Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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67. Itota T, Tashiro Y, Torii Y, Nishitani Y, McCabe JF, Yoshiyama M. Calbindin D- 28k distribution in odontoblasts underneath tertiary dentine in human carious teeth. Arch Oral Biol 2004; 49(1):37-43. 68. Avery JK. Agents affecting tooth and bone development. In: Avery JK, Steele PF, editors. Oral development and histology. Stuttgart: Thieme, 2001:141-152. 69. Purvis RJ, Barrie WJ, MacKay GS, Wilkinson EM, Cockburn F, Belton NR. Enamel hypoplasia <strong>of</strong> the teeth associated with neonatal tetany: a manifestation <strong>of</strong> maternal vitamin-D deficiency. Lancet 1973; 2(7833):811-814. 70. Large DM, Mawer EB, Davies M. Dystrophic calcification, cataracts, and enamel hypoplasia due to long- standing, privational vitamin D deficiency. Metab Bone Dis Relat Res 1984; 5(5):215-218. 71. Giunta JL. Dental changes in hypervitaminosis D. Oral Surg Oral Med Oral Pathol Oral Radiol Endod 1998; 85(4):410-413. 72. Taylor G, Day CDM. Relations <strong>of</strong> vitamin D and mineral deficiencies to dental caries. Br Med J 1939;919-921. 73. Mellanby M, Pattison C, Proud J. The effect <strong>of</strong> diet on the development and extension <strong>of</strong> caries in the teeth <strong>of</strong> children. Br Med J 1924; 2:354-355. 74. Mellanby M. An experimental study <strong>of</strong> the influence <strong>of</strong> diet on teeth formation. Lan 1918; ii:767-770. 75. Mellanby M. The influence <strong>of</strong> diet on teeth formation. Lancet 1918;(4971):767-770. 76. Mellanby E. An experimental investigation on rickets. Lancet 1919; 4985:407-412. 77. Seow WK, Brown JP, Tudehope DA, O'Callaghan M. Dental defects in the deciduous dentition <strong>of</strong> premature infants with low birth weight and neonatal rickets. Pediatr Dent 1984; 6(2):88-92. 78. Shelling DH, Anderson GM. Relation <strong>of</strong> rickets and vitamin D to the incidence <strong>of</strong> dental caries, enamel hypoplasia and malocclusions in children. J Am Dent Assoc 1936; 23:840-846. 79. Gottlieb B. Rachitis and enamel hypoplasia. Dent Cosmos 1920; 62:1209-1221- 1316-1326. 80. Eliot MM, Souther SP, Anderson BG, Arnim SS. A study <strong>of</strong> the teeth <strong>of</strong> a group <strong>of</strong> school children previously examined for rickets. Am J Dis Child 1934; 48(4):713- 729. 81. Eliot MM, Souther SP, Anderson BA. A study <strong>of</strong> the teeth <strong>of</strong> a group <strong>of</strong> school children previously examined for rickets. Am J Dis Child 1933; 46:458-461. 1.3-42
82. MacKay H, Rose S. <strong>Vitamin</strong> D deficiency, dental caries, and tonsillar enlargement. Lancet 1931; 2:1230-1235. 83. Fraser D, Nikiforuk G. The etiology <strong>of</strong> enamel hypoplasia in children--a unifying concept. J Int Assoc Dent Child 1982; 13(1):1-11. 84. Nikiforuk G, Fraser D. Etiology <strong>of</strong> enamel hypoplasia and interglobular dentin: the roles <strong>of</strong> hypocalcemia and hypophosphatemia. Metab Bone Dis & Rel Res 1979; 2:17-23. 85. Seow WK, Latham SC. The spectrum <strong>of</strong> dental manifestations in vitamin D- resistant rickets: implications for management. Pediatr Dent 1986; 8(3):245-250. 86. Mellanby M. The chief dietetic and environmental factors responsible for the high incidence <strong>of</strong> dental caries: correlation between animal and human investigations. Br Dent J 1928; 49(14):769-792. 87. Cockburn F, Belton NR, Purvis RJ, Giles MM, Brown JK, Turner TL et al. <strong>Maternal</strong> vitamin D intake and mineral metabolism in mothers and their newborn infants. Br Med J 1980; 281(6232):11-14. 88. Aine L, Backstrom MC, Maki R, Kuusela AL, Koivisto AM, Ikonen RS et al. Enamel defects in primary and permanent teeth <strong>of</strong> children born prematurely. J Oral Pathol Med 2000; 29(8):403-409. 