Penn, A (1991). Cultured human atherosclerotic plaquesmooth muscle cells retain transforming potential and displayenhanced expression of the myc protooncogene. Am JPathol 138, 765-775.Peeper, DS, Upton, TM, Ladha, MH, Neuman, E, Zalvide, J,Bernards, R, DeCaprio, JA, and Ewen, ME (1997). Rassignalling linked to the cell-cycle machinery by theretinoblastoma protein. Nature 386, 177-181.Perlman, H, Luo, Z, Krasinski, K, Le Roux, A, Mahfoudi, A,Smith, RC, Branellec, D, and Walsh, K (1999). Adenovirusmediateddelivery of the Gax transcription factor to ratcarotid arteries inhibits smooth muscle proliferation andinduces apoptosis. Gene Ther 6, 758-763.Perlman, H, Suzuki, E, Simonson, M, Smith, RC, and Walsh, K(1998). GATA-6 induces p21 Cip1 expression and G1 cellcycle arrest. J Biol Chem 273, 13713-13718.Philipp-Staheli, J, Payne, SR, and Kemp, CJ (2001). p27 Kip1 :regulation and function of a haploinsufficient tumorsuppressor and its misregulation in cancer. Exp Cell Res264, 148-168.Pickering, JG, Weir, L, Jekanowski, J, Kearney, MA, and Isner,JM (1993). Proliferative activity in peripheral and coronaryatherosclerotic plaque among patients undergoingpercutaneous revascularization. J Clin Invest 91, 1469-1480.Pronk, GJ, and Bos, JL (1994). The role of p21ras in receptortyrosine kinase signalling. Biochim Biophys Acta 1198,131-147.Pukac, LA, Castellot, JJ, Jr., Wright, TC, Jr., Caleb, BL, andKarnovsky, MJ (1990). Heparin inhibits c-fos and c-mycmRNA expression in vascular smooth muscle cells. CellRegul 1, 435-443.Pyles, JM, March, KL, Mehdi, K, Wilenski, RL, and Adam, LP(1997). Activation of MAP Kinase in vivo follows balloonoverstretch injury of porcine coronary and carotid arteries.Circ Res 81, 904-910.Reilly, CF, Kindy, MS, Brown, KE, Rosenberg, RD, andSonenshein, GE (1989). Heparin prevents vascular smoothmuscle cell progression through the G1 phase of the cellcycle. J Biol Chem 264, 6990-6995.Rekhter, MD, and Gordon, D (1995). Active proliferation ofdifferent cell types, including lymphocytes, in humanatherosclerotic plaques. Am J Pathol 147, 668-677.Robinson, KA, Chronos, NA, Schieffer, E, Palmer, SJ, Cipolla,GD, Milner, PG, and King, SB, 3rd (1997). Endoluminallocal delivery of PCNA/cdc2 antisense oligonucleotides byporous balloon catheter does not affect neointima formationor vessel size in the pig coronary artery model ofpostangioplasty restenosis. Cathet Cardiovasc Diagn 41,348-353.Rodriguez-Campos, A, Ruiz-Enriquez, P, Faraudo, S, andBadimon, L (2001). Mitogen-induced p53 downregulationprecedes vascular smooth muscle cell migration from healthytunica media and proliferation. Arterioscler Thromb VascBiol 21, 214-219.Roque, F, Mon, G, Belardi, J, Rodriguez, A, Grinfeld, L, Long,R, Grossman, S, Malcolm, A, Zon, G, Ormont, ML,Fischman, DL, Shi, Y, and Zalewski, A (2001a). Safety ofintracoronary administration of c-myc antisense oligomersafter percutaneous transluminal coronary angioplasty(PTCA). Antisense Nucleic Acid Drug Dev 11, 99-106.Roque, M, Reis, ED, Cordon-Cardo, C, Taubman, MB, Fallon,JT, Fuster, V, and Badimon, JJ (2001b). Effect of p27deficiency and rapamycin on intimal hyperplasia: in vivo andin vitro studies using a p27 knockout mouse model. LabInvest 81, 895-903.Rosenfeld, ME, and Ross, R (1990). Macrophage and smoothmuscle cell proliferation in atherosclerotic lesions of WHHLand comparably hypercholesterolemic fat-fed rabbits.Arteriosclerosis 10, 680-687.Ross, R (1999). Atherosclerosis: an inflammatory disease. NEngl J Med 340, 115-126.Rothman, A, Woler, B, Button, D, and Taylor, P (1994).Immediate-early gene expression in respose to hypertrophicand proliferative stimuli in pulmonary arterial smooth musclecells. J Biol Chem 269, 6399-6404.Scheinman, M, Ascher, E, Levi, GS, Hingorani, A, Shirazian, D,and Seth, P (1999). p53 gene transfer to the injured ratcarotid artery decreases neointimal formation. J Vasc Surg29, 360-369.Scott, S, O'Sullivan, M, Hafizi, S, Shapiro, LM, and Bennett, MR(2002). Human vascular smooth muscle cells from restenosisor in-stent stenosis sites demonstrate enhanced responses top53. Implications for brachytherapy and drug treatment forrestenosis. Circ Res 90, 398-404.Shi, Y, Dodge, GR, Hall, DJ, Desrochers, PE, Fard, A, Shaheen,F, Talbot, C, Yurgenev, L, and Zalewski, A (1994a).Inhibition of type 1 collagen synthesis in vascular smooth87