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GTMB 7 - Gene Therapy & Molecular Biology

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<strong>Gene</strong> <strong>Therapy</strong> and <strong>Molecular</strong> <strong>Biology</strong> Vol 7, page 89Yonemitsu, Y, Kaneda, Y, Tanaka, S, Nakashima, Y, Komori, K,Sugimachi, K, and Sueishi, K (1998). Transfer of wild-typep53 gene effectively inhibits vascular smooth muscle cellproliferation in vitro and in vivo. Circ Res 82, 147-156.Zhou, YF, Leon, MB, Waclawiw, MA, Popma, JJ, Yu, ZX,Finkel, T, and Epstein, SE (1996). Association between priorcytomegalovirus infection and the risk of restenosis aftercoronary atherectomy. N Engl J Med 335, 624-630.Zhou, YF, Yu, ZX, Wanishsawad, C, Shou, M, and Epstein, SE(1999). The immediate early gene products of humancytomegalovirus increase vascular smooth muscle cellmigration, proliferation, and expression of PDGF betareceptor.Biochem Biophys Res Commun 256, 608-613.Zhu, NL, Wu, L, Liu, PX, Gordon, EM, Anderson, WF, Starnes,VA, and Hall, FL (1997). Downregulation of cyclin G1expression by retrovirus-mediated antisense gene transferinhibits vascular smooth muscle cell proliferation andneointima formation. Circulation 96, 628-635.Zou, X, Rudchenko, S, Wong, K-k, and Calame, K (1997).Induction of c-myc transcription by the v-Abl tyrosine kinaserequires Ras, Raf1, and cyclin-dependent kinases. <strong>Gene</strong>sDev 11, 654-662.Zou, Y, Hu, Y, Metzler, B, and Xu, Q (1998). Signaltransduction in arteriosclerosis: mechanical stress-activatedMAP kinases in vascular smooth muscle cells (review). Int JMol Med 1, 827-834.89

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