89. Muratbegovic A, Markovic N, Ganibegovic SM. Molar incisor hypomineralisation in Bosnia and Herzegovina: aetiology and clinical consequences in medium caries activity population. Eur Arch Paediatr Dent 2007; 8(4):189-194. 90. Goodman AH, Martinez C, Chavez A. Nutritional supplementation and the development <strong>of</strong> linear enamel hypoplasias in children from Tezonteopan, Mexico. Am J Clin Nutr 1991; 53(3):773-781. 91. HARNDT E. [Caries prophylaxis in regard to enamel structure.]. Int Z <strong>Vitamin</strong>forsch Beih 1958; 28(Supp 7):52-67. 92. Greenebaum J, Johnson E, Mitchell A, Selkirk T, Stillwell F. Studies in rickets. IV. Relation <strong>of</strong> diet during pregnancy to the deciduous teeth. J Am Dent Assoc 1930; 17:717-725. 93. Hess AF, Abramson H. The etiology <strong>of</strong> dental caries. Dent Cosmos 1931; 73(9):849-866. 94. Buhl S, Wetzel WE, Bodeker RH. [Studies on the incidence <strong>of</strong> caries in 6- to 48- month old infants]. Dtsch Zahnarztl Z 1989; 44(9):673-677. 1.3-43
Page 1 and 2:
Influence
Page 3 and 4:
Abstract Influence
Page 5 and 6:
Acknowledgements This longitudinal
Page 7 and 8:
Table of Contents
Page 9 and 10:
Status and Non-<st
Page 11 and 12:
Table 1.1-1 - Recent studies report
Page 13 and 14:
Table 1.1-2 - Previous used terms f
Page 15 and 16:
The Link Between Enamel Hypoplasia
Page 17 and 18:
If enamel hypoplasia results from n
Page 19 and 20:
malocclusions. 145 Additional human
Page 21 and 22:
Daily intakes of v
Page 23 and 24:
13. Milnes AR, Rubin CW, Karpa M, T
Page 25 and 26:
40. Definition of
Page 27 and 28:
71. Watt RG. From victim blaming to
Page 29 and 30:
97. Fearne JM, Bryan EM, Elliman AM
Page 31 and 32:
124. Burt BA, Pai S. Does low birth
Page 33 and 34:
151. Tinanoff N, D
Page 35 and 36:
Section 2 - Risk Factors for Enamel
Page 37 and 38:
correspond to the timing of
Page 39 and 40:
een published. 3,4,17,19 The intent
Page 41 and 42:
Table 1.2-2 - Risk factors for DDE
Page 43 and 44:
Opacity 51 Implicated Risk Developm
Page 45 and 46:
Implicated Risk Factor Factors asso
Page 47 and 48:
o Metabolic Abnormalities and Healt
Page 49 and 50:
12 months of life.
Page 51 and 52: Other Factors Some uncommon risk fa
Page 53 and 54: devastating problem of</str
Page 55: 13. An epidemiological index <stron
Page 58 and 59: 54. Seow WK, Perham S. Enamel hypop
Page 60 and 61: 82. Berdal A, Papagerakis P, Hotton
Page 62 and 63: Vitamin D may be d
Page 64 and 65: have been used to prof</str
Page 66 and 67: health’. In addition, articles we
Page 68 and 69: 36 month trial had ended. 45 The nu
Page 70 and 71: Recently, a cross-sectional study <
Page 72 and 73: andomized, placebo-controlled study
Page 74 and 75: Further, amelogenin and enamelin, p
Page 76 and 77: A study of Austral
Page 78 and 79: Both dietary interventions and supp
Page 80 and 81: incidence of carie
Page 82 and 83: important nutrient. However, many <
Page 84 and 85: caries, and more hardening
Page 86 and 87: controls. 106 Interestingly, childr
Page 88 and 89: A few years later, additional resea
Page 90 and 91: Delayed Tooth Eruption Vita
Page 92 and 93: Description of Pop
Page 94 and 95: Influence
Page 96 and 97: D has an effect on the dentition, p
Page 98 and 99: 14. Utiger RD. The need for more vi
Page 100 and 101: 42. Spiller WF, Jr. A clinical eval
Page 104 and 105: 95. Bruszt P. Kariesuntersuchungen
Page 106 and 107: 121. Seow WK, Needleman HL, Holm IA
Page 108 and 109: Chapter 2 - Methods for a Prospecti
Page 110 and 111: Each participant also completed an
Page 112 and 113: Table 2.2 - Recognized case definit
Page 114 and 115: Population The target population fo
Page 116 and 117: allowed the project to suffer some
Page 118 and 119: women of limited m
Page 120 and 121: involving the surface of</s
Page 122 and 123: and training cup. In addition, each
Page 124 and 125: The questionnaire was administered
