Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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40. Sweeney EA, Cabrera J, Urrutia J, Mata L. Factors associated with linear hypoplasia <strong>of</strong> human deciduous incisors. J Dent Res 1969; 48(6):1275-1279. 41. Benusis KP, Pueschel SM, Hum C. Enamel hypoplasia in children with galactosemia associated with periods <strong>of</strong> poor control. ASDC J Dent Child 1978; 45(1):73-75. 42. Shusterman S, Fellers FX. The prevalence <strong>of</strong> enamel defects in childhood nephrotic syndrome. ASDC J Dent Child 1969; 36(6):435-440. 43. Arrow P. Enamel hypoplasia <strong>of</strong> the primary dentition in a 4-year-old with intestinal lymphangiectasia. Int J Paediatr Dent 2005; 15(5):380-384. 44. Ralph PM, Troutman KC. The oral manifestations <strong>of</strong> intestinal lymphangiectasia: case report. Pediatr Dent 1996; 18(7):461-464. 45. Dummer PM, Cardiff BD. Severe enamel hypoplasia in a case <strong>of</strong> intestinal lymphangiectasia: a rare protein-losing enteropathy. Oral Surg Oral Med Oral Pathol 1977; 43(5):702-706. 46. Aine L. Dental enamel defects and dental maturity in children and adolescents with coeliac disease. Proc Finn Dent Soc 1986; 82 Suppl 3:1-71. 47. Hallett KB, Radford DJ, Seow WK. Oral health <strong>of</strong> children with congenital cardiac diseases: a controlled study. Pediatr Dent 1992; 14(4):224-230. 48. Stagno S, Pass RF, Thomas JP, Navia JM, Dworsky ME. Defects <strong>of</strong> tooth structure in congenital cytomegalovirus infection. Pediatrics 1982; 69(5):646-648. 49. Kraus BS, Ames MD, Clark GR. Effects <strong>of</strong> maternal rubella on dental crown development. Clin Pediatr (Phila) 1969; 8(4):204-215. 50. Guggenheimer J, Nowak AJ, Michaels RH. Dental manifestations <strong>of</strong> the rubella syndrome. Oral Surg Oral Med Oral Pathol 1971; 32(1):30-37. 51. Aine L, Backstrom MC, Maki R, Kuusela AL, Koivisto AM, Ikonen RS et al. Enamel defects in primary and permanent teeth <strong>of</strong> children born prematurely. J Oral Pathol Med 2000; 29(8):403-409. 52. Li Y, Navia JM, Bian JY. Prevalence and distribution <strong>of</strong> developmental enamel defects in primary dentition <strong>of</strong> Chinese children 3-5 years old. Community Dent Oral Epidemiol 1995; 23(2):72-79. 53. Seow WK, Masel JP, Weir C, Tudehope DI. Mineral deficiency in the pathogenesis <strong>of</strong> enamel hypoplasia in prematurely born, very low birthweight children. Pediatr Dent 1989; 11(4):297-302. 1.2-23
Page 1 and 2:
Influence
Page 3 and 4:
Abstract Influence
Page 5 and 6: Acknowledgements This longitudinal
Page 7 and 8: Table of Contents
Page 9 and 10: Status and Non-<st
Page 11 and 12: Table 1.1-1 - Recent studies report
Page 13 and 14: Table 1.1-2 - Previous used terms f
Page 15 and 16: The Link Between Enamel Hypoplasia
Page 17 and 18: If enamel hypoplasia results from n
Page 19 and 20: malocclusions. 145 Additional human
Page 21 and 22: Daily intakes of v
Page 23 and 24: 13. Milnes AR, Rubin CW, Karpa M, T
Page 25 and 26: 40. Definition of
Page 27 and 28: 71. Watt RG. From victim blaming to
Page 29 and 30: 97. Fearne JM, Bryan EM, Elliman AM
Page 31 and 32: 124. Burt BA, Pai S. Does low birth
Page 33 and 34: 151. Tinanoff N, D
Page 35 and 36: Section 2 - Risk Factors for Enamel
Page 37 and 38: correspond to the timing of
Page 39 and 40: een published. 3,4,17,19 The intent
Page 41 and 42: Table 1.2-2 - Risk factors for DDE
Page 43 and 44: Opacity 51 Implicated Risk Developm
Page 45 and 46: Implicated Risk Factor Factors asso
Page 47 and 48: o Metabolic Abnormalities and Healt
Page 49 and 50: 12 months of life.
Page 51 and 52: Other Factors Some uncommon risk fa
Page 53 and 54: devastating problem of</str
Page 55: 13. An epidemiological index <stron
Page 59 and 60: 69. Dini EL, Holt RD, Bedi R. Preva
Page 61 and 62: Section 3 - Influence</stro
Page 63 and 64: from two forms: 1) animals (D 3 or
Page 65 and 66: Figure 1.3-2 - Schematic representa
Page 67 and 68: A 1971 publication presented result
Page 69 and 70: Among Chinese adults, the VDR Taq I
Page 71 and 72: Tooth Loss and Alveolar Bone Loss I
Page 73 and 74: increase in alveolar bone mass amon
Page 75 and 76: Long-standing, vitamin D deficiency
Page 77 and 78: vitamin D resistant rickets (VDRR)
Page 79 and 80: A 1958 review on the topic
Page 81 and 82: Gedicke’s paper examined the infl
Page 83 and 84: progression of unt
Page 85 and 86: oil retards the progression <strong
Page 87 and 88: new cavities in the permanent denti
Page 89 and 90: Hungarian preschool children as no
Page 91 and 92: Table 1.3-1 - Other dental conditio
Page 93 and 94: Table 1.3-2 - Other circumstantial
Page 95 and 96: Influence
Page 97 and 98: References 1. Norman AW, Bouillon R
Page 99 and 100: 29. Nozza JM, Rodda CP. Vit
Page 101 and 102: 55. Nibali L, Parkar M, D'Aiuto F,
Page 103 and 104: 82. MacKay H, Rose S. Vitam
Page 105 and 106: 108. McBeath E. Vitamin</st
Page 107 and 108:
135. Dunning J. The dental status <
Page 109 and 110:
2. To determine the prevalence <str
Page 111 and 112:
The final component of</str
Page 113 and 114:
methods used to minimize participan
Page 115 and 116:
iostatistician to ensure that the m
Page 117 and 118:
senior researchers and those health
Page 119 and 120:
annual income, to establish a socio
Page 121 and 122:
infants had evidence of</st
Page 123 and 124:
was recruited in July of</s
Page 125 and 126:
An honorarium was given to each par
Page 127 and 128:
Table 2.5 - Sources of</str
Page 129 and 130:
Funding Operating grants from the s
Page 131 and 132:
14. Gregg MB. Field epidemiology. N
Page 133 and 134:
Chapter 3 - Descriptive Results A t
Page 135 and 136:
24.2% (40/165) correctly indicated
Page 137 and 138:
Overall, 50.0% reported that they d
Page 139 and 140:
finances or lack of</strong
Page 141 and 142:
Table 3.5 - Frequency of</s
Page 143 and 144:
was 13 months. Males accounted for
Page 145 and 146:
their infant had sugar added to the
Page 147 and 148:
months of age. The
Page 149 and 150:
Table 3.8 - Frequency of</s
Page 151 and 152:
enamel, ten had caries into dentin,
Page 153 and 154:
References 1. Healthy Child Manitob
Page 155 and 156:
Figure 4.1 - Correlation of
Page 157 and 158:
Since endogenous synthesis
Page 159 and 160:
While those who were uncertain abou
Page 161 and 162:
arms and legs felt weak had signifi
Page 163 and 164:
Similarly, chi-square analysis reve
Page 165 and 166:
Exposure to Sunlight Only one <stro
Page 167 and 168:
Chi-square analysis revealed that w
Page 169 and 170:
Poisson regression was used to exam
Page 171 and 172:
T test analyses were undertaken to
Page 173 and 174:
Variable No Enamel Hypoplasia (%) E
Page 175 and 176:
Variable No ECC* (%) ECC* (%) P val
Page 177 and 178:
hypoplasia and gestational diabetes
Page 179 and 180:
Chapter 5 - Multivariate Regression
Page 181 and 182:
Table 5.2 - Multiple regression ana
Page 183 and 184:
controlling for the influence <stro
Page 185 and 186:
Table 5.7 - Logistic regression for
Page 187 and 188:
not drinking milk of</stron
Page 189 and 190:
Table 5.11 - Logistic regression fo
Page 191 and 192:
heard of vitamin D
Page 193 and 194:
Page 195 and 196:
Page 197 and 198:
only included annual income and ove
Page 199 and 200:
enamel hypoplasia (p $18,000) Drink
Page 201 and 202:
chapter, backwards logistic regress
Page 203 and 204:
Table 5.20(b) - Backwards logistic
Page 205 and 206:
Enamel hypoplasia (Yes = 1, No = 0)
Page 207 and 208:
that infant age, the presence <stro
Page 209 and 210:
Chapter 6 - Discussion There is no
Page 211 and 212:
these participants the loss <strong
Page 213 and 214:
70 years (66.9 nmol/L) than what is
Page 215 and 216:
The vast majority of</stron
Page 217 and 218:
emineralized with fluoride they sti
Page 219 and 220:
after controlling for other health
Page 221 and 222:
acquire nutritious foods. While the
Page 223 and 224:
defects. Low awareness of</
Page 225 and 226:
for additional factors such as the
Page 227 and 228:
elationship. However, infants belon
Page 229 and 230:
a significant association between 2
Page 231 and 232:
would have had to be administered a
Page 233 and 234:
the possibility that better materna
Page 235 and 236:
Over the last decade ECC has garner
Page 237 and 238:
ECC is theoretically preventable. S
Page 239 and 240:
caries-risk. This is not a universa
Page 241 and 242:
were able to assist with strategies
Page 243 and 244:
factors determine whether this data
Page 245 and 246:
References 1. Hildebolt CF. Effect
Page 247 and 248:
25. Caufield PW, Cutter GR, Dasanay
Page 249 and 250:
52. Valaitis R, Hesch R, Passarelli
Page 251 and 252:
77. Niessen LW, Grijseels EW, Rutte
Page 253 and 254:
104. World Health Organization. Twe
Page 255 and 256:
pregnancy may reduce the incidence
Page 257 and 258:
This is the first prospectively des
Page 270 and 271:
Prenatal Nutrition
Page 272 and 273:
How do you get to and from shopping
Page 274 and 275:
Green Vegetables Broccoli Calcium R
Page 276 and 277:
Exposure to Sunlight: During the su
Page 278 and 279:
Do you believe your child has any d
Page 280 and 281:
Have you had to use a foodbank sinc
Page 282:
ECC = 1 or more primary teeth with
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Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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