Page 126 and 127: statistics including frequencies an
Page 128 and 129: elationship between the two. Poisso
Page 130 and 131: References 1. Combs GF. Vit
Page 132 and 133: 27. Seow WK, Amaratunge A, Bennett
Page 134 and 135: Table 3.2 - Self-declared heritage
Page 136 and 137: Question Number Valid % Do you take
Page 138 and 139: Maternal Awareness
Page 140 and 141: time of recruitmen
Page 142 and 143: Table 3.6- Prenatal</strong
Page 144 and 145: A total of 77 care
Page 146 and 147: indicated that they were receiving
Page 148 and 149: and 11 hypoplasia). DDEs were also
Page 150 and 151: Caries rates for children in this p
Page 152 and 153:
Table 3.12 - Frequency of</
Page 154 and 155:
Chapter 4 - Results: Associations <
Page 156 and 157:
II. Relationship Between 25(OH)D Le
Page 158 and 159:
Pregnancy & Health Prof</st
Page 160 and 161:
The frequency of v
Page 162 and 163:
did not (42.9 ± 21.2 nmol/L vs. 49
Page 164 and 165:
Early Childhood Caries (ECC) Pr<str
Page 166 and 167:
likely to have vitamin D levels at
Page 168 and 169:
did not statistically differ from v
Page 170 and 171:
Figure 4.4 - Predicted number <stro
Page 172 and 173:
Table 4.7 - Relationship between ds
Page 174 and 175:
frequently, and less likely to have
Page 176 and 177:
health as average or poor, and more
Page 178 and 179:
tail (p=.034) suggesting that early
Page 180 and 181:
The next series of
Page 182 and 183:
Results from the second model appea
Page 184 and 185:
Table 5.6 - Multiple regression sta
Page 186 and 187:
vitamin D. However, it did suggest
Page 188 and 189:
significant at the bivariate level
Page 190 and 191:
Table 5.12(a) - Logistic regression
Page 192 and 193:
III. Multivariate Regression Analys
Page 194 and 195:
Table 5.14 - Logistic regression fo
Page 196 and 197:
Page 198 and 199:
Page 200 and 201:
Table 5.19(b) - Backwards logistic
Page 202 and 203:
Table 5.20(a) - Logistic regression
Page 204 and 205:
Table 5.21 - Poisson regression for
Page 206 and 207:
If enamel hypoplasia is present and
Page 208 and 209:
Since maternal calcium levels were
Page 210 and 211:
vitamin D deficiency, and rickets)
Page 212 and 213:
to obtain it. Health literacy may b
Page 214 and 215:
vitamin D from the diet as dietary
Page 216 and 217:
home environment can be determinant
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hormone, calcium absorption and bon
Page 220 and 221:
important nutrient. For many, this
Page 222 and 223:
this potential relationship with ac
Page 224 and 225:
genetic conditions, childhood medic
Page 226 and 227:
vitamin D status including milk int
Page 228 and 229:
y cariogenic oral flora. 49 Since e
Page 230 and 231:
(49.8 ± 26.3 nmol/L vs. 45.1 ± 20
Page 232 and 233:
pregnancy, which may have influence
Page 234 and 235:
observed and it allowed multiple ou
Page 236 and 237:
scientific evidence linking prenata
Page 238 and 239:
cognizant that children who undergo
Page 240 and 241:
preventive program would need to be
Page 242 and 243:
makers in the formative stages. As
Page 244 and 245:
Surprisingly, the final regression
Page 246 and 247:
13. First Nations IaMHCCPS. <strong
Page 248 and 249:
38. Hollis BW. Circulating 25-hydro
Page 250 and 251:
65. Li Y, Navia JM, Bian JY. Caries
Page 252 and 253:
91. Brownell MD, Guevremont A, Au W
Page 254 and 255:
Chapter 7 - Conclusions This thesis
Page 256 and 257:
This study provided an interesting
Page 258:
esiding in disadvantaged communitie
Page 271 and 272:
If you are taking vitamins during t
Page 273 and 274:
Do you Eat? Never Rarely (less than
Page 275 and 276:
What problems can early childhood c
Page 277 and 278:
Prenatal Nutrition
Page 279 and 280:
Did you ever add sugar to the bottl
Page 281 and 282:
Enamel Hypoplasia Enamel Hypoplasia
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Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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